T-cell-mediated cytotoxicity toward platelets in chronic idiopathic thrombocytopenic purpura (original) (raw)
- Brief Communication
- Published: 24 August 2003
- Per-Ola Andersson2 na1,
- Margareta Jernås1,
- Stefan Jacobsson3,
- Björn Carlsson1,
- Lena M S Carlsson1 &
- …
- Hans Wadenvik2
Nature Medicine volume 9, pages 1123–1124 (2003)Cite this article
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Abstract
Chronic idiopathic thrombocytopenic purpura (ITP) is a bleeding disorder that is characterized by increased platelet destruction and is believed to be autoantibody mediated. In this study, CD3+ T cells from ITP patients had increased expression of genes involved in cell-mediated cytotoxicity. In addition, cytotoxic cell-mediated lysis of autologous platelets was shown in active ITP. Our data suggest that T-cell-mediated cytotoxicity is an alternative mechanism for platelet destruction in ITP.
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Acknowledgements
Grants were from the Swedish Medical Research Council (projects no. 11630, 11285 and 13141) and the IngaBritt and Arne Lundberg Forskningsstiftelse.
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- Bob Olsson and Per-Ola Andersson: These authors contributed equally to this work.
Authors and Affiliations
- Research Center for Endocrinology and Metabolism, Sahlgrenska University Hospital, Göteborg University, Göteborg, SE-413 45, Sweden
Bob Olsson, Margareta Jernås, Björn Carlsson & Lena M S Carlsson - Department of Internal Medicine, Section of Hematology and Coagulation, Sahlgrenska University Hospital, Göteborg University, Göteborg, SE-413 45, Sweden
Per-Ola Andersson & Hans Wadenvik - Department of Clinical Chemistry and Transfusion Medicine, Sahlgrenska University Hospital, Göteborg University, Göteborg, SE-413 45, Sweden
Stefan Jacobsson
Authors
- Bob Olsson
- Per-Ola Andersson
- Margareta Jernås
- Stefan Jacobsson
- Björn Carlsson
- Lena M S Carlsson
- Hans Wadenvik
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Correspondence toPer-Ola Andersson.
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The authors declare no competing financial interests.
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Olsson, B., Andersson, PO., Jernås, M. et al. T-cell-mediated cytotoxicity toward platelets in chronic idiopathic thrombocytopenic purpura.Nat Med 9, 1123–1124 (2003). https://doi.org/10.1038/nm921
- Received: 19 March 2003
- Accepted: 04 August 2003
- Published: 24 August 2003
- Issue date: 01 September 2003
- DOI: https://doi.org/10.1038/nm921