Small-conductance, Ca2+-activated K+ channel SK3 generates age-related memory and LTP deficits (original) (raw)
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- Published: 27 July 2003
Nature Neuroscience volume 6, pages 911–912 (2003)Cite this article
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Abstract
Cognitive deficits are among the most devastating changes associated with the aging process. Age-related decrement in performance on learning tasks1,2 is correlated with substantial changes in neuronal signal processing in the hippocampus3,4,5. Here we show that elevated expression of small-conductance Ca2+-activated K+ channels (SK channels) of the SK3 type in hippocampi of aged mice contributes to reduced long-term potentiation (LTP) and impaired trace fear conditioning, a hippocampus-dependent learning task6,7.
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Acknowledgements
This work was supported by the Max Planck Society. We thank Dr. John Disterhoft for fruitful discussions and his generous support in obtaining the animals for this study.
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Authors and Affiliations
- Department of Molecular Neuroendocrinology, Max Planck Institute for Experimental Medicine, Goettingen, D-37075, Germany
Thomas Blank, Ingrid Nijholt, Min-Jeong Kye, Jelena Radulovic & Joachim Spiess
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- Thomas Blank
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Correspondence toThomas Blank.
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Blank, T., Nijholt, I., Kye, MJ. et al. Small-conductance, Ca2+-activated K+ channel SK3 generates age-related memory and LTP deficits.Nat Neurosci 6, 911–912 (2003). https://doi.org/10.1038/nn1101
- Received: 16 June 2003
- Accepted: 07 July 2003
- Published: 27 July 2003
- Issue Date: 01 September 2003
- DOI: https://doi.org/10.1038/nn1101