Dietary Fatty Acids Directly Impact Central Nervous System Autoimmunity via the Small Intestine - PubMed (original) (raw)

Comparative Study

. 2015 Oct 20;43(4):817-29.

doi: 10.1016/j.immuni.2015.09.007.

Stefanie Jörg 2, Alexander Duscha 3, Johannes Berg 3, Arndt Manzel 2, Anne Waschbisch 2, Anna Hammer 2, De-Hyung Lee 2, Caroline May 4, Nicola Wilck 5, Andras Balogh 5, Annika I Ostermann 6, Nils Helge Schebb 7, Denis A Akkad 8, Diana A Grohme 9, Markus Kleinewietfeld 9, Stefan Kempa 10, Jan Thöne 3, Seray Demir 3, Dominik N Müller 5, Ralf Gold 3, Ralf A Linker 11

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Comparative Study

Dietary Fatty Acids Directly Impact Central Nervous System Autoimmunity via the Small Intestine

Aiden Haghikia et al. Immunity. 2015.

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Abstract

Growing empirical evidence suggests that nutrition and bacterial metabolites might impact the systemic immune response in the context of disease and autoimmunity. We report that long-chain fatty acids (LCFAs) enhanced differentiation and proliferation of T helper 1 (Th1) and/or Th17 cells and impaired their intestinal sequestration via p38-MAPK pathway. Alternatively, dietary short-chain FAs (SCFAs) expanded gut T regulatory (Treg) cells by suppression of the JNK1 and p38 pathway. We used experimental autoimmune encephalomyelitis (EAE) as a model of T cell-mediated autoimmunity to show that LCFAs consistently decreased SCFAs in the gut and exacerbated disease by expanding pathogenic Th1 and/or Th17 cell populations in the small intestine. Treatment with SCFAs ameliorated EAE and reduced axonal damage via long-lasting imprinting on lamina-propria-derived Treg cells. These data demonstrate a direct dietary impact on intestinal-specific, and subsequently central nervous system-specific, Th cell responses in autoimmunity, and thus might have therapeutic implications for autoimmune diseases such as multiple sclerosis.

Copyright © 2015 Elsevier Inc. All rights reserved.

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