Transgenic expression of green fluorescence protein can cause dilated cardiomyopathy (original) (raw)
- Letters to the Editor
- Published: May 2000
Nature Medicine volume 6, pages 482–483 (2000)Cite this article
- 1924 Accesses
- 3 Altmetric
- Metrics details
To the editor
A common approach for determining the molecular mechanisms of cardiac hypertrophy and failure has been the use of transgenic mice1. Many transgenic models of dilated cardiac hypertrophy have been reported2. In these models, the non-transgenic littermates are generally used as controls. However, because non-specific effects of a transgene can be dose- and time-dependent, non-transgenic littermates might not be a sufficient control for transgenic experiments.
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 12 print issues and online access
$209.00 per year
only $17.42 per issue
Buy this article
- Purchase on SpringerLink
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Additional access options:
Figure 1: Confirmation of GFP expression by western blot analysis, and cardiac pathology.
References
- James, J.F., Hewett T.E. & Robbins, J. Cardiac physiology in transgenic mice. Circ. Res. 82, 407–415 ( 1998).
Article CAS Google Scholar - Kadambi, V. I. & Kranias, E. G. Genetically engineered mice: model systems for left ventricular failure. J. Card. Fail. 4, 349–361 (1998).
Article CAS Google Scholar - Heim, R. Prasher, D.C. & Tsien, R.Y. Wavelength mutations and posttranslational autoxidation of green fluorescence protein. Proc. Natl. Acad. Sci. USA 91, 12501–12504 (1994).
Article CAS Google Scholar - Godwin,A.R. et al. Detection of targeted GFP-Hox gene fusions during mouse embryogenesis . Proc. Natl. Acad. Sci. USA 95, 13042– 13047 (1998).
Article CAS Google Scholar - Subramaniam, A. et al. Tissue-specific regulation of the alpha-myosin heavy chain gene promoter in transgenic mice. J. Biol. Chem. 266 , 24613–24620 (1991).
CAS PubMed Google Scholar - Okabe, M. et al. Green mice as a source of ubiquitous green cells. FEBS Lett. 407, 313–319 (1997).
Article CAS Google Scholar
Author information
Authors and Affiliations
- Cardiovascular division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, 02215, Massachusetts , USA
Weei-Yuarn Huang, Pamela S. Douglas & Seigo Izumo - The Center for Blood Research, 200 Longwood Avenue, Boston, 02215, Massachusetts, USA
Jose Aramburu
Authors
- Weei-Yuarn Huang
You can also search for this author inPubMed Google Scholar - Jose Aramburu
You can also search for this author inPubMed Google Scholar - Pamela S. Douglas
You can also search for this author inPubMed Google Scholar - Seigo Izumo
You can also search for this author inPubMed Google Scholar
Corresponding author
Correspondence toSeigo Izumo.
Rights and permissions
About this article
Cite this article
Huang, WY., Aramburu, J., Douglas, P. et al. Transgenic expression of green fluorescence protein can cause dilated cardiomyopathy.Nat Med 6, 482–483 (2000). https://doi.org/10.1038/74914
- Issue Date: May 2000
- DOI: https://doi.org/10.1038/74914