Antonina Giammanco - Academia.edu (original) (raw)

Uploads

Volume 5 , Issue 2 by Antonina Giammanco

Research paper thumbnail of A Lesson from a Fatal Case of Leptospirosis Author's Details

Leptospirosis is probably the most widespread and prevalent zoonotic disease in the world and it ... more Leptospirosis is probably the most widespread and prevalent zoonotic disease in the world and it is difficult to diagnose. Therefore, the disease is frequently not recognized and consequently severely neglected. This infectious disease is re-emerging globally and numerous outbreaks have occurred worldwide during the past decade. It may occur wherever the risk of direct or indirect exposure to urine or kidneys of infected animals is present. Leptospirosis is likely to be underestimated because many patients have mild or subclinical infection and serological testing is imperfect and not always performed. Here it is presented a fatal case of Leptospirosis in Sicily, occurred at the Internal Medicine Division of the University Hospital of Palermo.

Papers by Antonina Giammanco

Research paper thumbnail of Albumin versus solvent/detergent-treated pooled plasma as replacement fluid for long-term plasma exchange therapy in a patient with primary hypertriglyceridemia and recurrent hyperlipidemic pancreatitis

Transfusion, Jan 4, 2015

Chylomicronemia syndrome is a metabolic condition characterized by severe fasting hypertrigliceri... more Chylomicronemia syndrome is a metabolic condition characterized by severe fasting hypertrigliceridemia (≥1000 mg/dL) and other clinical features including chronic abdominal pain and recurrent acute pancreatitis. In patients with acute or recurrent pancreatitis, plasma exchange (PEx) is indicated for the treatment of acute disease and prevention of recurrence. The use of plasma instead of albumin as replacement fluid has been suggested for its putative ability to replace the deficient enzyme possibly leading to better clinical improvement. A 40-year-old man with chylomicronemia syndrome due to a newly identified loss-of-function mutation in the lipoprotein lipase (LPL) gene (IVS2, c.250-1G/C) has been treated at our hospital since the age of 13. From age 18 to age 34, the patient had five episodes of acute pancreatitis while his triglyceride (TG) levels were extremely high (2500-4000 mg/dL). As the TG levels remained stable over 4000 mg/dL despite the maximum medical treatment, the p...

Research paper thumbnail of Novel CREB3L3 Nonsense Mutation in a Family With Dominant Hypertriglyceridemia

Arteriosclerosis, Thrombosis, and Vascular Biology, 2015

Cyclic AMP responsive element-binding protein 3-like 3 (CREB3L3) is a novel candidate gene for do... more Cyclic AMP responsive element-binding protein 3-like 3 (CREB3L3) is a novel candidate gene for dominant hypertriglyceridemia. To date, only 4 kindred with dominant hypertriglyceridemia have been found to be carriers of 2 nonsense mutations in CREB3L3 gene (245fs and W46X). We investigated a family in which hypertriglyceridemia displayed an autosomal dominant pattern of inheritance. The proband was a 49-year-old woman with high plasma triglycerides (≤1300 mg/dL; 14.68 mmol/L). Her father had a history of moderate hypertriglyceridemia, and her 51-year-old brother had triglycerides levels as high as 1600 mg/dL (18.06 mmol/L). To identify the causal mutation in this family, we analyzed the candidate genes of recessive and dominant forms of primary hypertriglyceridemia by direct sequencing. The sequencing of CREB3L3 gene led to the discovery of a novel minute frame shift mutation in exon 3 of CREB3L3 gene, predicted to result in the formation of a truncated protein devoid of function (c.359delG-p.K120fsX20). Heterozygosity for the c.359delG mutation resulted in a severe phenotype in the proband, and her brother with a late in life expression and a good response to diet and a hypotriglyceridemic treatment. The same mutation was detected in a 13-year-old daughter who to date is normotriglyceridemic. We have identified a novel pathogenic mutation in CREB3L3 gene in a family with dominant hypertriglyceridemia with a variable pattern of penetrance.

Research paper thumbnail of 230 Conditional Intestine-Specific HuR Deletion (HuR-Iko) Modifies Epithelial Growth, Response to Injury and Tumorigenesis

Research paper thumbnail of Tu1732 Conditional Intestinal-Epithelial Specific HuR Deletion Modifies Murine Intestinal Tumorigenesis

Research paper thumbnail of Effects of Steatosis on Hepatic Hemodynamics in Patients with Metabolic Syndrome

Ultrasound in medicine & biology, Jan 18, 2015

The aim of our study was to assess the hemodynamic changes in hepatic and splenic circulation usi... more The aim of our study was to assess the hemodynamic changes in hepatic and splenic circulation using B-mode ultrasonography and color Doppler ultrasonography, in a population of patients with metabolic syndrome divided with respect to the presence or absence of steatosis diagnosed by ultrasonography. One hundred forty-one patients were included in the study. The severity of non-alcoholic fatty liver disease was classified as mild, moderate or severe. Visceral fat thickness, longitudinal diameter of the spleen, diameter of the portal vein, mean maximum portal vein flow velocity, hepatic artery and splenic artery resistivity indexes and hepatic vein flow phasicity were measured. Non-alcoholic fatty liver disease was detected in 114 of 141 patients, with a prevalence of 80.8%. Patients with steatosis had significantly greater diameters of the portal vein, longitudinal diameters of the spleen, visceral fat thickness and hepatic artery and splenic artery resistivity indexes, whereas their...

Research paper thumbnail of The pathophysiology of intestinal lipoprotein production

Frontiers in Physiology, 2015

Intestinal lipoprotein production is a multistep process, essential for the absorption of dietary... more Intestinal lipoprotein production is a multistep process, essential for the absorption of dietary fats and fat-soluble vitamins. Chylomicron assembly begins in the endoplasmic reticulum with the formation of primordial, phospholipids-rich particles that are then transported to the Golgi for secretion. Several classes of transporters play a role in the selective uptake and/or export of lipids through the villus enterocytes. Once secreted in the lymph stream, triglyceride-rich lipoproteins (TRLs) are metabolized by Lipoprotein lipase (LPL), which catalyzes the hydrolysis of triacylglycerols of very low density lipoproteins (VLDLs) and chylomicrons, thereby delivering free fatty acids to various tissues. Genetic mutations in the genes codifying for these proteins are responsible of different inherited disorders affecting chylomicron metabolism. This review focuses on the molecular pathways that modulate the uptake and the transport of lipoproteins of intestinal origin and it will highlight recent findings on TRLs assembly.

Research paper thumbnail of Intestinal epithelial HuR modulates distinct pathways of proliferation and apoptosis and attenuates small intestinal and colonic tumor development

Cancer research, Jan 15, 2014

HuR is a ubiquitous nucleocytoplasmic RNA-binding protein that exerts pleiotropic effects on cell... more HuR is a ubiquitous nucleocytoplasmic RNA-binding protein that exerts pleiotropic effects on cell growth and tumorigenesis. In this study, we explored the impact of conditional, tissue-specific genetic deletion of HuR on intestinal growth and tumorigenesis in mice. Mice lacking intestinal expression of HuR (Hur (IKO) mice) displayed reduced levels of cell proliferation in the small intestine and increased sensitivity to doxorubicin-induced acute intestinal injury, as evidenced by decreased villus height and a compensatory shift in proliferating cells. In the context of Apc(min/+) mice, a transgenic model of intestinal tumorigenesis, intestinal deletion of the HuR gene caused a three-fold decrease in tumor burden characterized by reduced proliferation, increased apoptosis, and decreased expression of transcripts encoding antiapoptotic HuR target RNAs. Similarly, Hur(IKO) mice subjected to an inflammatory colon carcinogenesis protocol [azoxymethane and dextran sodium sulfate (AOM-DSS)...

Research paper thumbnail of 294 Conditional Intestine-Specific HuR Deletion (HuR-IKO) Modulates Distinct Wnt-Dependent and Independent Pathways of Proliferation and Apoptosis With No Effect on Ileal ASBT Expression or Bile Acid Metabolism

Research paper thumbnail of A Lesson from a Fatal Case of Leptospirosis Author's Details

Leptospirosis is probably the most widespread and prevalent zoonotic disease in the world and it ... more Leptospirosis is probably the most widespread and prevalent zoonotic disease in the world and it is difficult to diagnose. Therefore, the disease is frequently not recognized and consequently severely neglected. This infectious disease is re-emerging globally and numerous outbreaks have occurred worldwide during the past decade. It may occur wherever the risk of direct or indirect exposure to urine or kidneys of infected animals is present. Leptospirosis is likely to be underestimated because many patients have mild or subclinical infection and serological testing is imperfect and not always performed. Here it is presented a fatal case of Leptospirosis in Sicily, occurred at the Internal Medicine Division of the University Hospital of Palermo.

Research paper thumbnail of Albumin versus solvent/detergent-treated pooled plasma as replacement fluid for long-term plasma exchange therapy in a patient with primary hypertriglyceridemia and recurrent hyperlipidemic pancreatitis

Transfusion, Jan 4, 2015

Chylomicronemia syndrome is a metabolic condition characterized by severe fasting hypertrigliceri... more Chylomicronemia syndrome is a metabolic condition characterized by severe fasting hypertrigliceridemia (≥1000 mg/dL) and other clinical features including chronic abdominal pain and recurrent acute pancreatitis. In patients with acute or recurrent pancreatitis, plasma exchange (PEx) is indicated for the treatment of acute disease and prevention of recurrence. The use of plasma instead of albumin as replacement fluid has been suggested for its putative ability to replace the deficient enzyme possibly leading to better clinical improvement. A 40-year-old man with chylomicronemia syndrome due to a newly identified loss-of-function mutation in the lipoprotein lipase (LPL) gene (IVS2, c.250-1G/C) has been treated at our hospital since the age of 13. From age 18 to age 34, the patient had five episodes of acute pancreatitis while his triglyceride (TG) levels were extremely high (2500-4000 mg/dL). As the TG levels remained stable over 4000 mg/dL despite the maximum medical treatment, the p...

Research paper thumbnail of Novel CREB3L3 Nonsense Mutation in a Family With Dominant Hypertriglyceridemia

Arteriosclerosis, Thrombosis, and Vascular Biology, 2015

Cyclic AMP responsive element-binding protein 3-like 3 (CREB3L3) is a novel candidate gene for do... more Cyclic AMP responsive element-binding protein 3-like 3 (CREB3L3) is a novel candidate gene for dominant hypertriglyceridemia. To date, only 4 kindred with dominant hypertriglyceridemia have been found to be carriers of 2 nonsense mutations in CREB3L3 gene (245fs and W46X). We investigated a family in which hypertriglyceridemia displayed an autosomal dominant pattern of inheritance. The proband was a 49-year-old woman with high plasma triglycerides (≤1300 mg/dL; 14.68 mmol/L). Her father had a history of moderate hypertriglyceridemia, and her 51-year-old brother had triglycerides levels as high as 1600 mg/dL (18.06 mmol/L). To identify the causal mutation in this family, we analyzed the candidate genes of recessive and dominant forms of primary hypertriglyceridemia by direct sequencing. The sequencing of CREB3L3 gene led to the discovery of a novel minute frame shift mutation in exon 3 of CREB3L3 gene, predicted to result in the formation of a truncated protein devoid of function (c.359delG-p.K120fsX20). Heterozygosity for the c.359delG mutation resulted in a severe phenotype in the proband, and her brother with a late in life expression and a good response to diet and a hypotriglyceridemic treatment. The same mutation was detected in a 13-year-old daughter who to date is normotriglyceridemic. We have identified a novel pathogenic mutation in CREB3L3 gene in a family with dominant hypertriglyceridemia with a variable pattern of penetrance.

Research paper thumbnail of 230 Conditional Intestine-Specific HuR Deletion (HuR-Iko) Modifies Epithelial Growth, Response to Injury and Tumorigenesis

Research paper thumbnail of Tu1732 Conditional Intestinal-Epithelial Specific HuR Deletion Modifies Murine Intestinal Tumorigenesis

Research paper thumbnail of Effects of Steatosis on Hepatic Hemodynamics in Patients with Metabolic Syndrome

Ultrasound in medicine & biology, Jan 18, 2015

The aim of our study was to assess the hemodynamic changes in hepatic and splenic circulation usi... more The aim of our study was to assess the hemodynamic changes in hepatic and splenic circulation using B-mode ultrasonography and color Doppler ultrasonography, in a population of patients with metabolic syndrome divided with respect to the presence or absence of steatosis diagnosed by ultrasonography. One hundred forty-one patients were included in the study. The severity of non-alcoholic fatty liver disease was classified as mild, moderate or severe. Visceral fat thickness, longitudinal diameter of the spleen, diameter of the portal vein, mean maximum portal vein flow velocity, hepatic artery and splenic artery resistivity indexes and hepatic vein flow phasicity were measured. Non-alcoholic fatty liver disease was detected in 114 of 141 patients, with a prevalence of 80.8%. Patients with steatosis had significantly greater diameters of the portal vein, longitudinal diameters of the spleen, visceral fat thickness and hepatic artery and splenic artery resistivity indexes, whereas their...

Research paper thumbnail of The pathophysiology of intestinal lipoprotein production

Frontiers in Physiology, 2015

Intestinal lipoprotein production is a multistep process, essential for the absorption of dietary... more Intestinal lipoprotein production is a multistep process, essential for the absorption of dietary fats and fat-soluble vitamins. Chylomicron assembly begins in the endoplasmic reticulum with the formation of primordial, phospholipids-rich particles that are then transported to the Golgi for secretion. Several classes of transporters play a role in the selective uptake and/or export of lipids through the villus enterocytes. Once secreted in the lymph stream, triglyceride-rich lipoproteins (TRLs) are metabolized by Lipoprotein lipase (LPL), which catalyzes the hydrolysis of triacylglycerols of very low density lipoproteins (VLDLs) and chylomicrons, thereby delivering free fatty acids to various tissues. Genetic mutations in the genes codifying for these proteins are responsible of different inherited disorders affecting chylomicron metabolism. This review focuses on the molecular pathways that modulate the uptake and the transport of lipoproteins of intestinal origin and it will highlight recent findings on TRLs assembly.

Research paper thumbnail of Intestinal epithelial HuR modulates distinct pathways of proliferation and apoptosis and attenuates small intestinal and colonic tumor development

Cancer research, Jan 15, 2014

HuR is a ubiquitous nucleocytoplasmic RNA-binding protein that exerts pleiotropic effects on cell... more HuR is a ubiquitous nucleocytoplasmic RNA-binding protein that exerts pleiotropic effects on cell growth and tumorigenesis. In this study, we explored the impact of conditional, tissue-specific genetic deletion of HuR on intestinal growth and tumorigenesis in mice. Mice lacking intestinal expression of HuR (Hur (IKO) mice) displayed reduced levels of cell proliferation in the small intestine and increased sensitivity to doxorubicin-induced acute intestinal injury, as evidenced by decreased villus height and a compensatory shift in proliferating cells. In the context of Apc(min/+) mice, a transgenic model of intestinal tumorigenesis, intestinal deletion of the HuR gene caused a three-fold decrease in tumor burden characterized by reduced proliferation, increased apoptosis, and decreased expression of transcripts encoding antiapoptotic HuR target RNAs. Similarly, Hur(IKO) mice subjected to an inflammatory colon carcinogenesis protocol [azoxymethane and dextran sodium sulfate (AOM-DSS)...

Research paper thumbnail of 294 Conditional Intestine-Specific HuR Deletion (HuR-IKO) Modulates Distinct Wnt-Dependent and Independent Pathways of Proliferation and Apoptosis With No Effect on Ileal ASBT Expression or Bile Acid Metabolism