The S. cerevisiae SAGA complex functions in vivo as a coactivator for transcriptional activation by Gal4 (original) (raw)
- Erica Larschan and
- Fred Winston1
- Department of Genetics, Harvard Medical School, Boston, Massachusetts 02115, USA
Abstract
Previous studies demonstrated that the SAGA (Spt-Ada-Gcn5-Acetyltransferase) complex facilitates the binding of TATA-binding protein (TBP) during transcriptional activation of the GAL1 gene of_Saccharomyces cerevisiae_. TBP binding was shown to require the SAGA components Spt3 and Spt20/Ada5, but not the SAGA component Gcn5. We have now examined whether SAGA is directly required as a coactivator in vivo by using chromatin immunoprecipitation analysis. Our results demonstrate that SAGA is physically recruited in vivo to the upstream activation sequence (UAS) regions of the galactose-inducible_GAL_ genes. This recruitment is dependent on both induction by galactose and the Gal4 activation domain. Furthermore, we demonstrate that another well-characterized activator, Gal4–VP16, also recruits SAGA in vivo. Finally, we provide evidence that a specific interaction between Spt3 and TBP in vivo is important for Gal4 transcriptional activation at a step after SAGA recruitment. These results, taken together with previous studies, demonstrate a dependent pathway for the recruitment of TBP to GAL gene promoters consisting of the recruitment of SAGA by Gal4 and the subsequent recruitment of TBP by SAGA.
Footnotes
↵1 Corresponding author.
E-MAIL Winston{at}rascal.med.Harvard.edu; FAX (617) 432-3993.
Article and publication are at http://www.genesdev.org/cgi/doi/10.1101/gad.911501.
- Received May 15, 2001.
- Accepted June 14, 2001.
Cold Spring Harbor Laboratory Press