Emergence of a novel swine-origin influenza A (H1N1) virus in humans - PubMed (original) (raw)
. 2009 Jun 18;360(25):2605-15.
doi: 10.1056/NEJMoa0903810. Epub 2009 May 7.
Collaborators
- PMID: 19423869
- DOI: 10.1056/NEJMoa0903810
Free article
Emergence of a novel swine-origin influenza A (H1N1) virus in humans
Novel Swine-Origin Influenza A (H1N1) Virus Investigation Team et al. N Engl J Med. 2009.
Free article
Erratum in
- N Engl J Med. 2009 Jul 2;361(1):102
Abstract
Background: On April 15 and April 17, 2009, novel swine-origin influenza A (H1N1) virus (S-OIV) was identified in specimens obtained from two epidemiologically unlinked patients in the United States. The same strain of the virus was identified in Mexico, Canada, and elsewhere. We describe 642 confirmed cases of human S-OIV infection identified from the rapidly evolving U.S. outbreak.
Methods: Enhanced surveillance was implemented in the United States for human infection with influenza A viruses that could not be subtyped. Specimens were sent to the Centers for Disease Control and Prevention for real-time reverse-transcriptase-polymerase-chain-reaction confirmatory testing for S-OIV.
Results: From April 15 through May 5, a total of 642 confirmed cases of S-OIV infection were identified in 41 states. The ages of patients ranged from 3 months to 81 years; 60% of patients were 18 years of age or younger. Of patients with available data, 18% had recently traveled to Mexico, and 16% were identified from school outbreaks of S-OIV infection. The most common presenting symptoms were fever (94% of patients), cough (92%), and sore throat (66%); 25% of patients had diarrhea, and 25% had vomiting. Of the 399 patients for whom hospitalization status was known, 36 (9%) required hospitalization. Of 22 hospitalized patients with available data, 12 had characteristics that conferred an increased risk of severe seasonal influenza, 11 had pneumonia, 8 required admission to an intensive care unit, 4 had respiratory failure, and 2 died. The S-OIV was determined to have a unique genome composition that had not been identified previously.
Conclusions: A novel swine-origin influenza A virus was identified as the cause of outbreaks of febrile respiratory infection ranging from self-limited to severe illness. It is likely that the number of confirmed cases underestimates the number of cases that have occurred.
2009 Massachusetts Medical Society
Comment in
- Implications of the emergence of a novel H1 influenza virus.
Belshe RB. Belshe RB. N Engl J Med. 2009 Jun 18;360(25):2667-8. doi: 10.1056/NEJMe0903995. Epub 2009 May 7. N Engl J Med. 2009. PMID: 19423870 No abstract available.
Similar articles
- Triple-reassortant swine influenza A (H1) in humans in the United States, 2005-2009.
Shinde V, Bridges CB, Uyeki TM, Shu B, Balish A, Xu X, Lindstrom S, Gubareva LV, Deyde V, Garten RJ, Harris M, Gerber S, Vagasky S, Smith F, Pascoe N, Martin K, Dufficy D, Ritger K, Conover C, Quinlisk P, Klimov A, Bresee JS, Finelli L. Shinde V, et al. N Engl J Med. 2009 Jun 18;360(25):2616-25. doi: 10.1056/NEJMoa0903812. Epub 2009 May 7. N Engl J Med. 2009. PMID: 19423871 - Update: swine influenza A (H1N1) infections--California and Texas, April 2009.
Centers for Disease Control and Prevention (CDC). Centers for Disease Control and Prevention (CDC). MMWR Morb Mortal Wkly Rep. 2009 May 1;58(16):435-7. MMWR Morb Mortal Wkly Rep. 2009. PMID: 19407739 - Clinical and microbiologic features associated with novel swine-origin influenza A pandemic 2009 (H1N1) virus in children: a prospective cohort study.
Bryant PA, Tebruegge M, Papadakis G, Clarke C, Barnett P, Daley AJ, South M, Curtis N. Bryant PA, et al. Pediatr Infect Dis J. 2010 Aug;29(8):694-8. doi: 10.1097/INF.0b013e3181de4b9c. Pediatr Infect Dis J. 2010. PMID: 20458257 - The 2009 A (H1N1) influenza virus pandemic: A review.
Girard MP, Tam JS, Assossou OM, Kieny MP. Girard MP, et al. Vaccine. 2010 Jul 12;28(31):4895-902. doi: 10.1016/j.vaccine.2010.05.031. Epub 2010 May 27. Vaccine. 2010. PMID: 20553769 Review. - Pandemic novel 2009 H1N1 influenza: what have we learned?
LaRussa P. LaRussa P. Semin Respir Crit Care Med. 2011 Aug;32(4):393-9. doi: 10.1055/s-0031-1283279. Epub 2011 Aug 19. Semin Respir Crit Care Med. 2011. PMID: 21858744 Review.
Cited by
- The Central Role of the Global Emerging Infections Surveillance Program in Supporting Force Health Protection.
Early JM, Smith HJ, Cinkovich SS, Gallaway MS, Stager CN, Kasper MR. Early JM, et al. Emerg Infect Dis. 2024 Oct;30(14):3-8. doi: 10.3201/eid3014.240304. Emerg Infect Dis. 2024. PMID: 39530772 Free PMC article. - Editorial: The synthesis of secretory immunoglobulin A in mucosal tissue: mucosal-associated invariant T, T follicular helper, and B cells.
Booth JS, Wahid R, Bruder D, Salerno-Goncalves R. Booth JS, et al. Front Immunol. 2024 Oct 17;15:1504432. doi: 10.3389/fimmu.2024.1504432. eCollection 2024. Front Immunol. 2024. PMID: 39483477 Free PMC article. No abstract available. - Antiviral Susceptibility of Swine-Origin Influenza A Viruses Isolated from Humans, United States.
Gao R, Pascua PNQ, Chesnokov A, Nguyen HT, Uyeki TM, Mishin VP, Zanders N, Cui D, Jang Y, Jones J, La Cruz J, Di H, Davis CT, Gubareva LV. Gao R, et al. Emerg Infect Dis. 2024 Nov;30(11):2303-2312. doi: 10.3201/eid3011.240892. Epub 2024 Oct 8. Emerg Infect Dis. 2024. PMID: 39378870 Free PMC article. - Metabolic Dependency Shapes Bivalent Antiviral Response in Host Cells in Response to Poly:IC: The Role of Glutamine.
Lebeau G, Paulo-Ramos A, Hoareau M, El Safadi D, Meilhac O, Krejbich-Trotot P, Roche M, Viranaicken W. Lebeau G, et al. Viruses. 2024 Aug 30;16(9):1391. doi: 10.3390/v16091391. Viruses. 2024. PMID: 39339867 Free PMC article. - LINC01197 inhibits influenza A virus replication by serving as a PABPC1 decoy.
Wang Y, Shi N, Zhang H, Luo J, Yan H, Hou H, Guan Z, Zhao L, Duan M. Wang Y, et al. Vet Res. 2024 Sep 27;55(1):121. doi: 10.1186/s13567-024-01379-7. Vet Res. 2024. PMID: 39334466 Free PMC article.
MeSH terms
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical
Miscellaneous