Leukotriene C4 affects pulmonary and cardiovascular dynamics in monkey (original) (raw)
- Letter
- Published: 28 January 1982
- Per Hedqvist3,
- Sven-Erik Dahlén3,
- Björn Revenäs1,
- Sven Hammarström4 &
- …
- Bengt Samuelsson4
Nature volume 295, pages 327–329 (1982)Cite this article
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Abstract
Leukotriene C4 (LTC4) and its metabolites LTD4 and LTE4 (Fig. 1) have been identified as the major active constituents of slow-reacting substance of anaphylaxis (SRS-A)1–3. Leukotrienes C4, D4 and E4 have rapidly gained recognition as bronchoconstrictors of hitherto unsurpassed potency in the guinea pig in vivo and _in vitro_4–7 as well as acting as oedema-forming substances in guinea pig3,5,7 and hamster8. Here we report that LTC4 is also a prominent bronchoconstrictor in the monkey; it increases transpulmonary pressure in the anaesthetized animal, mainly by decreasing pulmonary dynamic compliance, and it contracts isolated tracheal spiral tissue. Furthermore, LTC4 causes transient pulmonary and systemic hypertension, followed by a prolonged hypotensive period associated with decreased cardiac output, haemoconcentration and reduction in the number of circulating leukocytes. These in vivo findings in a primate imply that leukotrienes may also cause important alterations of cardiovascular and pulmonary function in man.
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Authors and Affiliations
- Department of Experimental Medicine, Pharmacia AB, Uppsala, Sweden
Göran Smedegård & Björn Revenäs - Department of Experimental Allergy Research, Pharmacia AB, Uppsala, Sweden
Göran Smedegård - Department of Physiology, Karolinska Institutet, Stockholm, Sweden
Per Hedqvist & Sven-Erik Dahlén - Department of Chemistry, Karolinska Institutet, Stockholm, Sweden
Sven Hammarström & Bengt Samuelsson
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- Göran Smedegård
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Smedegård, G., Hedqvist, P., Dahlén, SE. et al. Leukotriene C4 affects pulmonary and cardiovascular dynamics in monkey.Nature 295, 327–329 (1982). https://doi.org/10.1038/295327a0
- Received: 10 September 1981
- Accepted: 13 November 1981
- Issue Date: 28 January 1982
- DOI: https://doi.org/10.1038/295327a0