Search for localized dysbiosis in Crohn's disease ulcerations by temporal temperature gradient gel electrophoresis of 16S rRNA - PubMed (original) (raw)
Search for localized dysbiosis in Crohn's disease ulcerations by temporal temperature gradient gel electrophoresis of 16S rRNA
Philippe Seksik et al. J Clin Microbiol. 2005 Sep.
Abstract
The mucosa-associated microbiota lining the gut epithelium might play a central role in the activation and/or perpetuation of mucosal inflammation in Crohn's disease (CD). We sought for localized dysbiosis by comparing the biodiversity and composition of the microbiotas in ulcerated and nonulcerated mucosal samples from patients with CD. Biopsy samples (n = 75) of ulcerated and adjacent nonulcerated mucosa were collected during colonoscopy from 15 patients, from the ileum, right colon, left colon, and rectum. Temporal temperature gradient gel electrophoresis (TTGE) of bacterial 16S rRNAs was used to evaluate the dominant bacterial species. TTGE profiles were compared using software that calculates similarity percentages. For a given patient, average similarity indexes between ulcerated and nonulcerated mucosal TTGE profiles ranged from 95.2% +/- 4.2% to 97.9% +/- 1.7% (means +/- standard deviations) for the different segments. The mean values did not differ significantly. Average interindividual similarity indexes for a given segment among the different patients ranged from 33.6% +/- 15.5% to 42.0% +/- 25.6%. In CD, the dominant microbiotas do not differ qualitatively between ulcerated and nonulcerated mucosae. Biodiversity remains high in ulcerated mucosa. This argues against a pathogenic role of localized qualitative dysbiosis in CD-associated ulceration.
Figures
FIG. 1.
TTGE of 16S rRNA gene amplicons (obtained using primers for the V6-V8 region) amplified from biopsy samples of ulcerated mucosa (UM) and nonulcerated mucosa (NUM) from the left colons of five CD patients. UM X, TTGE profile for UM of patient X; NUM X, TTGE profile for NUM of patient X. (Center) TTGE profiles ordered by Gel Compar II software. UM and NUM TTGE profiles for a given patient were always more similar than UM and NUM profiles for different patients. (Right) Pearson correlation coefficients yielded the calculated similarity indexes (expressed as percentages) of paired samples. (Left) The dendrogram represents a statistically optimal representation of the similarities between TTGE profiles based on the matrix of Pearson correlation coefficients and applying UPGMA.
FIG. 2.
Dendrogram representation of the TTGE profiles of 16S rRNA gene amplicons (obtained using primers for the V6-V8 region) amplified from biopsy samples of ulcerated mucosa (UM) and nonulcerated mucosa (NUM) from 15 CD patients. Biopsy samples were collected from the ileum (I), right colon (RC), left colon (LF), and rectum (R) of each patient. Gray designations, ulcerated mucosa; black designations, nonulcerated mucosa. The dendrogram represents a statistically optimal representation of the similarities between TTGE profiles based on the matrix of Pearson correlation coefficients and applying UPGMA. The vertical dotted line represents the threshold defining a cluster (80%).
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