Selenium, oxidative stress, and health aspects - PubMed (original) (raw)
Review
. 2005 Aug-Oct;26(4-5):256-67.
doi: 10.1016/j.mam.2005.07.004.
Affiliations
- PMID: 16105679
- DOI: 10.1016/j.mam.2005.07.004
Review
Selenium, oxidative stress, and health aspects
Peter Brenneisen et al. Mol Aspects Med. 2005 Aug-Oct.
Abstract
Metabolic processes which generate oxidants and antioxidants are governed by genetic disposition as well as environmental factors. Changes in lifestyle, including increased environmental pollution, sun exposure, and dietary habits modify the challenge of the organism by reactive oxygen species. Defense mechanisms are reinforced by increasing dietary intake of antioxidants and micronutrients such as vitamins and selenium (Se). Se deficiency has been recognized to promote some disease states. Epidemiological findings link a lowered Se status to neurodegenerative and cardiovascular diseases as well as to increased cancer risk. While evidence exists to suggest that additional selenocompounds would be beneficial in some health conditions, results from future intervention trials are needed to substantiate the argument for increasing Se intake. Several pieces of the puzzle concerning the molecular mechanisms underlying the reactive oxygen species-triggered disease state and intervention by enzymatic antioxidants have been elucidated. A novel concept of protection of stromal cells against the dominating influence of tumor cells in tumor-stroma interaction by selenocompounds and other antioxidants is presented herein, which may translate into therapeutic strategies in chemoprevention of tumor invasion.
Similar articles
- Cancer chemoprevention: selenium as a prooxidant, not an antioxidant.
Drake EN. Drake EN. Med Hypotheses. 2006;67(2):318-22. doi: 10.1016/j.mehy.2006.01.058. Epub 2006 Mar 30. Med Hypotheses. 2006. PMID: 16574336 - The argument for increasing selenium intake.
Rayman MP, Rayman MP. Rayman MP, et al. Proc Nutr Soc. 2002 May;61(2):203-15. doi: 10.1079/PNS2002153. Proc Nutr Soc. 2002. PMID: 12133202 Review. - Increased consumption of wheat biofortified with selenium does not modify biomarkers of cancer risk, oxidative stress, or immune function in healthy Australian males.
Wu J, Salisbury C, Graham R, Lyons G, Fenech M. Wu J, et al. Environ Mol Mutagen. 2009 Jul;50(6):489-501. doi: 10.1002/em.20490. Environ Mol Mutagen. 2009. PMID: 19365873 Clinical Trial. - Free radicals, metals and antioxidants in oxidative stress-induced cancer.
Valko M, Rhodes CJ, Moncol J, Izakovic M, Mazur M. Valko M, et al. Chem Biol Interact. 2006 Mar 10;160(1):1-40. doi: 10.1016/j.cbi.2005.12.009. Epub 2006 Jan 23. Chem Biol Interact. 2006. PMID: 16430879 Review. - A review of the interaction among dietary antioxidants and reactive oxygen species.
Seifried HE, Anderson DE, Fisher EI, Milner JA. Seifried HE, et al. J Nutr Biochem. 2007 Sep;18(9):567-79. doi: 10.1016/j.jnutbio.2006.10.007. Epub 2007 Mar 23. J Nutr Biochem. 2007. PMID: 17360173 Review.
Cited by
- Managing Cardiovascular Risk in Patients with Autoimmune Diseases: Insights from a Nutritional Perspective.
Chen H, Liu L, Wang Y, Hong L, Pan J, Yu X, Dai H. Chen H, et al. Curr Nutr Rep. 2024 Dec;13(4):718-728. doi: 10.1007/s13668-024-00563-7. Epub 2024 Jul 30. Curr Nutr Rep. 2024. PMID: 39078574 Review. - Metallomic analysis of brain tissues distinguishes between cases of dementia with Lewy bodies, Alzheimer's disease, and Parkinson's disease dementia.
Scholefield M, Church SJ, Xu J, Cooper GJS. Scholefield M, et al. Front Neurosci. 2024 Jun 26;18:1412356. doi: 10.3389/fnins.2024.1412356. eCollection 2024. Front Neurosci. 2024. PMID: 38988772 Free PMC article. - Dietary patterns; serum concentrations of selenium, copper, and zinc; copper/zinc ratio; and total antioxidant status in patients with glaucoma.
Zawadzka I, Młynarczyk M, Falkowska M, Socha K, Konopińska J. Zawadzka I, et al. PLoS One. 2024 Apr 2;19(4):e0301511. doi: 10.1371/journal.pone.0301511. eCollection 2024. PLoS One. 2024. PMID: 38564509 Free PMC article. - The effect of selenium on the proliferation of bovine endometrial epithelial cells in a lipopolysaccharide-induced damage model.
Li H, Wang H, Cui L, Liu K, Guo L, Li J, Dong J. Li H, et al. BMC Vet Res. 2024 Mar 18;20(1):109. doi: 10.1186/s12917-024-03958-4. BMC Vet Res. 2024. PMID: 38500165 Free PMC article. - Methylseleninic acid inhibits human glioma growth in vitro and in vivo by triggering ROS-dependent oxidative damage and apoptosis.
Chen W, Hao P, Song Q, Feng X, Zhao X, Wu J, Gong Z, Zhang J, Fu X, Wang X. Chen W, et al. Metab Brain Dis. 2024 Apr;39(4):625-633. doi: 10.1007/s11011-024-01344-5. Epub 2024 Feb 28. Metab Brain Dis. 2024. PMID: 38416338
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources