Mtt1 is a Upf1-like helicase that interacts with the translation termination factors and whose overexpression can modulate termination efficiency - PubMed (original) (raw)
Comparative Study
Mtt1 is a Upf1-like helicase that interacts with the translation termination factors and whose overexpression can modulate termination efficiency
K Czaplinski et al. RNA. 2000 May.
Abstract
Translation termination is the final step that completes the synthesis of a polypeptide. Premature translation termination by introduction of a nonsense mutation leads to the synthesis of a truncated protein. We report the identification and characterization of the product of the MTT1 gene, a helicase belonging to the Upfl-like family of helicases that is involved in modulating translation termination. MTT1 is homologous to UPF1, a factor previously shown to function in both mRNA turnover and translation termination. Overexpression of MTT1 induced a nonsense suppression phenotype in a wild-type yeast strain. Nonsense suppression is apparently not due to induction of [PSI+], even though cooverexpression of HSP104 alleviated the nonsense suppression phenotype observed in cells overexpressing MTT1, suggesting a more direct role of Hsp104p in the translation termination process. The MTT1 gene product was shown to interact with translation termination factors and is localized to polysomes. Taken together, these results indicate that at least two members of a family of RNA helicases modulate translation termination efficiency in cells.
Similar articles
- Identification and characterization of mutations in the UPF1 gene that affect nonsense suppression and the formation of the Upf protein complex but not mRNA turnover.
Weng Y, Czaplinski K, Peltz SW. Weng Y, et al. Mol Cell Biol. 1996 Oct;16(10):5491-506. doi: 10.1128/MCB.16.10.5491. Mol Cell Biol. 1996. PMID: 8816462 Free PMC article. - Leaky termination at premature stop codons antagonizes nonsense-mediated mRNA decay in S. cerevisiae.
Keeling KM, Lanier J, Du M, Salas-Marco J, Gao L, Kaenjak-Angeletti A, Bedwell DM. Keeling KM, et al. RNA. 2004 Apr;10(4):691-703. doi: 10.1261/rna.5147804. RNA. 2004. PMID: 15037778 Free PMC article. - Overexpression of Upf1p compensates for mitochondrial splicing deficiency independently of its role in mRNA surveillance.
de Pinto B, Lippolis R, Castaldo R, Altamura N. de Pinto B, et al. Mol Microbiol. 2004 Feb;51(4):1129-42. doi: 10.1046/j.1365-2958.2003.03889.x. Mol Microbiol. 2004. PMID: 14763985 - Modulation of efficiency of translation termination in Saccharomyces cerevisiae.
Nizhnikov AA, Antonets KS, Inge-Vechtomov SG, Derkatch IL. Nizhnikov AA, et al. Prion. 2014;8(3):247-60. doi: 10.4161/pri.29851. Epub 2014 Nov 1. Prion. 2014. PMID: 25486049 Free PMC article. Review. - RNA surveillance. Unforeseen consequences for gene expression, inherited genetic disorders and cancer.
Culbertson MR. Culbertson MR. Trends Genet. 1999 Feb;15(2):74-80. doi: 10.1016/s0168-9525(98)01658-8. Trends Genet. 1999. PMID: 10098411 Review.
Cited by
- Omnipotent decoding potential resides in eukaryotic translation termination factor eRF1 of variant-code organisms and is modulated by the interactions of amino acid sequences within domain 1.
Ito K, Frolova L, Seit-Nebi A, Karamyshev A, Kisselev L, Nakamura Y. Ito K, et al. Proc Natl Acad Sci U S A. 2002 Jun 25;99(13):8494-9. doi: 10.1073/pnas.142690099. Proc Natl Acad Sci U S A. 2002. PMID: 12084909 Free PMC article. - Cosuppression of eukaryotic release factor 1-1 in Arabidopsis affects cell elongation and radial cell division.
Petsch KA, Mylne J, Botella JR. Petsch KA, et al. Plant Physiol. 2005 Sep;139(1):115-26. doi: 10.1104/pp.105.062695. Epub 2005 Aug 19. Plant Physiol. 2005. PMID: 16113224 Free PMC article. - Gene overexpression as a tool for identifying new trans-acting factors involved in translation termination in Saccharomyces cerevisiae.
Namy O, Hatin I, Stahl G, Liu H, Barnay S, Bidou L, Rousset JP. Namy O, et al. Genetics. 2002 Jun;161(2):585-94. doi: 10.1093/genetics/161.2.585. Genetics. 2002. PMID: 12072456 Free PMC article. - Absence of Dbp2p alters both nonsense-mediated mRNA decay and rRNA processing.
Bond AT, Mangus DA, He F, Jacobson A. Bond AT, et al. Mol Cell Biol. 2001 Nov;21(21):7366-79. doi: 10.1128/MCB.21.21.7366-7379.2001. Mol Cell Biol. 2001. PMID: 11585918 Free PMC article. - The contribution and therapeutic implications of IGHMBP2 mutations on IGHMBP2 biochemical activity and ABT1 association.
Vadla GP, Singh K, Lorson CL, Lorson MA. Vadla GP, et al. Biochim Biophys Acta Mol Basis Dis. 2024 Apr;1870(4):167091. doi: 10.1016/j.bbadis.2024.167091. Epub 2024 Feb 24. Biochim Biophys Acta Mol Basis Dis. 2024. PMID: 38403020
References
- Mol Microbiol. 1993 Mar;7(5):683-92 - PubMed
- Proc Natl Acad Sci U S A. 1993 Aug 1;90(15):7034-8 - PubMed
- J Biol Chem. 1993 Oct 15;268(29):21783-90 - PubMed
- Biochemistry. 1993 Dec 14;32(49):13393-8 - PubMed
- Biochemistry. 1994 Apr 5;33(13):3906-12 - PubMed
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Molecular Biology Databases