The class B, type I scavenger receptor promotes the selective uptake of high density lipoprotein cholesterol ethers into caveolae - PubMed (original) (raw)
. 1999 Apr 23;274(17):12043-8.
doi: 10.1074/jbc.274.17.12043.
Affiliations
- PMID: 10207027
- DOI: 10.1074/jbc.274.17.12043
Free article
The class B, type I scavenger receptor promotes the selective uptake of high density lipoprotein cholesterol ethers into caveolae
G A Graf et al. J Biol Chem. 1999.
Free article
Abstract
The uptake of cholesterol esters from high density lipoproteins (HDLs) is characterized by the initial movement of cholesterol esters into a reversible plasma membrane pool. Cholesterol esters are subsequently internalized to a nonreversible pool. Unlike the uptake of cholesterol from low density lipoproteins, cholesterol ester uptake from HDL does not involve the internalization and degradation of the particle and is therefore termed selective. The class B, type I scavenger receptor (SR-BI) has been identified as an HDL receptor and shown to mediate selective cholesterol ester uptake. SR-BI is localized to cholesterol- and sphingomyelin-rich microdomains called caveolae. Caveolae are directly involved in cholesterol trafficking. Therefore, we tested the hypothesis that caveolae are acceptors for HDL-derived cholesterol ether (CE). Our studies demonstrate that in Chinese hamster ovary cells expressing SR-BI, >80% of the plasma membrane associated CE is present in caveolae after 7.5 min of selective cholesterol ether uptake. We also show that excess, unlabeled HDL can extract the radiolabeled CE from caveolae, demonstrating that caveolae constitute a reversible plasma membrane pool of CE. Furthermore, 50% of the caveolae-associated CE can be chased into a nonreversible pool. We conclude that caveolae are acceptors for HDL-derived cholesterol ethers, and that caveolae constitute a reversible, plasma membrane pool of cholesterol ethers.
Similar articles
- Caveolin-1 negatively regulates SR-BI mediated selective uptake of high-density lipoprotein-derived cholesteryl ester.
Matveev S, Uittenbogaard A, van Der Westhuyzen D, Smart EJ. Matveev S, et al. Eur J Biochem. 2001 Nov;268(21):5609-16. doi: 10.1046/j.1432-1033.2001.02496.x. Eur J Biochem. 2001. PMID: 11683884 - Probucol enhances selective uptake of HDL-associated cholesteryl esters in vitro by a scavenger receptor B-I-dependent mechanism.
Rinninger F, Wang N, Ramakrishnan R, Jiang XC, Tall AR. Rinninger F, et al. Arterioscler Thromb Vasc Biol. 1999 May;19(5):1325-32. doi: 10.1161/01.atv.19.5.1325. Arterioscler Thromb Vasc Biol. 1999. PMID: 10323786 - Advanced glycation end products (AGE) inhibit scavenger receptor class B type I-mediated reverse cholesterol transport: a new crossroad of AGE to cholesterol metabolism.
Ohgami N, Miyazaki A, Sakai M, Kuniyasu A, Nakayama H, Horiuchi S. Ohgami N, et al. J Atheroscler Thromb. 2003;10(1):1-6. doi: 10.5551/jat.10.1. J Atheroscler Thromb. 2003. PMID: 12621157 Review. - Roles of scavenger receptor BI and APO A-I in selective uptake of HDL cholesterol by adrenal cells.
Williams DL, Temel RE, Connelly MA. Williams DL, et al. Endocr Res. 2000 Nov;26(4):639-51. doi: 10.3109/07435800009048584. Endocr Res. 2000. PMID: 11196441 Review.
Cited by
- The New General Biological Property of Stem-like Tumor Cells (Part II: Surface Molecules, Which Belongs to Distinctive Groups with Particular Functions, Form a Unique Pattern Characteristic of a Certain Type of Tumor Stem-like Cells).
Petrova DD, Dolgova EV, Proskurina AS, Ritter GS, Ruzanova VS, Efremov YR, Potter EA, Kirikovich SS, Levites EV, Taranov OS, Ostanin AA, Chernykh ER, Kolchanov NA, Bogachev SS. Petrova DD, et al. Int J Mol Sci. 2022 Dec 13;23(24):15800. doi: 10.3390/ijms232415800. Int J Mol Sci. 2022. PMID: 36555446 Free PMC article. - Energy and Dynamics of Caveolae Trafficking.
Matthaeus C, Taraska JW. Matthaeus C, et al. Front Cell Dev Biol. 2021 Jan 21;8:614472. doi: 10.3389/fcell.2020.614472. eCollection 2020. Front Cell Dev Biol. 2021. PMID: 33692993 Free PMC article. Review. - Cell Intrinsic and Extrinsic Mechanisms of Caveolin-1-Enhanced Metastasis.
Campos A, Burgos-Ravanal R, González MF, Huilcaman R, Lobos González L, Quest AFG. Campos A, et al. Biomolecules. 2019 Jul 29;9(8):314. doi: 10.3390/biom9080314. Biomolecules. 2019. PMID: 31362353 Free PMC article. Review. - Danshensu Promotes Cholesterol Efflux in RAW264.7 Macrophages.
Gao H, Li L, Li L, Gong B, Dong P, Fordjour PA, Zhu Y, Fan G. Gao H, et al. Lipids. 2016 Sep;51(9):1083-92. doi: 10.1007/s11745-016-4178-1. Epub 2016 Aug 11. Lipids. 2016. PMID: 27514857 - From embryonic development to human diseases: The functional role of caveolae/caveolin.
Sohn J, Brick RM, Tuan RS. Sohn J, et al. Birth Defects Res C Embryo Today. 2016 Mar;108(1):45-64. doi: 10.1002/bdrc.21121. Epub 2016 Mar 17. Birth Defects Res C Embryo Today. 2016. PMID: 26991990 Free PMC article. Review.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical
Research Materials