Promotion of spontaneous diabetes in non-obese diabetes-prone mice by cyclophosphamide (original) (raw)

References

  1. Ohtori H, Yoshida T, Inuta T (1968) “Small eye and cataract”, a new dominant mutation in the mouse. Exp Anim 17: 91–96 (in Japanese)
    Google Scholar
  2. Ohtori H, Yoshida T (1975) The CTS mouse. Exp Anim 24: 123 (In Japanese)
    Google Scholar
  3. Makino S, Kunimoto K, Muraoka Y, Mizushima Y, Katagiri K, Tochino Y (1980) Breeding of a non-obese, diabetic strain of mice. Exp Anim 29: 1–13
    Google Scholar
  4. Nakhooda AF, Like AA, Chappel CI, Wei CN, Marliss EB (1978) The spontaneously diabetic Wistar rat (the “BB” rat). Diabetologia 14: 199–207
    Google Scholar
  5. Bottazzo GF, Florin-Christensen A, Doniach D (1974) Islet-cell antibodies in diabetes mellitus with autoimmune polyendocrine deficiencies. Lancet 2: 1279–1283
    Google Scholar
  6. Irvine WJ, McCallum CJ, Gray RS, Campbell CJ, Duncan LJP, Farquhar JW, Vaughan H, Morris PJ (1977) Pancreatic islet-cell antibodies in diabetes mellitus correlated with the duration and type of diabetes, coexistent autoimmune disease and HLA type. Diabetes 26: 138–147
    Google Scholar
  7. Lernmark A, Freedman ZR, Hofmann C, Rubenstein AH, Steiner DF, Jackson RL, Winter RJ, Traisman HS (1978) Islet-cell-surface antibodies in juvenile diabetes mellitus. New Engl J Med 299: 375–380
    Google Scholar
  8. Nerup J, Andersen OO, Bendixen G, Eglaberg J, Poulsen JE (1971) Anti-pancreatic cellular hypersensitivity in diabetes mellitus. Diabetes 20: 424–427
    Google Scholar
  9. MacCuish AC, Jordan J, Campbell CJ, Duncan LJP, Irvine WJ (1974) Cell-mediated immunity to human pancreas in diabetes mellitus. Diabetes 23: 693–697
    Google Scholar
  10. Like AA, Butler L, Williams RM, Appel MC, Weringer EJ, Rossini AA (1982) Spontaneous autoimmune diabetes mellitus in the BB rats. Diabetes 31 (Suppl 1): 7–13
    Google Scholar
  11. Haspel MV, Onodera T, Prabharker BS, McClintock PR, Essani K, Ray UR, Yagihash S, Notkins AL (1983) Multiple organ-reactive monoclonal autoantibodies. Nature 304: 73–76
    Google Scholar
  12. Lando Z, Teitelbaum D, Arnon R (1980) Induction of experimental allergic encephalomyelitis in genetically resistant strains of mice. Nature (Lond) 287: 551–552
    Google Scholar
  13. Kiesel U, Greulich B, Marx-Soho Moume, Kolb H (1981) Induction of experimental autoimmune diabetes by low-dose streptozotocin treatment in genetically resistant mice. Immunol Lett 3: 227–230
    Google Scholar
  14. Taniguchi M, Tada T (1971) Regulation of homocytotropic antibody formation in the rat. IV. Effect of various immunosuppressive drugs. J Immunol 107: 579–585
    Google Scholar
  15. Chiorazzi N, Fox DA, Katz DT (1976) Hapten specific IgE antibody responses in mice. VI. Selective enhancement of IgE antibody production by low doses of X-irradiation and by cyclophosphamide. J Immunol 117: 1629–1637
    Google Scholar
  16. Mitsuoka A, Baba M, Morikawa S (1976) Enhancement of delayed hypersensitivity by depletion of suppressor T cells with cyclophosphamide in mice. Nature (Lond) 262: 77–78
    Google Scholar
  17. Lagrange PH, Mackaness GB, Miller TE (1974) Potentiation of T-cell-mediated immunity by selective suppression of antibody formation with cyclophosphamide. J Exp Med 139: 1529–1539
    Google Scholar
  18. Askenase PW, Hayden BJ, Gershon RK (1975) Augmentation of delayed type hypersensitivity by doses of cyclophosphamide which do not affect antibody response. J Exp Med 141: 697–702
    Google Scholar

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