Experimental Suppression of Increased Capillary Permeability in Thermal Burns in Rats (original) (raw)

Nature volume 182, pages 949–950 (1958) Cite this article

Abstract

THE possibility has been widely considered that œdema in thermal injury is partly due to release of substances or activation of endogenous mechanisms which increase capillary permeability. There is little convincing evidence in support of such a hypothesis, and the œdema associated with burning has never been suppressed by pharmacological means1.

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References

  1. Sevitt, S., “Burns, Pathology and Therapeutic Applications” (Butterworth, London, 1957).
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  2. Sevitt, S., J. Path. Bact., 75, 27 (1958).
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  3. Spector, W. G., and Willoughby, D. A., Nature, 181, 708 (1958).
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  4. Spector, W. G., and Willoughby, D. A., J. Path. Bact. (in the press).

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Authors and Affiliations

  1. Department of Morbid Anatomy and Medical Unit, University College Hospital Medical School, London, W.C.1.
    W. G. SPECTOR & D. A. WILLOUGHBY

Authors

  1. W. G. SPECTOR
  2. D. A. WILLOUGHBY

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SPECTOR, W., WILLOUGHBY, D. Experimental Suppression of Increased Capillary Permeability in Thermal Burns in Rats.Nature 182, 949–950 (1958). https://doi.org/10.1038/182949a0

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