Varenicline for treatment of alcohol dependence: a randomized, placebo-controlled trial - PubMed (original) (raw)
Randomized Controlled Trial
. 2015 Nov;39(11):2189-99.
doi: 10.1111/acer.12854. Epub 2015 Sep 28.
Affiliations
- PMID: 26414337
- DOI: 10.1111/acer.12854
Randomized Controlled Trial
Varenicline for treatment of alcohol dependence: a randomized, placebo-controlled trial
Andrea de Bejczy et al. Alcohol Clin Exp Res. 2015 Nov.
Abstract
Background: Alcohol dependence is a devastating illness affecting a large population, and new pharmacological treatments with good efficacy are greatly needed. One potential candidate is varenicline, a smoking cessation agent with partial agonist action at α4 β2 nicotinic acetylcholine receptors.
Methods: A total of 160 subjects, 30 to 70 years of age, fulfilling DSM-IV criteria for alcohol dependence without any serious physical or mental disorders, were recruited through advertisement at 3 university clinics in Sweden during March 2009 to January 2011. After a 2-week placebo run-in period, subjects received 2 mg varenicline daily (titrated from 0.5 mg during first week) or placebo for 12 weeks in a double-blind manner.
Results: The primary outcome was the proportion of heavy drinking days, measured by self-reported alcohol consumption. Primary and secondary outcomes were calculated as a mean over the 10-week steady-state active treatment period. In the primary outcome analysis, no effect of varenicline over placebo was found (p = 0.73 for the intention to treat [ITT] and 0.92 for per protocol [PP]). Secondary outcome analysis found a significant reduction of specific alcohol marker phosphatidylethanol (PEth) in the blood in the varenicline group compared to placebo (p = 0.02 ITT). Craving (p = 0.048 PP) and Alcohol Use Disorders Identification Test (AUDIT) scores (p = 0.015 ITT) were also reduced in the active treatment group. PEth more strongly correlated with self-reported alcohol consumption than carbohydrate-deficient ttransferrin and γ-glutamyl transferase, and correlation coefficients were higher in the varenicline group than in the placebo group for all markers.
Conclusions: Although the results of the main outcome of this study did not support an effect of varenicline in alcohol-dependent individuals, the secondary analyses of PEth, craving and AUDIT score support an effect of varenicline on alcohol consumption. The disclosure of a treatment effect and the lack of a clear placebo effect when using PEth as outcome variable, together with a nonsymmetric bias associated with self-reported data, strongly argue for using the specific biomarker PEth in studies of treatments of alcohol dependence.
Keywords: Alcohol Dependence; Alcohol Markers; Nicotinergic Acetylcholine Receptors; PEth; Phospha-tidylethanol; Varenicline.
Copyright © 2015 by the Research Society on Alcoholism.
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