Reduction of central nervous system ischemic injury by monoclonal antibody to intercellular adhesion molecule (original) (raw)

• 1. Ames A, , Wright L, & Masayoshi K, et al: Cerebral ischemia. The no-reflow phenomenon. Am J Pathol 52:437–443, 1968 Ames A, Wright L, Masayoshi K, et al: Cerebral ischemia. The no-reflow phenomenon. **Am J Pathol 52:**437–443, 1968
• PubMed
  Ames A, Wright L, Masayoshi K, et al: Cerebral ischemia. The no-reflow phenomenon. Am J Pathol 52: 437– 443, 1968 Ames A, Wright L, Masayoshi K, et al: Cerebral ischemia. The no-reflow phenomenon. Am J Pathol 52:437–443, 1968
  )| false
• Search Google Scholar
• Export Citation
• 1. Arfors K, , Lundberg C, & Lindbom L, et al: A monoclonal antibody to the membrane glycoprotein complex CD 18 inhibits PMN accumulation and plasma leakage in vivo. Blood 69:338–340, 1987 Arfors K, Lundberg C, Lindbom L, et al: A monoclonal antibody to the membrane glycoprotein complex CD 18 inhibits PMN accumulation and plasma leakage in vivo. **Blood 69:**338–340, 1987
• PubMed
  Arfors K, Lundberg C, Lindbom L, et al: A monoclonal antibody to the membrane glycoprotein complex CD 18 inhibits PMN accumulation and plasma leakage in vivo. Blood 69: 338– 340, 1987 Arfors K, Lundberg C, Lindbom L, et al: A monoclonal antibody to the membrane glycoprotein complex CD 18 inhibits PMN accumulation and plasma leakage in vivo. Blood 69:338–340, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Barton R, , Rothlein R, & Ksiazek J, et al: The effect of anti-intercellular adhesion molecule-1 on phorbol ester induced rabbit lung inflammation. J Immunol 142:1278–1282, 1989 Barton R, Rothlein R, Ksiazek J, et al: The effect of anti-intercellular adhesion molecule-1 on phorbol ester induced rabbit lung inflammation. **J Immunol 142:**1278–1282, 1989
• PubMed
  Barton R, Rothlein R, Ksiazek J, et al: The effect of anti-intercellular adhesion molecule-1 on phorbol ester induced rabbit lung inflammation. J Immunol 142: 1278– 1282, 1989 Barton R, Rothlein R, Ksiazek J, et al: The effect of anti-intercellular adhesion molecule-1 on phorbol ester induced rabbit lung inflammation. J Immunol 142:1278–1282, 1989
  )| false
• Search Google Scholar
• Export Citation
• 1. Clark W, , Madden K, & Rothlein R, et al: Reduction of central nervous system ischemic injury in rabbits using leukocyte adhesion antibody treatment. Stroke 22:877–883, 1991 Clark W, Madden K, Rothlein R, et al: Reduction of central nervous system ischemic injury in rabbits using leukocyte adhesion antibody treatment. **Stroke 22:**877–883, 1991
• PubMed
  Clark W, Madden K, Rothlein R, et al: Reduction of central nervous system ischemic injury in rabbits using leukocyte adhesion antibody treatment. Stroke 22: 877– 883, 1991 Clark W, Madden K, Rothlein R, et al: Reduction of central nervous system ischemic injury in rabbits using leukocyte adhesion antibody treatment. Stroke 22:877–883, 1991
  )| false
• Search Google Scholar
• Export Citation
• 1. Cosimi A, , Conti D, & Delmonico F, et al: In vivo effects of monoclonal antibody to ICAM in nonhuman primates with renal allografts. J Immunol 144:4604–4612, 1990 Cosimi A, Conti D, Delmonico F, et al: In vivo effects of monoclonal antibody to ICAM in nonhuman primates with renal allografts. **J Immunol 144:**4604–4612, 1990
• PubMed
  Cosimi A, Conti D, Delmonico F, et al: In vivo effects of monoclonal antibody to ICAM in nonhuman primates with renal allografts. J Immunol 144: 4604– 4612, 1990 Cosimi A, Conti D, Delmonico F, et al: In vivo effects of monoclonal antibody to ICAM in nonhuman primates with renal allografts. J Immunol 144:4604–4612, 1990
  )| false
• Search Google Scholar
• Export Citation
• 1. Dustin M, , Rothlein R, & Bhan C, et al: Induction by IL 1 and interferon gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM). J Immunol 137:245, 1986 Dustin M, Rothlein R, Bhan C, et al: Induction by IL 1 and interferon gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM). **J Immunol 137:**245, 1986 (Abstract)
• PubMed
  Dustin M, Rothlein R, Bhan C, et al: Induction by IL 1 and interferon gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM). J Immunol 137: 245, 1986 (Abstract) Dustin M, Rothlein R, Bhan C, et al: Induction by IL 1 and interferon gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM). J Immunol 137:245, 1986 (Abstract)
  )| false
• Search Google Scholar
• Export Citation
• 1. Engler R: Consequences of activation and adenosine mediated inhibition of granulocytes during myocardial ischemia. Fed Proc 46:2407–2412, 1987 Engler R: Consequences of activation and adenosine mediated inhibition of granulocytes during myocardial ischemia. **Fed Proc 46:**2407–2412, 1987
• PubMed
  Engler R: Consequences of activation and adenosine mediated inhibition of granulocytes during myocardial ischemia. Fed Proc 46: 2407– 2412, 1987 Engler R: Consequences of activation and adenosine mediated inhibition of granulocytes during myocardial ischemia. Fed Proc 46:2407–2412, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Engler R, , Dahlgren M, & Morris D, et al: Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. Am J Physiol 251:H314–H322, 1986 Engler R, Dahlgren M, Morris D, et al: Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. **Am J Physiol 251:**H314–H322, 1986
• PubMed
  Engler R, Dahlgren M, Morris D, et al: Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. Am J Physiol 251: H314– H322, 1986 Engler R, Dahlgren M, Morris D, et al: Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. Am J Physiol 251:H314–H322, 1986
  )| false
• Search Google Scholar
• Export Citation
• 1. Hallenbeck J, , Dutka A, & Tanishima T, et al: Polymorphonuclear leukocyte accumulation in brain regions with low blood flow during the early postischemic period. Stroke 17:246–253, 1986 Hallenbeck J, Dutka A, Tanishima T, et al: Polymorphonuclear leukocyte accumulation in brain regions with low blood flow during the early postischemic period. **Stroke 17:**246–253, 1986
• PubMed
  Hallenbeck J, Dutka A, Tanishima T, et al: Polymorphonuclear leukocyte accumulation in brain regions with low blood flow during the early postischemic period. Stroke 17: 246– 253, 1986 Hallenbeck J, Dutka A, Tanishima T, et al: Polymorphonuclear leukocyte accumulation in brain regions with low blood flow during the early postischemic period. Stroke 17:246–253, 1986
  )| false
• Search Google Scholar
• Export Citation
• 1. Harlan J: Leukocyte-endothelial interactions. J Am Soc Hematol 65:513–525, 1985 Harlan J: Leukocyte-endothelial interactions. **J Am Soc Hematol 65:**513–525, 1985
• PubMed
  Harlan J: Leukocyte-endothelial interactions. J Am Soc Hematol 65: 513– 525, 1985 Harlan J: Leukocyte-endothelial interactions. J Am Soc Hematol 65:513–525, 1985
  )| false
• Search Google Scholar
• Export Citation
• 1. Harlan J: Neutrophil mediated vascular injury. Acta Med Scand 715:123–129, 1985 Harlan J: Neutrophil mediated vascular injury. **Acta Med Scand 715:**123–129, 1985
• PubMed
  Harlan J: Neutrophil mediated vascular injury. Acta Med Scand 715: 123– 129, 1985 Harlan J: Neutrophil mediated vascular injury. Acta Med Scand 715:123–129, 1985
  )| false
• Search Google Scholar
• Export Citation
• 1. Hernandez L, , Grisham M, & Twohig B, et al: Role of neutrophils in ischemia reperfusion induced microvascular injury. Am J Physiol 253:H699–H703, 1987 Hernandez L, Grisham M, Twohig B, et al: Role of neutrophils in ischemia reperfusion induced microvascular injury. **Am J Physiol 253:**H699–H703, 1987
• PubMed
  Hernandez L, Grisham M, Twohig B, et al: Role of neutrophils in ischemia reperfusion induced microvascular injury. Am J Physiol 253: H699– H703, 1987 Hernandez L, Grisham M, Twohig B, et al: Role of neutrophils in ischemia reperfusion induced microvascular injury. Am J Physiol 253:H699–H703, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Jacobs T, , Shohami E, & Baze W, et al: Deteriorating stroke model: histopathology, edema, and eicosanoid changes following spinal cord ischemia in rabbits. Stroke 18:741–750, 1987 Jacobs T, Shohami E, Baze W, et al: Deteriorating stroke model: histopathology, edema, and eicosanoid changes following spinal cord ischemia in rabbits. **Stroke 18:**741–750, 1987
• PubMed
  Jacobs T, Shohami E, Baze W, et al: Deteriorating stroke model: histopathology, edema, and eicosanoid changes following spinal cord ischemia in rabbits. Stroke 18: 741– 750, 1987 Jacobs T, Shohami E, Baze W, et al: Deteriorating stroke model: histopathology, edema, and eicosanoid changes following spinal cord ischemia in rabbits. Stroke 18:741–750, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Janoff A, & Zweifach B: Production of inflammatory changes in the microcirculation by cationic proteins extracted from lysosomes. J Exp Med 120:747–764, 1964 Janoff A, Zweifach B: Production of inflammatory changes in the microcirculation by cationic proteins extracted from lysosomes. **J Exp Med 120:**747–764, 1964
• PubMed
  Janoff A, Zweifach B: Production of inflammatory changes in the microcirculation by cationic proteins extracted from lysosomes. J Exp Med 120: 747– 764, 1964 Janoff A, Zweifach B: Production of inflammatory changes in the microcirculation by cationic proteins extracted from lysosomes. J Exp Med 120:747–764, 1964
  )| false
• Search Google Scholar
• Export Citation
• 1. Lyden P, , Zivin J, & Kochhar A, et al: Effects of calcium channel blockers on neurologic outcome after focal ischemia in rabbits. Stroke 19:1020–1025, 1988 Lyden P, Zivin J, Kochhar A. et al: Effects of calcium channel blockers on neurologic outcome after focal ischemia in rabbits. **Stroke 19:**1020–1025, 1988
• PubMed
  Lyden P, Zivin J, Kochhar A, et al: Effects of calcium channel blockers on neurologic outcome after focal ischemia in rabbits. Stroke 19: 1020– 1025, 1988 Lyden P, Zivin J, Kochhar A. et al: Effects of calcium channel blockers on neurologic outcome after focal ischemia in rabbits. Stroke 19:1020–1025, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Mehta J, & Nichols W: Neutrophils as potential participants in acute myocardial ischemia: relevance to reperfusion. J Am Coll Cardiol 11:1309–1316, 1988 Mehta J, Nichols W: Neutrophils as potential participants in acute myocardial ischemia: relevance to reperfusion. **J Am Coll Cardiol 11:**1309–1316, 1988
• PubMed
  Mehta J, Nichols W: Neutrophils as potential participants in acute myocardial ischemia: relevance to reperfusion. J Am Coll Cardiol 11: 1309– 1316, 1988 Mehta J, Nichols W: Neutrophils as potential participants in acute myocardial ischemia: relevance to reperfusion. J Am Coll Cardiol 11:1309–1316, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Price T, , Beatty P, & Corpuz S: In vivo inhibition of neutrophil function in the rabbit using monoclonal antibody to CD 18. J Immunol 139:4174–4177, 1987 Price T, Beatty P, Corpuz S: In vivo inhibition of neutrophil function in the rabbit using monoclonal antibody to CD 18. **J Immunol 139:**4174–4177, 1987
• PubMed
  Price T, Beatty P, Corpuz S: In vivo inhibition of neutrophil function in the rabbit using monoclonal antibody to CD 18. J Immunol 139: 4174– 4177, 1987 Price T, Beatty P, Corpuz S: In vivo inhibition of neutrophil function in the rabbit using monoclonal antibody to CD 18. J Immunol 139:4174–4177, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Romson J, , Hook B, & Lucchesi B: Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog. Circulation 67:1016–1023, 1983 Romson J, Hook B, Lucchesi B: Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog. **Circulation 67:**1016–1023, 1983
• PubMed
  Romson J, Hook B, Lucchesi B: Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog. Circulation 67: 1016– 1023, 1983 Romson J, Hook B, Lucchesi B: Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog. Circulation 67:1016–1023, 1983
  )| false
• Search Google Scholar
• Export Citation
• 1. Rothlein R, , Czajkowski M, & O'Neill M, et al: Induction of ICAM on primary and continuous cell lines by proinflammatory cytokines. J Immunol 141:1665–1669, 1988 Rothlein R, Czajkowski M, O'Neill M, et al: Induction of ICAM on primary and continuous cell lines by proinflammatory cytokines. **J Immunol 141:**1665–1669, 1988
• PubMed
  Rothlein R, Czajkowski M, O'Neill M, et al: Induction of ICAM on primary and continuous cell lines by proinflammatory cytokines. J Immunol 141: 1665– 1669, 1988 Rothlein R, Czajkowski M, O'Neill M, et al: Induction of ICAM on primary and continuous cell lines by proinflammatory cytokines. J Immunol 141:1665–1669, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Schmid-Schonbein G: Capillary plugging by granulocytes and the no-reflow phenomenon in the microcirculation. FASEB 46:2397–2401, 1987 Schmid-Schonbein G: Capillary plugging by granulocytes and the no-reflow phenomenon in the microcirculation. **FASEB 46:**2397–2401, 1987
• PubMed
  Schmid-Schonbein G: Capillary plugging by granulocytes and the no-reflow phenomenon in the microcirculation. FASEB 46: 2397– 2401, 1987 Schmid-Schonbein G: Capillary plugging by granulocytes and the no-reflow phenomenon in the microcirculation. FASEB 46:2397–2401, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Schmid-Schonbein G, & Engler R: Granulocytes as active participants in acute myocardial ischemia and infarction. Am J Cardiovasc Pathol 1:15–29, 1987 Schmid-Schonbein G, Engler R: Granulocytes as active participants in acute myocardial ischemia and infarction. **Am J Cardiovasc Pathol 1:**15–29, 1987
• PubMed
  Schmid-Schonbein G, Engler R: Granulocytes as active participants in acute myocardial ischemia and infarction. Am J Cardiovasc Pathol 1: 15– 29, 1987 Schmid-Schonbein G, Engler R: Granulocytes as active participants in acute myocardial ischemia and infarction. Am J Cardiovasc Pathol 1:15–29, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Simpson P, , Todd R, & Fantoen J, et al: Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody that inhibits leukocyte adhesion. J Clin Invest 81:624–629, 1988 Simpson P, Todd R, Fantoen J, et al: Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody that inhibits leukocyte adhesion. **J Clin Invest 81:**624–629, 1988
• PubMed
  Simpson P, Todd R, Fantoen J, et al: Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody that inhibits leukocyte adhesion. J Clin Invest 81: 624– 629, 1988 Simpson P, Todd R, Fantoen J, et al: Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody that inhibits leukocyte adhesion. J Clin Invest 81:624–629, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Smith C, , Martin S, & Rothlein R, et al: Cooperative interactions of LFA-1 and Mac-1 with intercellular adhesion molecule-1 in facilitating adherence and transendothelial migration of human neutrophils in vitro. J Clin Invest 83:2008–2017, 1989 Smith C, Martin S, Rothlein R, et al: Cooperative interactions of LFA-1 and Mac-1 with intercellular adhesion molecule-1 in facilitating adherence and transendothelial migration of human neutrophils in vitro. **J Clin Invest 83:**2008–2017, 1989
• PubMed
  Smith C, Martin S, Rothlein R, et al: Cooperative interactions of LFA-1 and Mac-1 with intercellular adhesion molecule-1 in facilitating adherence and transendothelial migration of human neutrophils in vitro. J Clin Invest 83: 2008– 2017, 1989 Smith C, Martin S, Rothlein R, et al: Cooperative interactions of LFA-1 and Mac-1 with intercellular adhesion molecule-1 in facilitating adherence and transendothelial migration of human neutrophils in vitro. J Clin Invest 83:2008–2017, 1989
  )| false
• Search Google Scholar
• Export Citation
• 1. Smith C, , Rothlein R, & Hughes B, et al: Recognition of an endothelial determinant for CD-18 dependent human neutrophil adherence and transendothelial migration. J Clin Invest 82:1746–1756, 1988 Smith C, Rothlein R, Hughes B, et al: Recognition of an endothelial determinant for CD-18 dependent human neutrophil adherence and transendothelial migration. **J Clin Invest 82:**1746–1756, 1988
• PubMed
  Smith C, Rothlein R, Hughes B, et al: Recognition of an endothelial determinant for CD-18 dependent human neutrophil adherence and transendothelial migration. J Clin Invest 82: 1746– 1756, 1988 Smith C, Rothlein R, Hughes B, et al: Recognition of an endothelial determinant for CD-18 dependent human neutrophil adherence and transendothelial migration. J Clin Invest 82:1746–1756, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Sobel R, , Mitchell M, & Fondren G: Intercellular adhesion molecule (ICAM) in cellular immune reactions in the human central nervous system. Am J Pathol 136:1309–1315, 1990 Sobel R, Mitchell M, Fondren G: Intercellular adhesion molecule (ICAM) in cellular immune reactions in the human central nervous system. **Am J Pathol 136:**1309–1315, 1990
• PubMed
  Sobel R, Mitchell M, Fondren G: Intercellular adhesion molecule (ICAM) in cellular immune reactions in the human central nervous system. Am J Pathol 136: 1309– 1315, 1990 Sobel R, Mitchell M, Fondren G: Intercellular adhesion molecule (ICAM) in cellular immune reactions in the human central nervous system. Am J Pathol 136:1309–1315, 1990
  )| false
• Search Google Scholar
• Export Citation
• 1. Vedder N, , Winn C, & Rice C, et al: A monoclonal antibody to the adherence promoting leukocyte glycoprotein, CD 18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. J Clin Invest 81:939–944, 1988 Vedder N, Winn C, Rice C, et al: A monoclonal antibody to the adherence promoting leukocyte glycoprotein, CD 18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. **J Clin Invest 81:**939–944, 1988
• PubMed
  Vedder N, Winn C, Rice C, et al: A monoclonal antibody to the adherence promoting leukocyte glycoprotein, CD 18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. J Clin Invest 81: 939– 944, 1988 Vedder N, Winn C, Rice C, et al: A monoclonal antibody to the adherence promoting leukocyte glycoprotein, CD 18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. J Clin Invest 81:939–944, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Waud D: On biological assays involving quantal responses. J Pharmacol Exp Ther 183:577–607, 1972 Waud D: On biological assays involving quantal responses. **J Pharmacol Exp Ther 183:**577–607, 1972
• PubMed
  Waud D: On biological assays involving quantal responses. J Pharmacol Exp Ther 183: 577– 607, 1972 Waud D: On biological assays involving quantal responses. J Pharmacol Exp Ther 183:577–607, 1972
  )| false
• Search Google Scholar
• Export Citation
• 1. Wegner C, , Gundel R, & Reilly P, et al: Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma. Science 247:456–459, 1990 Wegner C, Gundel R, Reilly P, et al: Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma. **Science 247:**456–459, 1990
• PubMed
  Wegner C, Gundel R, Reilly P, et al: Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma. Science 247: 456– 459, 1990 Wegner C, Gundel R, Reilly P, et al: Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma. Science 247:456–459, 1990
  )| false
• Search Google Scholar
• Export Citation
• 1. Zimmerman G, & Mclntyre T: Neutrophil adherence to human endothelium in vitro occurs by CD-18 glycoprotein dependent and independent mechanisms. J Clin Invest 81:531–537, 1988 Zimmerman G, Mclntyre T: Neutrophil adherence to human endothelium in vitro occurs by CD-18 glycoprotein dependent and independent mechanisms. **J Clin Invest 81:**531–537, 1988
• PubMed
  Zimmerman G, Mclntyre T: Neutrophil adherence to human endothelium in vitro occurs by CD-18 glycoprotein dependent and independent mechanisms. J Clin Invest 81: 531– 537, 1988 Zimmerman G, Mclntyre T: Neutrophil adherence to human endothelium in vitro occurs by CD-18 glycoprotein dependent and independent mechanisms. J Clin Invest 81:531–537, 1988
  )| false
• Search Google Scholar
• Export Citation
• 1. Zivin J, , DeGirolami U, & Hurwitz E: Spectrum of neurological deficits in experimental CNS ischemia. Arch Neurol 39:408–412, 1982 Zivin J, DeGirolami U, Hurwitz E: Spectrum of neurological deficits in experimental CNS ischemia. **Arch Neurol 39:**408–412, 1982
• PubMed
  Zivin J, DeGirolami U, Hurwitz E: Spectrum of neurological deficits in experimental CNS ischemia. Arch Neurol 39: 408– 412, 1982 Zivin J, DeGirolami U, Hurwitz E: Spectrum of neurological deficits in experimental CNS ischemia. Arch Neurol 39:408–412, 1982
  )| false
• Search Google Scholar
• Export Citation
• 1. Zivin J, , DeGirolami U, & Lyden P, et al: A model for quantitative evaluation of embolic stroke therapy. Brain Res 435:305–308, 1987 Zivin J, DeGirolami U, Lyden P, et al: A model for quantitative evaluation of embolic stroke therapy. **Brain Res 435:**305–308, 1987
• PubMed
  Zivin J, DeGirolami U, Lyden P, et al: A model for quantitative evaluation of embolic stroke therapy. Brain Res 435: 305– 308, 1987 Zivin J, DeGirolami U, Lyden P, et al: A model for quantitative evaluation of embolic stroke therapy. Brain Res 435:305–308, 1987
  )| false
• Search Google Scholar
• Export Citation
• 1. Zivin J, & Venditto J: Experimental central nervous system ischemia: serotonin antagonists reduce or prevent damage. Neurology 34:469–474, 1984 Zivin J, Venditto J: Experimental central nervous system ischemia: serotonin antagonists reduce or prevent damage. **Neurology 34:**469–474, 1984
• PubMed
  Zivin J, Venditto J: Experimental central nervous system ischemia: serotonin antagonists reduce or prevent damage. Neurology 34: 469– 474, 1984 Zivin J, Venditto J: Experimental central nervous system ischemia: serotonin antagonists reduce or prevent damage. Neurology 34:469–474, 1984
  )| false
• Search Google Scholar
• Export Citation