The ins and outs of the epithelial to mesenchymal transition in health and disease - PubMed (original) (raw)
Review
The ins and outs of the epithelial to mesenchymal transition in health and disease
M Angela Nieto. Annu Rev Cell Dev Biol. 2011.
Abstract
The epithelial to mesenchymal transition (EMT) converts epithelial cells into migratory and invasive cells and is a fundamental event in morphogenesis. Although its relevance in the progression of cancer and organ fibrosis had been debated until recently, the EMT is now established as an important step in the metastatic cascade of epithelial tumors. The similarities between pathological and developmental EMTs validate the embryo as the best model to understand the molecular and cellular mechanisms involved in this process, identifying those that are hijacked during the progression of cancer and organ degeneration. Our ever-increasing understanding of how transcription factors regulate the EMT has revealed complex regulatory loops coupled to posttranscriptional and epigenetic regulatory programs. The EMT is now integrated into the systemic activities of whole organisms, establishing links with cell survival, stemness, inflammation, and immunity. In addition, the EMT now constitutes a promising target for the treatment of cancer and organ-degenerative diseases.
Similar articles
- [Epithelial-mesenchymal transition in cancer progression].
Gos M, Miłoszewska J, Przybyszewska M. Gos M, et al. Postepy Biochem. 2009;55(2):121-8. Postepy Biochem. 2009. PMID: 19824467 Review. Polish. - Reprogramming during epithelial to mesenchymal transition under the control of TGFβ.
Tan EJ, Olsson AK, Moustakas A. Tan EJ, et al. Cell Adh Migr. 2015;9(3):233-46. doi: 10.4161/19336918.2014.983794. Epub 2014 Nov 17. Cell Adh Migr. 2015. PMID: 25482613 Free PMC article. Review. - Epithelial-mesenchymal transition in development and cancer.
Micalizzi DS, Ford HL. Micalizzi DS, et al. Future Oncol. 2009 Oct;5(8):1129-43. doi: 10.2217/fon.09.94. Future Oncol. 2009. PMID: 19852726 Review. - TGF-β signaling and epithelial-mesenchymal transition in cancer progression.
Katsuno Y, Lamouille S, Derynck R. Katsuno Y, et al. Curr Opin Oncol. 2013 Jan;25(1):76-84. doi: 10.1097/CCO.0b013e32835b6371. Curr Opin Oncol. 2013. PMID: 23197193 Review. - [Epithelial-mesenchymal Transition in Tumor Tissue and Its Role for Metastatic Spread of Cancer].
Matějka VM, Fínek J, Králíčková M. Matějka VM, et al. Klin Onkol. 2017 Winter;30(1):20-27. doi: 10.14735/amko201720. Klin Onkol. 2017. PMID: 28185461 Review. Czech.
Cited by
- miR-210, a modulator of hypoxia-induced epithelial-mesenchymal transition in ovarian cancer cell.
Ding L, Zhao L, Chen W, Liu T, Li Z, Li X. Ding L, et al. Int J Clin Exp Med. 2015 Feb 15;8(2):2299-307. eCollection 2015. Int J Clin Exp Med. 2015. PMID: 25932166 Free PMC article. - Zeb1 regulates E-cadherin and Epcam (epithelial cell adhesion molecule) expression to control cell behavior in early zebrafish development.
Vannier C, Mock K, Brabletz T, Driever W. Vannier C, et al. J Biol Chem. 2013 Jun 28;288(26):18643-59. doi: 10.1074/jbc.M113.467787. Epub 2013 May 10. J Biol Chem. 2013. PMID: 23667256 Free PMC article. - Tissue-based identification of stem cells and epithelial-to-mesenchymal transition in breast cancer.
Anwar TE, Kleer CG. Anwar TE, et al. Hum Pathol. 2013 Aug;44(8):1457-64. doi: 10.1016/j.humpath.2013.01.005. Epub 2013 Apr 8. Hum Pathol. 2013. PMID: 23574782 Free PMC article. Review. - PTTG2 silencing results in induction of epithelial-to-mesenchymal transition and apoptosis.
Méndez-Vidal C, Gámez-Del Estal MM, Moreno-Mateos MA, Espina-Zambrano AG, Torres B, Pintor-Toro JA. Méndez-Vidal C, et al. Cell Death Dis. 2013 Mar 7;4(3):e530. doi: 10.1038/cddis.2013.48. Cell Death Dis. 2013. PMID: 23470537 Free PMC article. - Role of Akt/Protein Kinase B in Cancer Metastasis.
Islam M, Jones S, Ellis I. Islam M, et al. Biomedicines. 2023 Nov 8;11(11):3001. doi: 10.3390/biomedicines11113001. Biomedicines. 2023. PMID: 38002001 Free PMC article. Review.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources