Phenotypic knock-out of the latent membrane protein 1 of Epstein–Barr virus by an intracellular single-chain antibody (original) (raw)
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- Published: 03 September 1998
Gene Therapy volume 5, pages 1171–1179 (1998)Cite this article
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Abstract
Epstein–Barr virus (EBV) causes lymphoproliferative diseases in immunocompromised patients and is associated with endemic Burkitt lymphoma, nasopharyngeal carcinoma and some cases of Hodgkin disease. The latent membrane protein 1 (LMP1) of EBV is a transmembrane protein that is essential for the transformation of B lymphocytes. LMP1-mediated up-regulation of Bcl-2 is thought to be an important element in this process. As an approach to explore novel treatments for EBV-associated lymphomas, we constructed a single-chain antibody (sFv) directed against LMP1 to achieve functional inhibition of this oncoprotein in EBV-transformed B lymphocytes. We demonstrated that intracellular expression of an endoplasmic reticulum (ER)-targeted form of this sFv markedly reduced LMP1 protein levels. We also observed a decrease in intracellular level of this protein which correlated with a marked reduction of Bcl-2 expression in EBV-transformed B lymphocytes. We further demonstrated that anti-LMP1 sFv-mediated reduction of Bcl-2 correlated with increased sensitivity of these cells to drug-induced cell death. Therefore, these data suggest that an anti-LMP1 sFv used in combination with conventional chemotherapy may be useful for gene therapy of EBV-associated lymphomas in immunocompromised patients.
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Authors and Affiliations
- Gene Therapy Program, Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA
A Piché, K Kasono, F Johanning & DT Curiel - Divisions of Infectious Diseases and Medical Oncology, University of Colorado Health Sciences Center, Denver, CO, USA
TJ Curiel
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- A Piché
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Piché, A., Kasono, K., Johanning, F. et al. Phenotypic knock-out of the latent membrane protein 1 of Epstein–Barr virus by an intracellular single-chain antibody.Gene Ther 5, 1171–1179 (1998). https://doi.org/10.1038/sj.gt.3300706
- Received: 11 November 1997
- Accepted: 23 March 1998
- Published: 03 September 1998
- Issue Date: 01 September 1998
- DOI: https://doi.org/10.1038/sj.gt.3300706
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