Transient increase in obese gene expression after food intake or insulin administration (original) (raw)
- Letter
- Published: 12 October 1995
- Piet De Vos1,
- Michele Guerre-Millot2,
- Armelle Leturque3,
- Jean Girard3,
- Bart Staels1 &
- …
- Johan Auwerx1
Nature volume 377, pages 527–528 (1995)Cite this article
- 1317 Accesses
- 1055 Citations
- 20 Altmetric
- Metrics details
Abstract
OBESITY is a disorder of energy balance, indicating a chronic disequilibrium between energy intake and expenditure1. Recently, the mouse ob gene2, and subsequently its human and rat homologues2-6, have been cloned. The ob gene product, leptin7, is expressed exclusively in adipose tissue, and appears to be a signalling factor regulating body-weight homeostasis and energy balance2,7-9. Because the level of ob gene expression might indicate the size of the adipose depot, we suggest that it is regulated by factors modulating adipose tissue size. Here we show that ob gene exhibits diurnal variation, increasing during the night, after rats start eating. This variation was linked to changes in food intake, as fasting prevented the cyclic variation and decreased ob messenger RNA. Furthermore, refeeding fasted rats restored ob mRNA within 4 hours to levels of fed animals. A single insulin injection in fasted animals increased ob mRNA to levels of fed controls. Experiments to control glucose and insulin independently in animals, and studies in primary adipocytes, showed that insulin regulates ob gene expression directly in rats, regardless of its glucose-lowering effects. Whereas the ob gene product, leptin, has been shown to reduce food intake and increase energy expenditure7-9, our data demonstrate that ob gene expression is increased after food ingestion in rats, perhaps through a direct action of insulin on the adipocyte.
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 51 print issues and online access
$199.00 per year
only $3.90 per issue
Buy this article
- Purchase on SpringerLink
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Additional access options:
Similar content being viewed by others
References
- Grundy, S. M. & Barnett, J. P. Disease-a-Month 36, 641–731 (1990).
CAS PubMed Google Scholar - Zhang, Y. et al. Nature 372, 425–432 (1994).
Article ADS CAS Google Scholar - Masuzaki, H. et al. Diabetes 44, 855–858 (1995).
Article CAS Google Scholar - Geffroy, S. et al. Genomics 28, 603–604 (1995).
Article CAS Google Scholar - Murakami, T. & Shima, K. Biochem. biophys. Res. Commun. 209, 944–952 (1995).
Article CAS Google Scholar - Funahashi, T. et al. Biochem. biophys. Res. Commun. 211, 469–475 (1995).
Article CAS Google Scholar - Halaas, J. L. et al. Science 269, 543–546 (1995).
Article ADS CAS Google Scholar - Pelleymounter, M. A. et al. Science 269, 540–543 (1995).
Article ADS CAS Google Scholar - Campfield, L. A., Smith,, F. J., Guisez, Y., Devos, R. & Burn, P. Science 269, 546–549 (1995).
Article ADS CAS Google Scholar - Maffei, M. et al. Proc. natn. Acad. Sci. U.SA. 92, 6957–6960 (1995).
Article ADS CAS Google Scholar - Peret, J., Macaire, I. & Chanez, M. J. Nutrition 103, 866–874 (1973).
Article CAS Google Scholar - Jécquier, E. in Obesity (eds Bjorntorp. P. & Brodoff, B. N.) 130–135 (Lippincot, Philadelphia, 1992).
Google Scholar - Gazdar, A. F. et al. Cancer Res. 50, 5488–5496 (1990).
CAS PubMed Google Scholar - De Vos, P., Saladin, R., Auwerx, J. & Staels, B. J. biol. Chem. 270, 15958–15961 (1995).
Article CAS Google Scholar - Cleveland, D. W. et al. Cell 20, 95–105 (1980).
Article CAS Google Scholar - Postic, C. et al. Diabetes 42, 922–929 (1993).
Article CAS Google Scholar - Hajduch, E. J., Guerre-Millo, M., Hainault, I. A., Guichard, C. M. & Lavau, M. M. J. cell. Biochem. 49, 251–258 (1992).
Article CAS Google Scholar
Author information
Authors and Affiliations
- Laboratoire de Biologie des Regulations chez les Eucaryotes, INSERM U325, Institut Pasteur, 1 Rue Calmette, F-59019, Lille, France
Régis Saladin, Piet De Vos, Bart Staels & Johan Auwerx - INSERM U177, Institut des Cordeliers, 75006, Paris, France
Michele Guerre-Millot - Ceremod CNRS UPR 1511, 92190, Meudon, France
Armelle Leturque & Jean Girard
Authors
- Régis Saladin
You can also search for this author inPubMed Google Scholar - Piet De Vos
You can also search for this author inPubMed Google Scholar - Michele Guerre-Millot
You can also search for this author inPubMed Google Scholar - Armelle Leturque
You can also search for this author inPubMed Google Scholar - Jean Girard
You can also search for this author inPubMed Google Scholar - Bart Staels
You can also search for this author inPubMed Google Scholar - Johan Auwerx
You can also search for this author inPubMed Google Scholar
Rights and permissions
About this article
Cite this article
Saladin, R., De Vos, P., Guerre-Millot, M. et al. Transient increase in obese gene expression after food intake or insulin administration.Nature 377, 527–528 (1995). https://doi.org/10.1038/377527a0
- Received: 14 August 1995
- Accepted: 07 September 1995
- Issue Date: 12 October 1995
- DOI: https://doi.org/10.1038/377527a0