Myelin made to measure (original) (raw)
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- Published: 01 May 2004
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Nature Reviews Neuroscience volume 5, page 343 (2004)Cite this article
The speed at which action potentials are conducted along an axon depends largely on the diameter of the axon and the thickness of its myelin sheath — two factors that are closely related. Maintaining the precise control of conductance velocity is essential for the proper function of the nervous system, and it has been suggested that myelin thickness might be controlled by interactions between ligands produced by the axon and receptors on the myelinating glia. Michailov et al. tested the idea that the axonal ligand that is responsible — in the periphery, at least — is Nrg1, interacting with ErbB receptors on Schwann cells.
The Nrg1 gene is expressed in neurons of the sciatic nerve in mice, and ErbB2 and ErbB3 are expressed by Schwann cells. If interactions between Nrg1 and ErbB receptors control myelin thickness, changes in the amount of ligand or receptor might alter the thickness of the myelin sheath. When the authors generated compound heterozygote mice that had reduced gene dosages of both Nrg1 and ErbB2, they found that the myelin in the sciatic nerves of the mice was significantly thinner than usual. Although the mice seemed normal, the conduction velocity in their nerves was also reduced, even though the sizes of the axons were unchanged.
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ORIGINAL RESEARCH PAPER
- Michailov, G. V. et al. Axonal neuregulin-1 regulates myelin sheath thickness. Science 25 March 2004 (10.1126/science.1095862)
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- Rachel Jones
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Jones, R. Myelin made to measure.Nat Rev Neurosci 5, 343 (2004). https://doi.org/10.1038/nrn1400
- Issue Date: 01 May 2004
- DOI: https://doi.org/10.1038/nrn1400