SNPs in BRAP associated with risk of myocardial infarction in Asian populations (original) (raw)
Accession codes
Accessions
NCBI Reference Sequence
References
- Ozaki, K. et al. Functional SNPs in the lymphotoxin-α gene that are associated with susceptibility to myocardial infarction. Nat. Genet. 32, 650–654 (2002).
Article CAS Google Scholar - Ozaki, K. et al. Functional variation in LGALS2 confers risk of myocardial infarction and regulates lymphotoxin-alpha secretion in vitro. Nature 429, 72–75 (2004).
Article CAS Google Scholar - PROCARDIS Consortium. A trio family study showing association of the lymphotoxin-alpha N26 (804A) allele with coronary artery disease. Eur. J. Hum. Genet. 12, 770–774 (2004).
- Clarke, R. et al. Lymphotoxin-alpha gene and risk of myocardial infarction in 6,928 cases and 2,712 controls in the ISIS case-control study. PLoS. Genet. 2, e107 (2006).
Article Google Scholar - Asselbergs, F.W. et al. Effects of lymphotoxin-alpha gene and galectin-2 gene polymorphisms on inflammatory biomarkers, cellular adhesion molecules and risk of coronary heart disease. Clin. Sci. 112, 291–298 (2007).
Article CAS Google Scholar - Mangino, M. et al. LGALS2 functional variant rs7291467 is not associated with susceptibility to myocardial infarction in Caucasians. Atherosclerosis 194, 112–115 (2007).
Article CAS Google Scholar - Rigaut, G. et al. A generic protein purification method for protein complex characterization and proteome exploration. Nat. Biotechnol. 17, 1030–1032 (1999).
Article CAS Google Scholar - Barrett, J.C., Fry, B., Maller, J. & Daly, M.J. Haploview: analysis and visualization of LD and haplotype maps. Bioinformatics 21, 263–265 (2005).
Article CAS Google Scholar - Tregouet, D.A. & Garelle, V. A new JAVA interface implementation of THESIAS: testing haplotype effects in association studies. Bioinformatics 23, 1038–1039 (2007).
Article CAS Google Scholar - International HapMap Consortium. A haplotype map of the human genome. Nature 437, 1299–1320 (2005).
- Wellcome Trust Case Control Consortium. Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls. Nature 447, 661–678 (2007).
- Cordell, H.J. & Clayton, D.G. A unified stepwise regression procedure for evaluating the relative effects of polymorphisms within a gene using case/control or family data: application to HLA in type 1 diabetes. Am. J. Hum. Genet. 70, 124–141 (2002).
Article CAS Google Scholar - Ozaki, K. et al. A functional SNP in PSMA6 confers risk of myocardial infarction in the Japanese population. Nat. Genet. 38, 921–925 (2006).
Article CAS Google Scholar - Heinemeyer, T. et al. Databases on transcriptional regulation: TRANSFAC, TRRD, and COMPEL. Nucleic Acids Res. 26, 362–367 (1998).
Article CAS Google Scholar - Li, S. et al. Identification of a novel cytoplasmic protein that specifically binds to nuclear localization signal motifs. J. Biol. Chem. 273, 6183–6189 (1998).
Article CAS Google Scholar - Matheny, S.A. et al. Ras regulates assembly of mitogenic signalling complexes through the effector protein IMP. Nature 427, 256–260 (2004).
Article CAS Google Scholar - Ory, S. & Morrison, D.K. Signal transduction: implications for Ras-dependent ERK signaling. Curr. Biol. 14, R277–R278 (2004).
Article CAS Google Scholar - O'Neill, L.A. Targeting signal transduction as a strategy to treat inflammatory diseases. Nat. Rev. Drug Discov. 5, 549–563 (2006).
Article CAS Google Scholar - Karin, M. & Delhase, M. The I kappa B kinase (IKK) and NF-kappa B: key elements of proinflammatory signalling. Semin. Immunol. 12, 85–98 (2000).
Article CAS Google Scholar - Breslow, J.L. Cardiovascular disease burden increases, NIH funding decreases. Nat. Med. 3, 600–601 (1997).
Article CAS Google Scholar - Mizuno, H. et al. Impact of atherosclerosis-related gene polymorphisms on mortality and recurrent events after myocardial infarction. Atherosclerosis 185, 400–405 (2006).
Article CAS Google Scholar - Andrews, N.C. & Faller, D.V. A rapid micropreparation teqhnique fore extraction of DNA-binding proteins from limiting numbers of mammalian cells. Nucleic Acids Res. 19, 2499 (1991).
Article CAS Google Scholar
Acknowledgements
We thank M. Takahashi, M. Yoshii, S. Abiko, W. Yamanobe, R. Ohishi, M. Watabe and T. Furusho for their assistance. We also thank all the other members of Center for Genomic Medicine, RIKEN and OACIS for their contribution to the completion of our study. We are also grateful to members of The Rotary Club of Osaka-Midosuji District 2660 Rotary International in Japan for supporting our study. This work was supported in part by grants from the Takeda Science Foundation, the Uehara Science Foundation, the Japanese Millennium Project and the Ministry of Education, Science, Sports, Culture and Technology of Japan and NHRI-Ex96-9607PI (Taiwan).
Author information
Authors and Affiliations
- Laboratory for Cardiovascular Diseases, Center for Genomic Medicine, RIKEN, Yokohama, 230-0045, Japan
Kouichi Ozaki, Asako Aoki, Yoshihiro Onouchi & Toshihiro Tanaka - Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine, Osaka, 565-0871, Japan
Hiroshi Sato, Yasuhiko Sakata, Hiroya Mizuno & Masatsugu Hori - Department of Cardiology, Kokura Memorial Hospital, Kitakyushu, 802-8555, Japan
Katsumi Inoue, Keita Odashiro & Masakiyo Nobuyoshi - Laboratory for Medical Informatics, Center for Genomic Medicine, RIKEN, Yokohama, 230-0045, Japan
Tatsuhiko Tsunoda - Division of Cardiology, Department of Internal Medicine, Kaohsiung Medical University Hospital, Kaohsiung, 807, Taiwan
Tsung-Hsien Lin & Sheng-Hsiung Sheu - Department of Internal Medicine, Kaohsiung Medical University, Kaohsiung, 807, Taiwan
Tsung-Hsien Lin & Sheng-Hsiung Sheu - Laboratory for Bone and Joint Disease, Center for Genomic Medicine, RIKEN, Tokyo, 108-8639, Japan
Yoshinari Miyamoto & Shiro Ikegawa - Department of Medical Research, Kaohsiung Medical University Hospital, Kaohsiung, 807, Taiwan
Suh-Hang H Juo - Graduate Institute of Medical Genetics, Kaohsiung Medical University, Kaohsiung, 807, Taiwan
Suh-Hang H Juo - Center of Excellence for Environmental Medicine, Kaohsiung Medical University, Kaohsiung, 807, Taiwan
Suh-Hang H Juo - Center for Genomic Medicine, RIKEN, Yokohama, 230-0045, Japan
Yusuke Nakamura
Authors
- Kouichi Ozaki
- Hiroshi Sato
- Katsumi Inoue
- Tatsuhiko Tsunoda
- Yasuhiko Sakata
- Hiroya Mizuno
- Tsung-Hsien Lin
- Yoshinari Miyamoto
- Asako Aoki
- Yoshihiro Onouchi
- Sheng-Hsiung Sheu
- Shiro Ikegawa
- Keita Odashiro
- Masakiyo Nobuyoshi
- Suh-Hang H Juo
- Masatsugu Hori
- Yusuke Nakamura
- Toshihiro Tanaka
Contributions
K. Ozaki performed most of the experiments and wrote the manuscript; H.S., Y.S., H.M., T.-H.L., Y.M., S.-H.S., S.I., S.-H.H.J. and M.H. managed DNA samples and clinical information; K.I., K. Odashiro and M.N. performed the immunohistochemistry; T. Tsunoda performed the statistical analyses; A.A. and Y.O. performed the data analyses; Y.N. contributed to SNP discovery and preparation of the manuscript; and T. Tanaka supervised this study.
Corresponding author
Correspondence toToshihiro Tanaka.
Supplementary information
Rights and permissions
About this article
Cite this article
Ozaki, K., Sato, H., Inoue, K. et al. SNPs in BRAP associated with risk of myocardial infarction in Asian populations.Nat Genet 41, 329–333 (2009). https://doi.org/10.1038/ng.326
- Received: 01 July 2008
- Accepted: 20 November 2008
- Published: 08 February 2009
- Issue date: March 2009
- DOI: https://doi.org/10.1038/ng.326