Product of a transferred H–2Ld gene acts as restriction element for LCMV-specific killer T cells (original) (raw)
- Letter
- Published: 03 June 1982
- Robert S. Goodenow1,
- Leroy Hood1,
- Peter R. Brayton2,
- Jerold G. Woodward3,
- Richard C. Harmon3 &
- …
- Jeffrey A. Frelinger3
Nature volume 297, pages 415–417 (1982)Cite this article
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Abstract
The major histocompatibility complex (MHC) of the mouse, H–2 on chromosome 17, contains several families of genes encoding cell-surface molecules which have a role in mediating immune responses1,2. The class I genes encode a family of homologous membrane proteins including the transplantation antigens K, D and L. These antigens reflect extensive genetic polymorphism which is apparent in the many different class I gene constellations or haplotypes found in mice (for example, BALB/c mice exhibit the H–2 d haplotype and their class I molecules are denoted Kd, DD and Ld). Transplantation antigens serve as targets for T-cell killing in allogeneic immune responses such as in vivo graft rejection3 and destruction of allogeneic cells by cytotoxic T cells _in vitro_4. However, the physiological role of transplantation antigens may be to serve as restricting elements in virus-mediated T-cell killing of infected self cells. Virus infection of mice generates killer T cells whose receptors must interact with the foreign viral antigen and a class I molecule or restricting element for the cytotoxic effector function to be activated5,6. Thus the T-cell receptor recognizes the viral antigen in the context of a class I molecule. To study the interaction between the T-cell receptor and the class I restricting element, we have used the mouse L-cell transformant 8–5 which expresses Ld molecules7 and the K7-65 transformant expressing K molecules (R.S.G. et al., in preparation). Mouse L cells are fibroblasts derived from C3H mice of H–2 k haplotype and monoclonal antibodies can be used to distinguish H–2 d molecules from the endogenous H–2 k products. Recently, we have demonstrated that both Ld (ref. 8) and Kd molecules (unpublished data) expressed on transformed L cells can act as target antigens for alloreactive cytotoxic T lymphocytes. Here we show that the Ld molecule on transformed mouse L cells can serve as a restricting element in lymphocytic choriomeningiris virus (LCMV) infection, whereas its Kd counterpart cannot.
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Authors and Affiliations
- Division of Biology, California Institute of Technology, Pasadena, California, 91125, USA
Anders Örn, Robert S. Goodenow & Leroy Hood - Department of Neurology, University of Southern California Medical School, Los Angeles, California, 90033, USA
Peter R. Brayton - Department of Microbiology, University of Southern California Medical School, Los Angeles, California, 90033, USA
Jerold G. Woodward, Richard C. Harmon & Jeffrey A. Frelinger
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- Anders Örn
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Örn, A., Goodenow, R., Hood, L. et al. Product of a transferred H–2L d gene acts as restriction element for LCMV-specific killer T cells.Nature 297, 415–417 (1982). https://doi.org/10.1038/297415a0
- Received: 23 March 1982
- Accepted: 14 April 1982
- Issue Date: 03 June 1982
- DOI: https://doi.org/10.1038/297415a0