High Endothelial Venule Reporter Mice to Probe Regulation of Lymph Node Vasculature (original) (raw)
Abstract
Lymphotoxin (LT) is crucial for the regulation of HEV adhesion molecules MAdCAM-1 and PNAd and a sulfotransferase, GlcNAc6ST-2 (gene symbol Chst4); here called HEC-6ST. Following immunization, some HEVs express markers of both HEVs (PNAd) and LVs (LYVE-1). In order to evaluate this process in real time, we have developed mice transgenic for a construct that consists of an HEV specific gene driving a green fluorescent reporter gene (eGFP). These mice express the reporter gene in HEVs in concurrence with the endogenous gene and PNAd. Additional mice transgenic for lymphatic vessel reporter constructs are in development. These will provide material for in vivo imaging and allow us to evaluate the regulation and interaction of HEVs and LVs.
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Abbreviations
LN:
lymph node
PLN:
peripheral lymph node
LTβR:
lymphotoxin-β receptor
LV:
lymphatic vessel
HEV:
high endothelial venule
PNAd:
peripheral node addressin
HEC-6ST:
high endothelial cell sulfotransferase, HEC-GlcNA6ST, GlcNAc6ST-2, N-acetyl glucosamine 6-O-sulfotransferase-2; carbohydrate (chondroitin 6/keratan) sulfotransferase 4
MAdCAM-1:
mucosal addressin cell adhesion molecule 1
GlyCAM-1:
glycosylation-dependent cell adhesion molecule-1
OX:
oxazolone
LYVE-1:
lymphatic vessel endothelial hyaluronan receptor 1
GFP:
green fluorescent protein
L-sel:
L-selectin
PROX1:
prospero-related homeobox 1
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Acknowledgments
We thank Myriam Hill for figure preparation and Myriam Hill and Daniel Reagan for outstanding technical assistance. This work was supported by U.S. Public Health Service Grants R01 DK057731 and CA016885 (to N.H.R.) and GM09966 (to F.H.R.) from the National Institutes of Health, a Mount Holyoke College Faculty Research Fellowship (to S.S) and a Lymphatic Research Foundation Fellowship (to R.A.M.)
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- Department of Epidemiology and Public Health, Yale University School of Medicine, 10 Amistad Street, P.O. Box 208089, New Haven, CT, 06520, USA
Kevin L. Bentley, Shan Liao, Rawad M. Mounzer & Nancy H. Ruddle - Department of Biological Sciences, Mount Holyoke College, South Hadley, MA, 01075, USA
Sharon Stranford - Department of Radiation Oncology, E.L. Steele Laboratory for Tumor Biology, Massachusetts General Hospital, 100 Blossom St., Cox 7, Boston, MA, 02114, USA
Shan Liao - Department of Internal Medicine, UPMC Montefiore Hospital, N-713, 200 Lothrop St., Pittsburgh, PA, 15213, USA
Rawad M. Mounzer - Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, CT, 06510, USA
Frank H. Ruddle - Departments of Epidemiology and Public Health and Immunobiology, Yale University School of Medicine, New Haven, CT, USA
Nancy H. Ruddle
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Correspondence toNancy H. Ruddle .
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- Dept. Biological Chemistry, Weizmann Institute of Science, 1 Herzl Street, Rehovot, 76100, Israel
David Wallach - Dept. Biological Chemistry, Weizmann Institute of Science, 1 Herzl Street, Rehovot, 76100, Israel
Andrew Kovalenko - Kennedy Institute of Rheumatology, Imperial College London, Aspenlea Rd. 1, London, W6 8LH, United Kingdom
Marc Feldmann
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Bentley, K.L., Stranford, S., Liao, S., Mounzer, R.M., Ruddle, F.H., Ruddle, N.H. (2011). High Endothelial Venule Reporter Mice to Probe Regulation of Lymph Node Vasculature. In: Wallach, D., Kovalenko, A., Feldmann, M. (eds) Advances in TNF Family Research. Advances in Experimental Medicine and Biology, vol 691. Springer, New York, NY. https://doi.org/10.1007/978-1-4419-6612-4\_4
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- DOI: https://doi.org/10.1007/978-1-4419-6612-4\_4
- Published: 22 October 2010
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