Cyclin A and the retinoblastoma gene product complex with a common transcription factor (original) (raw)
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- Published: 18 July 1991
Nature volume 352, pages 249–251 (1991)Cite this article
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Abstract
THE retinoblastoma gene (Rb) product is a negative regulator of cellular proliferation1, an effect that could be mediated in part at the transcriptional level through its ability to complex with the sequence-specific transcription factor DRTF1 (ref. 2). This interaction is modulated by adenovirus El a, which sequesters the Rb protein3 and several other cellular proteins<3>, including cyclin A (refs 4, 5), a molecule that undergoes cyclical accumulation and destruction during each cell cycle6, 7 and which is required for cell cycle progression8. Cyclin A, which also complexes with DRTF1, facilitates the efficient assembly of the Rb protein into the complex. This suggests a role for cyclin A in regulating transcription and defines a transcription factor through which molecules that regulate the cell cycle in a negative fashion, such as Rb, and in a positive fashion, such as cyclin A, interact. Mutant loss-of-function Rb alleles, which occur in a variety of tumour cells, also fail to complex with Ela and large T antigen9, 10. Here we report on a naturally occurring loss-of-function Rb allele encoding a protein that fails to complex with DRTF1. This might explain how mutation in the Rb gene prevents negative growth control.
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Authors and Affiliations
- Laboratory of Eukaryotic Molecular Genetics, MRC National Institute for Medical Research, The Ridgeway, Mill Hill, London, NW7 1AA, UK
Lasantha R. Bandara, Jörg P. Adamczewski, Tim Hunt & Nicholas B. La Thangue - Imperial Cancer Research Fund, Clare Hall Laboratories, Blanche Lane, South Mimms, Potters Bar, Herts, EN6 3LD, UK
Jörg P. Adamczewski & Tim Hunt
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- Lasantha R. Bandara
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Bandara, L., Adamczewski, J., Hunt, T. et al. Cyclin A and the retinoblastoma gene product complex with a common transcription factor.Nature 352, 249–251 (1991). https://doi.org/10.1038/352249a0
- Received: 05 June 1991
- Accepted: 27 June 1991
- Issue Date: 18 July 1991
- DOI: https://doi.org/10.1038/352249a0