Changes in osmolality sensitize the response to capsaicin in trigeminal sensory neurons - PubMed (original) (raw)
. 2007 Mar;97(3):2001-15.
doi: 10.1152/jn.00887.2006.
Affiliations
- PMID: 17353553
- DOI: 10.1152/jn.00887.2006
Free article
Changes in osmolality sensitize the response to capsaicin in trigeminal sensory neurons
Lieju Liu et al. J Neurophysiol. 2007 Mar.
Free article
Abstract
Changes in tonicity in the peripheral nervous system can activate nociceptors and produce pain. Under local inflammatory conditions the peripheral terminals of nociceptors are subject to deviations from isotonicity. Previously it was shown that several members of the TRP(V) family of ion channels are responsive to changes in tonicity. Here we explore how changes in tonicity affect TRPV1 receptor-mediated responses to capsaicin in dissociated rat trigeminal ganglion (TG) neurons. Using whole cell patch-clamp and calcium imaging, we found that mild anisotonicity (260 and 348 mOsm/kg for hypotonicity and hypertonicity, respectively) strikingly sensitized the capsaicin-evoked current, I(caps). Confocal immunolocalization studies also revealed a modest anisotonicity-mediated redistribution of TRPV1 toward the plasma membrane of TG neurons. With respect to downstream signaling pathways, tonicity-induced sensitization of I(caps) was dependent on whether hypo- or hypertonic stimuli were applied. Specifically, antagonism of PKA- and PI3K-activated pathways appreciably reduced the hypertonicity-induced sensitization of I(caps), whereas inhibition of PKC-mediated pathways selectively reduced the sensitization produced by hypotonic solutions. In summary, whereas the overall effects of hypo- and hypertonicity resulted in a similar pattern of potentiation of I(caps), intracellular signaling pathways were selective for hypo- versus hypertonicity-induced tuning of capsaicin-activated currents.
Similar articles
- Modulation of IA currents by capsaicin in rat trigeminal ganglion neurons.
Liu L, Simon SA. Liu L, et al. J Neurophysiol. 2003 Mar;89(3):1387-401. doi: 10.1152/jn.00210.2002. J Neurophysiol. 2003. PMID: 12626618 - Effects of capsaicin on VGSCs in TRPV1-/- mice.
Cao X, Cao X, Xie H, Yang R, Lei G, Li F, Li A, Liu C, Liu L. Cao X, et al. Brain Res. 2007 Aug 13;1163:33-43. doi: 10.1016/j.brainres.2007.04.085. Epub 2007 Jun 2. Brain Res. 2007. PMID: 17632091 - Sensory neuron-specific actions of capsaicin: mechanisms and applications.
Bevan S, Szolcsányi J. Bevan S, et al. Trends Pharmacol Sci. 1990 Aug;11(8):330-3. doi: 10.1016/0165-6147(90)90237-3. Trends Pharmacol Sci. 1990. PMID: 2203194 Review. - Hypertonicity: Pathophysiologic Concept and Experimental Studies.
Argyropoulos C, Rondon-Berrios H, Raj DS, Malhotra D, Agaba EI, Rohrscheib M, Khitan Z, Murata GH, Shapiro JI, Tzamaloukas AH. Argyropoulos C, et al. Cureus. 2016 May 2;8(5):e596. doi: 10.7759/cureus.596. Cureus. 2016. PMID: 27382523 Free PMC article. Review.
Cited by
- Luminal hypertonicity and acidity modulate colorectal afferents and induce persistent visceral hypersensitivity.
La JH, Feng B, Schwartz ES, Brumovsky PR, Gebhart GF. La JH, et al. Am J Physiol Gastrointest Liver Physiol. 2012 Oct;303(7):G802-9. doi: 10.1152/ajpgi.00259.2012. Epub 2012 Aug 2. Am J Physiol Gastrointest Liver Physiol. 2012. PMID: 22859365 Free PMC article. - Improving NKCC1 Function Increases the Excitability of DRG Neurons Exacerbating Pain Induced After TRPV1 Activation of Primary Sensory Neurons.
Deng SY, Tang XC, Chang YC, Xu ZZ, Chen QY, Cao N, Kong LJ, Wang Y, Ma KT, Li L, Si JQ. Deng SY, et al. Front Cell Neurosci. 2021 May 25;15:665596. doi: 10.3389/fncel.2021.665596. eCollection 2021. Front Cell Neurosci. 2021. PMID: 34113239 Free PMC article. - Gustatory modulation of the responses of trigeminal subnucleus caudalis neurons to noxious stimulation of the tongue in rats.
Boucher Y, Felizardo R, Klein AH, Carstens MI, Carstens E. Boucher Y, et al. Eur J Neurosci. 2013 Sep;38(6):2812-22. doi: 10.1111/ejn.12282. Epub 2013 Jun 27. Eur J Neurosci. 2013. PMID: 23802589 Free PMC article. - The role of urea in neuronal degeneration and sensitization: An in vitro model of uremic neuropathy.
Anand U, Korchev Y, Anand P. Anand U, et al. Mol Pain. 2019 Jan-Dec;15:1744806919881038. doi: 10.1177/1744806919881038. Mol Pain. 2019. PMID: 31549574 Free PMC article. - Control of excitatory synaptic transmission by capsaicin is unaltered in TRPV1 vanilloid receptor knockout mice.
Benninger F, Freund TF, Hájos N. Benninger F, et al. Neurochem Int. 2008 Jan;52(1-2):89-94. doi: 10.1016/j.neuint.2007.06.008. Epub 2007 Jun 21. Neurochem Int. 2008. PMID: 17651868 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Medical
Miscellaneous