The group-specific murine coronavirus genes are not essential, but their deletion, by reverse genetics, is attenuating in the natural host - PubMed (original) (raw)
Comparative Study
The group-specific murine coronavirus genes are not essential, but their deletion, by reverse genetics, is attenuating in the natural host
Cornelis A M de Haan et al. Virology. 2002.
Abstract
In addition to a characteristic set of essential genes coronaviruses contain several so-called group-specific genes. These genes differ distinctly among the three coronavirus groups and are specific for each group. While the essential genes encode replication and structural functions, hardly anything is known about the products and functions of the group-specific genes. As a first step to elucidate their significance, we deleted the group-specific genes from the group 2 mouse hepatitis virus (MHV) genome via a novel targeted recombination system based on host switching (L. Kuo, G. J.Godeke, M. J. Raamsman, P. S. Masters, and P. J. M. Rottier, 2000, J. Virol. 74, 1393-1406). Thus, we obtained recombinant viruses from which the two clusters of group-specific genes were deleted either separately or in combination in a controlled genetic background. As all recombinant deletion mutant viruses appeared to be viable, we conclude that the MHV group-specific genes are nonessential, accessory genes. Importantly, all deletion mutant viruses were attenuated when inoculated into their natural host, the mouse. Therefore, deletion of the coronavirus group-specific genes seems to provide an attractive approach to generate attenuated live coronavirus vaccines.
Similar articles
- Coronavirus non-structural protein 1 is a major pathogenicity factor: implications for the rational design of coronavirus vaccines.
Züst R, Cervantes-Barragán L, Kuri T, Blakqori G, Weber F, Ludewig B, Thiel V. Züst R, et al. PLoS Pathog. 2007 Aug 10;3(8):e109. doi: 10.1371/journal.ppat.0030109. PLoS Pathog. 2007. PMID: 17696607 Free PMC article. - Live, attenuated coronavirus vaccines through the directed deletion of group-specific genes provide protection against feline infectious peritonitis.
Haijema BJ, Volders H, Rottier PJ. Haijema BJ, et al. J Virol. 2004 Apr;78(8):3863-71. doi: 10.1128/jvi.78.8.3863-3871.2004. J Virol. 2004. PMID: 15047802 Free PMC article. - The viral spike protein is not involved in the polarized sorting of coronaviruses in epithelial cells.
Rossen JW, de Beer R, Godeke GJ, Raamsman MJ, Horzinek MC, Vennema H, Rottier PJ. Rossen JW, et al. J Virol. 1998 Jan;72(1):497-503. doi: 10.1128/JVI.72.1.497-503.1998. J Virol. 1998. PMID: 9420251 Free PMC article. - Analysis of nonessential gene function in recombinant MHV-JHM. Gene 4 knockout recombinant virus.
Ontiveros E, Kuo L, Masters P, Perlman S. Ontiveros E, et al. Adv Exp Med Biol. 2001;494:83-9. doi: 10.1007/978-1-4615-1325-4_13. Adv Exp Med Biol. 2001. PMID: 11774550 No abstract available. - Development of mouse hepatitis virus and SARS-CoV infectious cDNA constructs.
Baric RS, Sims AC. Baric RS, et al. Curr Top Microbiol Immunol. 2005;287:229-52. doi: 10.1007/3-540-26765-4_8. Curr Top Microbiol Immunol. 2005. PMID: 15609514 Free PMC article. Review.
Cited by
- Potential Transcriptional Enhancers in Coronaviruses: From Infectious Bronchitis Virus to SARS-CoV-2.
Patarca R, Haseltine WA. Patarca R, et al. Int J Mol Sci. 2024 Jul 23;25(15):8012. doi: 10.3390/ijms25158012. Int J Mol Sci. 2024. PMID: 39125583 Free PMC article. - SARS-CoV-2 accessory protein 7b forms homotetramers in detergent.
Surya W, Queralt-Martin M, Mu Y, Aguilella VM, Torres J. Surya W, et al. Virol J. 2022 Nov 21;19(1):193. doi: 10.1186/s12985-022-01920-0. Virol J. 2022. PMID: 36414943 Free PMC article. - Host-Genome Similarity Characterizes the Adaption of SARS-CoV-2 to Humans.
Sun W. Sun W. Biomolecules. 2022 Jul 12;12(7):972. doi: 10.3390/biom12070972. Biomolecules. 2022. PMID: 35883528 Free PMC article. - Reduced subgenomic RNA expression is a molecular indicator of asymptomatic SARS-CoV-2 infection.
Wong CH, Ngan CY, Goldfeder RL, Idol J, Kuhlberg C, Maurya R, Kelly K, Omerza G, Renzette N, De Abreu F, Li L, Browne FA, Liu ET, Wei CL. Wong CH, et al. Commun Med (Lond). 2021 Sep 22;1:33. doi: 10.1038/s43856-021-00034-y. eCollection 2021. Commun Med (Lond). 2021. PMID: 35602196 Free PMC article. - Viral and cellular translation during SARS-CoV-2 infection.
Eriani G, Martin F. Eriani G, et al. FEBS Open Bio. 2022 Sep;12(9):1584-1601. doi: 10.1002/2211-5463.13413. Epub 2022 Apr 25. FEBS Open Bio. 2022. PMID: 35429230 Free PMC article. Review.
References
- Bredenbeek P.J., Pachuk C.J., Noten A.F., Charite J., Luytjes W., Weiss S.R., Spaan W.J. The primary structure and expression of the second open reading frame of the polymerase gene of the coronavirus MHV-A59; a highly conserved polymerase is expressed by an efficient ribosomal frameshifting mechanism. Nucleic Acids Res. 1990;18:1825–1832. - PMC - PubMed
- Brian D.A., Hogue B.G., Kienzle T.E. The coronavirus hemagglutinin esterase glycoprotein. In: Siddell S.G., editor. The Coronaviridae. Plenum Press; New York: 1995. pp. 165–179.
- Brown T.D.K., Brierly I. The coronavirus nonstructural proteins. The coronavirus hemagglutinin esterase glycoprotein. In: Siddell S.G., editor. The Coronaviridae. Plenum Press; New York: 1995. pp. 191–217.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Research Materials