Membrane-insertion fragments of Bcl-xL, Bax, and Bid - PubMed (original) (raw)
. 2004 Aug 31;43(34):10930-43.
doi: 10.1021/bi036044c.
Affiliations
- PMID: 15323553
- DOI: 10.1021/bi036044c
Membrane-insertion fragments of Bcl-xL, Bax, and Bid
Ana J García-Sáez et al. Biochemistry. 2004.
Abstract
Apoptosis regulators of the Bcl-2 family associate with intracellular membranes from mitochondria and the endoplasmic reticulum, where they perform their function. The activity of these proteins is related to the release of apoptogenic factors, sequestered in the mitochondria, to the cytoplasm, probably through the formation of ion and/or protein transport channels. Most of these proteins contain a C-terminal putative transmembrane (TM) fragment and a pair of hydrophobic alpha helices (alpha5-alpha6) similar to the membrane insertion fragments of the ion-channel domain of diphtheria toxin and colicins. Here, we report on the membrane-insertion properties of different segments from antiapoptotic Bcl-x(L) and proapoptotic Bax and Bid, that correspond to defined alpha helices in the structure of their soluble forms. According to prediction methods, there are only two putative TM fragments in Bcl-x(L) and Bax (the C-terminal alpha helix and alpha-helix 5) and one in activated tBid (alpha-helix 6). The rest of their sequence, including the second helix of the pore-forming domain, displays only weak hydrophobic peaks, which are below the prediction threshold. Subsequent analysis by glycosylation mapping of single alpha-helix segments in a model chimeric system confirms the above predictions and allows finding an extra TM fragment made of helix alpha1 of Bax. Surprisingly, the amphipathic helices alpha6 of Bcl-x(L) and Bax and alpha7 of Bid do insert in membranes only as part of the alpha5-alpha6 (Bcl-x(L) and Bax) or alpha6-alpha7 (Bid) hairpins but not when assayed individually. This behavior suggests a synergistic insertion and folding of the two helices of the hairpin that could be due to charge complementarity and additional stability provided by turn-inducing residues present at the interhelical region. Although these data come from chimeric systems, they show direct potentiality for acquiring a membrane inserted state. Thus, the above fragments should be considered for the definition of plausible models of the active, membrane-bound species of Bcl-2 proteins.
Similar articles
- Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis.
Chipuk JE, Kuwana T, Bouchier-Hayes L, Droin NM, Newmeyer DD, Schuler M, Green DR. Chipuk JE, et al. Science. 2004 Feb 13;303(5660):1010-4. doi: 10.1126/science.1092734. Science. 2004. PMID: 14963330 - Investigation of the role of the C-terminus of Bax and of tc-Bid on Bax interaction with yeast mitochondria.
Priault M, Cartron PF, Camougrand N, Antonsson B, Vallette FM, Manon S. Priault M, et al. Cell Death Differ. 2003 Sep;10(9):1068-77. doi: 10.1038/sj.cdd.4401270. Cell Death Differ. 2003. PMID: 12934081 - Bax forms multispanning monomers that oligomerize to permeabilize membranes during apoptosis.
Annis MG, Soucie EL, Dlugosz PJ, Cruz-Aguado JA, Penn LZ, Leber B, Andrews DW. Annis MG, et al. EMBO J. 2005 Jun 15;24(12):2096-103. doi: 10.1038/sj.emboj.7600675. Epub 2005 May 26. EMBO J. 2005. PMID: 15920484 Free PMC article. - Structural biology of the Bcl-2 family of proteins.
Petros AM, Olejniczak ET, Fesik SW. Petros AM, et al. Biochim Biophys Acta. 2004 Mar 1;1644(2-3):83-94. doi: 10.1016/j.bbamcr.2003.08.012. Biochim Biophys Acta. 2004. PMID: 14996493 Review. - Bid: a Bax-like BH3 protein.
Billen LP, Shamas-Din A, Andrews DW. Billen LP, et al. Oncogene. 2008 Dec;27 Suppl 1:S93-104. doi: 10.1038/onc.2009.47. Oncogene. 2008. PMID: 19641510 Review.
Cited by
- Mechanisms of BCL-2 family proteins in mitochondrial apoptosis.
Czabotar PE, Garcia-Saez AJ. Czabotar PE, et al. Nat Rev Mol Cell Biol. 2023 Oct;24(10):732-748. doi: 10.1038/s41580-023-00629-4. Epub 2023 Jul 12. Nat Rev Mol Cell Biol. 2023. PMID: 37438560 Review. - Apoptotic mitochondrial poration by a growing list of pore-forming BCL-2 family proteins.
Moldoveanu T. Moldoveanu T. Bioessays. 2023 Mar;45(3):e2200221. doi: 10.1002/bies.202200221. Epub 2023 Jan 17. Bioessays. 2023. PMID: 36650950 Free PMC article. - The BCL-2 family member BID plays a role during embryonic development in addition to its BH3-only protein function by acting in parallel to BAX, BAK and BOK.
Ke FS, Holloway S, Uren RT, Wong AW, Little MH, Kluck RM, Voss AK, Strasser A. Ke FS, et al. EMBO J. 2022 Aug 1;41(15):e110300. doi: 10.15252/embj.2021110300. Epub 2022 Jun 27. EMBO J. 2022. PMID: 35758142 Free PMC article. - Accessing local structural disruption of Bid protein during thermal denaturation by absorption-mode ESR spectroscopy.
Hung CL, Lin YY, Chang HH, Chiang YW. Hung CL, et al. RSC Adv. 2018 Oct 9;8(60):34656-34669. doi: 10.1039/c8ra06740f. eCollection 2018 Oct 4. RSC Adv. 2018. PMID: 35548640 Free PMC article. - Bcl-2 Family Members and the Mitochondrial Import Machineries: The Roads to Death.
Lalier L, Vallette F, Manon S. Lalier L, et al. Biomolecules. 2022 Jan 19;12(2):162. doi: 10.3390/biom12020162. Biomolecules. 2022. PMID: 35204663 Free PMC article. Review.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Research Materials