Behavioral and neurochemical pharmacology of six psychoactive substituted phenethylamines: mouse locomotion, rat drug discrimination and in vitro receptor and transporter binding and function - PubMed (original) (raw)
Behavioral and neurochemical pharmacology of six psychoactive substituted phenethylamines: mouse locomotion, rat drug discrimination and in vitro receptor and transporter binding and function
Amy J Eshleman et al. Psychopharmacology (Berl). 2014 Mar.
Abstract
Rationale: Psychoactive-substituted phenethylamines 2,5-dimethoxy-4-chlorophenethylamine (2C-C); 2,5-dimethoxy-4-methylphenethylamine (2C-D); 2,5-dimethoxy-4-ethylphenethylamine (2C-E); 2,5-dimethoxy-4-iodophenethylamine (2C-I); 2,5-dimethoxy-4-ethylthiophenethylamine (2C-T-2); and 2,5-dimethoxy-4-chloroamphetamine (DOC) are used recreationally and may have deleterious side effects.
Objectives: This study compares the behavioral effects and the mechanisms of action of these substituted phenethylamines with those of hallucinogens and a stimulant.
Methods: The effects of these compounds on mouse locomotor activity and in rats trained to discriminate dimethyltryptamine, (-)-DOM, (+)-LSD, (±)-MDMA, and S(+)-methamphetamine were assessed. Binding and functional activity of the phenethylamines at 5-HT1A, 5-HT2A, 5-HT2C receptors and monoamine transporters were assessed using cells heterologously expressing these proteins.
Results: The phenethylamines depressed mouse locomotor activity, although 2C-D and 2C-E stimulated activity at low doses. The phenethylamines except 2C-T-2 fully substituted for at least one hallucinogenic training compound, but none fully substituted for (+)-methamphetamine. At 5-HT1A receptors, only 2C-T-2 and 2C-I were partial-to-full very low potency agonists. In 5-HT2A arachidonic acid release assays, the phenethylamines were partial to full agonists except 2C-I which was an antagonist. All compounds were full agonists at 5-HT2A and 5-HT2C receptor inositol phosphate assays. Only 2C-I had moderate affinity for, and very low potency at, the serotonin transporter.
Conclusions: The discriminative stimulus effects of 2C-C, 2C-D, 2C-E, 2C-I, and DOC were similar to those of several hallucinogens, but not methamphetamine. Additionally, the substituted phenethylamines were full agonists at 5-HT2A and 5-HT2C receptors, but for 2C-T-2, this was not sufficient to produce hallucinogen-like discriminative stimulus effects. Additionally, the 5-HT2A inositol phosphate pathway may be important in 2C-I's psychoactive properties.
Conflict of interest statement
Conflict of interest.
All authors declare that they have no conflicts of interest.
Figures
Fig. 1
Structures of 2C-C, 2C-D, 2C-E, 2C-I, 2C-T-2 and DOC
Fig. 2
Average horizontal activity counts/10 min (ambulation counts) as a function of time (0–8 hr) and dose of test compound. Data for the vehicle and the dose which produced peak depressant effects are shown in each panel. 2C-D and DOC also produced stimulant effects, and data for the dose which produced peak stimulant effects are also shown. N=8 for each treatment.
Fig. 3
[3H]AA release from HEK-5-HT2A cells. Experiments were conducted as described in methods. Data presented are means ± sem. (A) Agonist assay. Basal activity is subtracted, and data are normalized to the maximal stimulation by serotonin on each experimental day. n=3–5 except n=2 for 2C-I. (B) Antagonist assay. Nonspecific release, measured in the presence of 30 μM ketanserin, is subtracted from all data and data are normalized to the maximal release stimulated by serotonin. n=3–4.
Fig. 4
Stimulation of IP-1 formation in HEK-5-HT2A and HEK-5-HT2C cells. Experiments were conducted as described in methods. A. HEK-5-HT2A cells. All compounds are full or partial agonists. The average maximal stimulation by serotonin was 565 ± 46 nM IP1. n=3–8. B. HEK-5-HT2C cells. All compounds are full agonists. The average maximal stimulation by serotonin was 1390 ± 180 nM. n=4–7.
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