Genetic and functional analysis of the chromosome-encoded carbapenem-hydrolyzing oxacillinase OXA-40 of Acinetobacter baumannii - PubMed (original) (raw)
Genetic and functional analysis of the chromosome-encoded carbapenem-hydrolyzing oxacillinase OXA-40 of Acinetobacter baumannii
Claire Héritier et al. Antimicrob Agents Chemother. 2003 Jan.
Abstract
Clinical isolate Acinetobacter baumannii CLA-1 was resistant to a series of antibiotic molecules, including carbapenems. Cloning and expression of the beta-lactamase gene content of this isolate in Escherichia coli DH10B identified a chromosome-encoded oxacillinase, OXA-40, that differed by one or two amino acid changes from OXA-24, -25, and -26 and an AmpC-type cephalosporinase. The OXA-40 beta-lactamase had a mainly narrow-spectrum hydrolytic profile, but it included ceftazidime and imipenem. Its activity was resistant to inhibition by clavulanic acid, tazobactam, sulbactam, and, like most of the other carbapenem-hydrolyzing oxacillinases, NaCl. OXA-40 had an FGN triad replacing a YGN motif at class D beta-lactamase (DBL) positions 144 to 146. Site-directed DNA mutagenesis leading to a Phe-to-Tyr change at DBL position 144 in OXA-40 gave a mutant enzyme with increased hydrolytic activity against most beta-lactams, including imipenem. Conversely, with a gene encoding the narrow-spectrum oxacillinase OXA-1 as the template, a nucleotide substitution leading to a Tyr-to-Phe change in the YGN motif of OXA-1 gave a mutant enzyme with decreased hydrolytic activity without an increase in carbapenem-hydrolyzing activity. Thus, the Phe residue in the FGN motif was not associated with carbapenem-hydrolyzing activity by itself but instead was associated with weak overall hydrolytic activity. Finally, this Phe residue in OXA-40 explained resistance to inhibition by NaCl whereas a Tyr residue in motif YGN was related to susceptibility to NaCl.
Figures
FIG. 1.
Comparison of the amino acid sequence of OXA-40 to those of the most closely related oxacillinases, OXA-24, -25, and -26 (3, 10). Dashes indicate identical amino acids. The numbering is in accordance with the DBL numbering system (14). The motifs usually conserved among DBLs are shaded.
Similar articles
- Contribution of acquired carbapenem-hydrolyzing oxacillinases to carbapenem resistance in Acinetobacter baumannii.
Héritier C, Poirel L, Lambert T, Nordmann P. Héritier C, et al. Antimicrob Agents Chemother. 2005 Aug;49(8):3198-202. doi: 10.1128/AAC.49.8.3198-3202.2005. Antimicrob Agents Chemother. 2005. PMID: 16048925 Free PMC article. - Characterization of the naturally occurring oxacillinase of Acinetobacter baumannii.
Héritier C, Poirel L, Fournier PE, Claverie JM, Raoult D, Nordmann P. Héritier C, et al. Antimicrob Agents Chemother. 2005 Oct;49(10):4174-9. doi: 10.1128/AAC.49.10.4174-4179.2005. Antimicrob Agents Chemother. 2005. PMID: 16189095 Free PMC article. - OXA-24, a novel class D beta-lactamase with carbapenemase activity in an Acinetobacter baumannii clinical strain.
Bou G, Oliver A, Martínez-Beltrán J. Bou G, et al. Antimicrob Agents Chemother. 2000 Jun;44(6):1556-61. doi: 10.1128/AAC.44.6.1556-1561.2000. Antimicrob Agents Chemother. 2000. PMID: 10817708 Free PMC article. - Emergence of Carbapenem-Hydrolyzing Oxacillinases in Acinetobacter baumannii in Children from Croatia.
Lukić-Grlić A, Kos M, Žižek M, Luxner J, Grisold A, Zarfel G, Bedenić B. Lukić-Grlić A, et al. Chemotherapy. 2019;64(4):167-172. doi: 10.1159/000503746. Epub 2019 Nov 8. Chemotherapy. 2019. PMID: 31707391 - Carbapenem resistance in Acinetobacter baumannii: mechanisms and epidemiology.
Poirel L, Nordmann P. Poirel L, et al. Clin Microbiol Infect. 2006 Sep;12(9):826-36. doi: 10.1111/j.1469-0691.2006.01456.x. Clin Microbiol Infect. 2006. PMID: 16882287 Review.
Cited by
- Regional occurrence of plasmid-mediated carbapenem-hydrolyzing oxacillinase OXA-58 in Acinetobacter spp. in Europe.
Marqué S, Poirel L, Héritier C, Brisse S, Blasco MD, Filip R, Coman G, Naas T, Nordmann P. Marqué S, et al. J Clin Microbiol. 2005 Sep;43(9):4885-8. doi: 10.1128/JCM.43.9.4885-4888.2005. J Clin Microbiol. 2005. PMID: 16145167 Free PMC article. - Epidemiology and Mechanisms of Resistance of Extensively Drug Resistant Gram-Negative Bacteria.
Eichenberger EM, Thaden JT. Eichenberger EM, et al. Antibiotics (Basel). 2019 Apr 6;8(2):37. doi: 10.3390/antibiotics8020037. Antibiotics (Basel). 2019. PMID: 30959901 Free PMC article. Review. - β-Lactamase inhibition by 7-alkylidenecephalosporin sulfones: allylic transposition and formation of an unprecedented stabilized acyl-enzyme.
Rodkey EA, McLeod DC, Bethel CR, Smith KM, Xu Y, Chai W, Che T, Carey PR, Bonomo RA, van den Akker F, Buynak JD. Rodkey EA, et al. J Am Chem Soc. 2013 Dec 11;135(49):18358-69. doi: 10.1021/ja403598g. Epub 2013 Dec 3. J Am Chem Soc. 2013. PMID: 24219313 Free PMC article. - Association of plasmid-mediated quinolone resistance with extended-spectrum beta-lactamase VEB-1.
Poirel L, Van De Loo M, Mammeri H, Nordmann P. Poirel L, et al. Antimicrob Agents Chemother. 2005 Jul;49(7):3091-4. doi: 10.1128/AAC.49.7.3091-3094.2005. Antimicrob Agents Chemother. 2005. PMID: 15980408 Free PMC article. - Multicity outbreak of carbapenem-resistant Acinetobacter baumannii isolates producing the carbapenemase OXA-40.
Lolans K, Rice TW, Munoz-Price LS, Quinn JP. Lolans K, et al. Antimicrob Agents Chemother. 2006 Sep;50(9):2941-5. doi: 10.1128/AAC.00116-06. Antimicrob Agents Chemother. 2006. PMID: 16940085 Free PMC article.
References
- Afzal-Shah, M., and D. M. Livermore. 1998. Worldwide emergence of carbapenem-resistant Acinetobacter spp. J. Antimicrob. Chemother. 41:576-577. - PubMed
- Afzal-Shah, M., H. E. Villar, and D. M. Livermore. 1999. Biochemical characteristics of a carbapenemase from an Acinetobacter baumannii isolate collected in Buenos Aires, Argentina. J. Antimicrob. Chemother. 43:127-131. - PubMed
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources