tRNAHis maturation: An essential yeast protein catalyzes addition of a guanine nucleotide to the 5′ end of tRNAHis (original) (raw)
- Weifeng Gu1,
- Jane E. Jackman1,
- Amanda J. Lohan2,
- Michael W. Gray2, and
- Eric M. Phizicky1,3
- 1 Department of Biochemistry and Biophysics, University of Rochester School of Medicine, Rochester, New York 14642, USA
- 2 Department of Biochemistry and Molecular Biology, Dalhousie University, Halifax NS, B3H 1X5, Canada
Abstract
All tRNAHis molecules are unusual in having an extra 5′ GMP residue (G-1) that, in eukaryotes, is added after transcription and RNase P cleavage. Incorporation of this G-1 residue is a rare example of nucleotide addition occurring at an RNA 5′ end in a normal phosphodiester linkage. We show here that the essential Saccharomyces cerevisiae ORF YGR024c (THG1) is responsible for this guanylyltransferase reaction. Thg1p was identified by survey of a genomic collection of yeast GST-ORF fusion proteins for addition of [α-32P]GTP to tRNAHis. End analysis confirms the presence of G-1. Thg1p is required for tRNAHis guanylylation in vivo, because cells depleted of Thg1p lack G-1 in their tRNAHis.His6-Thg1p purified from Escherichia coli catalyzes the guanylyltransferase step of G-1 addition using a ppp-tRNAHis substrate, and appears to catalyze the activation step using p-tRNAHis and ATP. Thg1p is highly conserved in eukaryotes, where G-1 addition is necessary, and is not found in eubacteria, where G-1 is genome-encoded. Thus, Thg1p is the first member of a new family of enzymes that can catalyze phosphodiester bond formation at the 5′ end of RNAs, formally in a 3′-5′ direction. Surprisingly, despite its varied activities, Thg1p contains no recognizable catalytic or functional domains.
Footnotes
Supplemental material is available at http://www.genesdev.org.
Article published online ahead of print. Article and publication date are at http://www.genesdev.org/cgi/doi/10.1101/gad.1148603.
↵3 Corresponding author. E-MAIL eric_phizicky{at}urmc.rochester.edu; FAX (585) 271-2683.
- Accepted October 15, 2003.
- Received August 29, 2003.
Cold Spring Harbor Laboratory Press