Spectrum of mutations in the Fanconi anaemia group G gene, FANCG/XRCC9 (original) (raw)
- Article
- Published: 31 October 2000
- Marcin Wlodarski1,
- Alex J Tipping2,
- Neil V Morgan2,
- Johan P de Winter3,
- Michaela Thiel4,
- Sonja Gräsl4,
- Detlev Schindler4,
- Alan D D'Andrea5,
- Cigdem Altay6,
- Hülya Kayserili7,
- Adriana Zatterale8,
- Jürgen Kunze1,
- Wolfram Ebell9,
- Christopher G Mathew2,
- Hans Joenje3,
- Karl Sperling1 &
- …
- Martin Digweed1
European Journal of Human Genetics volume 8, pages 861–868 (2000)Cite this article
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Abstract
FANCG was the third Faconi anaemia gene identified and proved to be identical to the previously cloned XRCC9 gene. We present the pathogenic mutations and sequence variants we have so far identified in a panel of FA-G patients. Mutation screening was performed by PCR, single strand conformational polymorphism analysis and protein truncation tests. Altogether 18 mutations have been determined in 20 families – 97% of all expected mutant alleles. All mutation types have been found, with the exception of large deletions, the large majority is predicted to lead to shortened proteins. One stop codon mutation, E105X, has been found in several German patients and this founder mutation accounts for 44% of the mutant FANCG alleles in German FA-G patients. Comparison of clinical phenotypes shows that patients homozygous for this mutation have an earlier onset of the haematological disorder than most other FA-G patients. The mouse Fancg sequence was established in order to evaluate missense mutations. A putative missense mutation, L71P, in a possible leucine zipper motif may affect FANCG binding of FANCA and seems to be associated with a milder clinical phenotype.
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Authors and Affiliations
- Institute of Human Genetics, Charité, Campus Virchow, Humboldt University, Berlin, Germany
Ilja Demuth, Marcin Wlodarski, Jürgen Kunze, Karl Sperling & Martin Digweed - Division of Medical and Molecular Genetics, Guy's, King's and St Thomas' School of Medicine, London, UK
Alex J Tipping, Neil V Morgan & Christopher G Mathew - Department of Clinical Genetics and Human Genetics, Free University Medical Center, Amsterdam, The Netherlands
Johan P de Winter & Hans Joenje - Institute of Human Genetics, University of Würzburg, Germany
Michaela Thiel, Sonja Gräsl & Detlev Schindler - Department of Pediatric Oncology, Dana-Faber Cancer Institute, Boston, USA
Alan D D'Andrea - Department of Pediatrics, Hematology Unit, Hacettepe University, Ankara
Cigdem Altay - Institute of Child Health, Istanbul, Turkey
Hülya Kayserili - Servizio di Citogenetica, Ospetale Elena d'Aosta, Napoli, Italy
Adriana Zatterale - Children's Hospital, Charité, Campus Virchow, Humboldt University, Berlin, Germany
Wolfram Ebell
Authors
- Ilja Demuth
- Marcin Wlodarski
- Alex J Tipping
- Neil V Morgan
- Johan P de Winter
- Michaela Thiel
- Sonja Gräsl
- Detlev Schindler
- Alan D D'Andrea
- Cigdem Altay
- Hülya Kayserili
- Adriana Zatterale
- Jürgen Kunze
- Wolfram Ebell
- Christopher G Mathew
- Hans Joenje
- Karl Sperling
- Martin Digweed
Corresponding author
Correspondence toMartin Digweed.
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Demuth, I., Wlodarski, M., Tipping, A. et al. Spectrum of mutations in the Fanconi anaemia group G gene, FANCG/XRCC9.Eur J Hum Genet 8, 861–868 (2000). https://doi.org/10.1038/sj.ejhg.5200552
- Received: 24 March 2000
- Revised: 06 July 2000
- Accepted: 11 July 2000
- Published: 31 October 2000
- Issue date: 01 November 2000
- DOI: https://doi.org/10.1038/sj.ejhg.5200552