Cyclooxygenase-2 and epidermal growth factor receptor: pharmacologic targets for chemoprevention - PubMed (original) (raw)
Review
. 2005 Jan 10;23(2):254-66.
doi: 10.1200/JCO.2005.09.112.
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- PMID: 15637389
- DOI: 10.1200/JCO.2005.09.112
Review
Cyclooxygenase-2 and epidermal growth factor receptor: pharmacologic targets for chemoprevention
Andrew J Dannenberg et al. J Clin Oncol. 2005.
Abstract
Understanding the mechanisms underlying carcinogenesis provides insights that are necessary for the development of therapeutic strategies to prevent cancer. Chemoprevention, the use of drugs or natural substances to inhibit carcinogenesis, is a rapidly evolving aspect of cancer research. Evidence is presented that cyclooxygenase-2 (COX-2) and epidermal growth factor receptor (EGFR) are potential pharmacologic targets to prevent cancer. In this paper, we review key data implicating a causal relationship between COX-2, EGFR, and carcinogenesis and possible mechanisms of action. We discuss evidence of crosstalk between COX-2 and EGFR in order to strengthen the rationale for combination chemoprevention, and review plans for a clinical trial that will evaluate the concept of combination chemoprevention targeting COX-2 and EGFR.
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- P01 CA 10645 1/CA/NCI NIH HHS/United States
- P01 CA 77839/CA/NCI NIH HHS/United States
- R01 89578/PHS HHS/United States
- R01 CA 82578/CA/NCI NIH HHS/United States
- R01 DK 62112/DK/NIDDK NIH HHS/United States
- R37 DK 47297/DK/NIDDK NIH HHS/United States
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