Just when you thought it was safe to go into the membrane: the growing complexities of extra-nuclear progesterone signaling - PubMed (original) (raw)
Editorial
Just when you thought it was safe to go into the membrane: the growing complexities of extra-nuclear progesterone signaling
Aritro Sen et al. Breast Cancer Res. 2010.
Abstract
The diversity of membrane-initiated progesterone actions has made characterization and establishment of its biological importance a complicated endeavor. A new study by Zuo and colleagues shows that progesterone via endogenous membrane progesterone receptor-alpha acts as a negative regulator of proliferation and epithelial to mesenchymal transition in a breast cancer cell line. These progesterone-mediated actions appear to be regulated through epidermal growth factor receptor and phosphatidylinositol 3-kinase signaling localized in caveolae. Moreover, the study shows expression of membrane progesterone receptor-alpha in benign and malignant breast cancer tissues. These data bring forth novel concepts with regard to progesterone actions in the breast; however, further work is warranted to fully characterize the physiologic actions of extra-nuclear progesterone signaling in the breast.
Comment on
- Progesterone reverses the mesenchymal phenotypes of basal phenotype breast cancer cells via a membrane progesterone receptor mediated pathway.
Zuo L, Li W, You S. Zuo L, et al. Breast Cancer Res. 2010;12(3):R34. doi: 10.1186/bcr2588. Epub 2010 Jun 11. Breast Cancer Res. 2010. PMID: 20540763 Free PMC article.
References
- Krietsch T, Fernandes MS, Kero J, Losel R, Heyens M, Lam EW, Huhtaniemi I, Brosens JJ, Gellersen B. Human homologs of the putative G proteincoupled membrane progestin receptors (mPRalpha, beta, and gamma) localize to the endoplasmic reticulum and are not activated by progesterone. Mol Endocrinol. 2006;20:3146–3164. doi: 10.1210/me.2006-0129. - DOI - PubMed
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