The role of a mutant CCR5 allele in HIV–1 transmission and disease progression (original) (raw)
- Article
- Published: 01 November 1996
- William A. Paxton1 na1,
- Steven M. Wolinsky2,
- Avidan U. Neumann3 nAff6,
- Linqi Zhang1,
- Tian He1,
- Stanley Kang1,
- Daniel Ceradini1,
- Zhanqun Jin1,
- Karina Yazdanbakhsh4,
- Kevin Kunstman2,
- Daniel Erickson2,
- Elizabeth Dragon5,
- Nathaniel R. Landau1,
- John Phair2,
- David D. Ho1 &
- …
- Richard A. Koup1
Nature Medicine volume 2, pages 1240–1243 (1996)Cite this article
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Abstract
A 32–nucleotide deletion (δ32) within the β–chemokine receptor 5 (CCR5) gene has been described in subjects who remain uninfected despite extensive exposure to HIV–1. This allele was found to be common in the Caucasian population with a frequency of 0.0808, but was not found in people of African or Asian ancestry. To determine its role in HIV–1 transmission and disease progression, we analyzed the CCR5 genotype of 1252 homosexual men enrolled in the Chicago component of the Multicenter AIDS Cohort Study (MACS). No infected participant was found to be homozygous for the 32 allele, whereas 3.6% of at–risk but uninfected Caucasian participants were homozygous, showing the highly protective role of this genotype against sexual acquisition of HIV–1. No evidence was found to suggest that heterozygotes were protected against HIV–1 infection, but a limited protective role against disease progression was noted. The 32 allele of CCR5 is therefore an important host factor in HIV–1 transmission and pathogenesis.
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Author notes
- Avidan U. Neumann
Present address: Human Biology Research Center, Hadassah Hospital, Jerusalem, 91120, Israel - Yaoxing Huang and William A. Paxton: These two authors contributed equally to this work.
Authors and Affiliations
- The Aaron Diamond AIDS Research Center and The Rockefeller University, 455 First Avenue, New York, New York, 10016, USA
Yaoxing Huang, William A. Paxton, Linqi Zhang, Tian He, Stanley Kang, Daniel Ceradini, Zhanqun Jin, Nathaniel R. Landau, David D. Ho & Richard A. Koup - Department of Medicine, Northwestern University Medical School, 303 East Chicago Avenue, Chicago, Illinois, 60611, USA
Steven M. Wolinsky, Kevin Kunstman, Daniel Erickson & John Phair - Theoretical Division, T-10, Los Alamos National Laboratories, Los Alamos, New Mexico, 87545, USA
Avidan U. Neumann - Laboratory of Immunochemistry, The Lindsley F. Kimball Research Institute of the New York Blood Center, New York, New York, 10021, USA
Karina Yazdanbakhsh - Roche Molecular Systems, Branchburg, New Jersey, 08876, USA
Elizabeth Dragon
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Huang, Y., Paxton, W., Wolinsky, S. et al. The role of a mutant CCR5 allele in HIV–1 transmission and disease progression.Nat Med 2, 1240–1243 (1996). https://doi.org/10.1038/nm1196-1240
- Received: 03 October 1996
- Accepted: 07 October 1996
- Issue Date: 01 November 1996
- DOI: https://doi.org/10.1038/nm1196-1240