Physiology of microglial cells - PubMed (original) (raw)
Review
Physiology of microglial cells
Katrin Färber et al. Brain Res Brain Res Rev. 2005 Apr.
Abstract
Microglial cells in culture and in situ express a defined pattern of K(+) channels, which is distinct from that of other glial cells and neurons. This pattern undergoes defined changes with microglial activation. As expected for a cell with immunological properties, microglia express a variety of cytokine and chemokine receptors, which are linked to the mobilization of Ca(2+) (cytosolic free calcium) from internal stores. Microglial cells also have the capacity to respond to neuronal activity: they express receptors for the major excitatory receptor glutamate and the main inhibitory receptor GABA (gamma-amino butyric acid). By expressing purinergic receptors, microglia can sense astrocyte activity in the form of Ca(2+) waves. Activation of transmitter receptors can affect cytokine release which is a potential means as to how brain activity can affect immune function.
Similar articles
- Microglia express GABA(B) receptors to modulate interleukin release.
Kuhn SA, van Landeghem FK, Zacharias R, Färber K, Rappert A, Pavlovic S, Hoffmann A, Nolte C, Kettenmann H. Kuhn SA, et al. Mol Cell Neurosci. 2004 Feb;25(2):312-22. doi: 10.1016/j.mcn.2003.10.023. Mol Cell Neurosci. 2004. PMID: 15019947 - Synaptically-silent immature neurons show gaba and glutamate receptor-mediated currents in adult rat dentate gyrus.
Ambrogini P, Minelli A, Lattanzi D, Ciuffoli S, Fanelli M, Cuppini R. Ambrogini P, et al. Arch Ital Biol. 2006 May;144(2):115-26. Arch Ital Biol. 2006. PMID: 16642790 - Microglial neurotransmitter receptors trigger superoxide production in microglia; consequences for microglial-neuronal interactions.
Mead EL, Mosley A, Eaton S, Dobson L, Heales SJ, Pocock JM. Mead EL, et al. J Neurochem. 2012 Apr;121(2):287-301. doi: 10.1111/j.1471-4159.2012.07659.x. Epub 2012 Mar 13. J Neurochem. 2012. PMID: 22243365 - The function of microglia through purinergic receptors: neuropathic pain and cytokine release.
Inoue K. Inoue K. Pharmacol Ther. 2006 Jan;109(1-2):210-26. doi: 10.1016/j.pharmthera.2005.07.001. Epub 2005 Sep 19. Pharmacol Ther. 2006. PMID: 16169595 Review. - Purinergic receptors modulate MAP kinases and transcription factors that control microglial inflammatory gene expression.
Potucek YD, Crain JM, Watters JJ. Potucek YD, et al. Neurochem Int. 2006 Jul;49(2):204-14. doi: 10.1016/j.neuint.2006.04.005. Epub 2006 Jun 2. Neurochem Int. 2006. PMID: 16735081 Review.
Cited by
- Involvement of Kv1.3 and p38 MAPK signaling in HIV-1 glycoprotein 120-induced microglia neurotoxicity.
Liu J, Xu C, Chen L, Xu P, Xiong H. Liu J, et al. Cell Death Dis. 2012 Jan 19;3(1):e254. doi: 10.1038/cddis.2011.140. Cell Death Dis. 2012. PMID: 22258405 Free PMC article. - Microglia as a pharmacological target in infectious and inflammatory diseases of the brain.
Rock RB, Peterson PK. Rock RB, et al. J Neuroimmune Pharmacol. 2006 Jun;1(2):117-26. doi: 10.1007/s11481-006-9012-8. J Neuroimmune Pharmacol. 2006. PMID: 18040778 Review. - Microglial activation underlies cerebellar deficits produced by repeated cannabis exposure.
Cutando L, Busquets-Garcia A, Puighermanal E, Gomis-González M, Delgado-García JM, Gruart A, Maldonado R, Ozaita A. Cutando L, et al. J Clin Invest. 2013 Jul;123(7):2816-31. doi: 10.1172/JCI67569. Epub 2013 Jun 24. J Clin Invest. 2013. PMID: 23934130 Free PMC article. - Neuronal and microglial mechanisms of neuropathic pain.
Zhuo M, Wu G, Wu LJ. Zhuo M, et al. Mol Brain. 2011 Jul 30;4:31. doi: 10.1186/1756-6606-4-31. Mol Brain. 2011. PMID: 21801430 Free PMC article. Review. - Elevated TRPV4 Levels Contribute to Endothelial Damage and Scarring in Experimental Spinal Cord Injury.
Kumar H, Lim CS, Choi H, Joshi HP, Kim KT, Kim YH, Park CK, Kim HM, Han IB. Kumar H, et al. J Neurosci. 2020 Feb 26;40(9):1943-1955. doi: 10.1523/JNEUROSCI.2035-19.2020. Epub 2020 Jan 23. J Neurosci. 2020. PMID: 31974206 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Research Materials
Miscellaneous