The presence of persistent high-risk HPV genotypes in dysplastic cervical lesions is associated with progressive disease: natural history up to 36 months - PubMed (original) (raw)
. 1995 May 4;61(3):306-11.
doi: 10.1002/ijc.2910610305.
Affiliations
- PMID: 7729939
- DOI: 10.1002/ijc.2910610305
The presence of persistent high-risk HPV genotypes in dysplastic cervical lesions is associated with progressive disease: natural history up to 36 months
A J Remmink et al. Int J Cancer. 1995.
Abstract
To evaluate the clinical significance of HPV genotyping for the prediction of progressive cervical intraepithelial neoplasia (CIN) in women with cytomorphologically abnormal smears, a prospective, blind, non-intervention study was performed. A total of 342 patients screened with cytomorphologically abnormal cervical smears were monitored every 3-4 months by cervical cytology, colposcopy and HPV testing using PCR. Women with progressive CIN disease were defined as patients developing lesions with a colposcopic impression of CIN III over more than 2 quadrants or resulting in a cytological smear equivalent to Pap 5. These patients were subsequently treated according to standard procedures. If any doubt arose about the true status of the patients (n = 75) these patients were censored and biopsied. The mean follow-up time was 16.5 months (range 3-36 months). Nineteen women showed progressive CIN disease and all appeared to be continuously HPV-positive from the start of the study. At biopsy, all these patients were histologically classified as CIN III. Seventeen of these women were positive for high-risk HPV types. Two cases were classified as still unidentified HPV. No progression was seen in the absence of HPV DNA or in the presence of low-risk HPV types. In life-table analysis the cumulative rate of progressive, histologically verified CIN disease was 17% after 36 months. Further analyses showed that other risk factors such as age, sexarche, number of sexual partners or smoking hardly influenced the effect of HPV on progression. The results show that the continuous presence of high-risk HPV types in women with cytomorphologically abnormal smears is a strong marker for progressive CIN disease.
Similar articles
- Evaluation of human papillomavirus testing in primary screening for cervical abnormalities: comparison of sensitivity, specificity, and frequency of referral.
Kulasingam SL, Hughes JP, Kiviat NB, Mao C, Weiss NS, Kuypers JM, Koutsky LA. Kulasingam SL, et al. JAMA. 2002 Oct 9;288(14):1749-57. doi: 10.1001/jama.288.14.1749. JAMA. 2002. PMID: 12365959 - PCR-based high-risk HPV test in cervical cancer screening gives objective risk assessment of women with cytomorphologically normal cervical smears.
Rozendaal L, Walboomers JM, van der Linden JC, Voorhorst FJ, Kenemans P, Helmerhorst TJ, van Ballegooijen M, Meijer CJ. Rozendaal L, et al. Int J Cancer. 1996 Dec 11;68(6):766-9. doi: 10.1002/(SICI)1097-0215(19961211)68:6<766::AID-IJC13>3.0.CO;2-Z. Int J Cancer. 1996. PMID: 8980181 - Spontaneous evolution of intraepithelial lesions according to the grade and type of the implicated human papillomavirus (HPV).
Syrjänen KJ. Syrjänen KJ. Eur J Obstet Gynecol Reprod Biol. 1996 Mar;65(1):45-53. doi: 10.1016/0028-2243(95)02303-a. Eur J Obstet Gynecol Reprod Biol. 1996. PMID: 8706956 Review. - Tissue effects and host response. The key to the rational triage of cervical neoplasia.
Ferenczy A, Jenson AB. Ferenczy A, et al. Obstet Gynecol Clin North Am. 1996 Dec;23(4):759-82. doi: 10.1016/s0889-8545(05)70276-x. Obstet Gynecol Clin North Am. 1996. PMID: 8989775 Review.
Cited by
- Cutaneous Human Papillomaviruses and the Risk of Keratinocyte Carcinomas.
Rollison DE, Amorrortu RP, Zhao Y, Messina JL, Schell MJ, Fenske NA, Cherpelis BS, Giuliano AR, Sondak VK, Pawlita M, McKay-Chopin S, Gheit T, Waterboer T, Tommasino M. Rollison DE, et al. Cancer Res. 2021 Sep 1;81(17):4628-4638. doi: 10.1158/0008-5472.CAN-21-0805. Epub 2021 Jul 15. Cancer Res. 2021. PMID: 34266893 Free PMC article. - CD103+ tumor-infiltrating lymphocytes are tumor-reactive intraepithelial CD8+ T cells associated with prognostic benefit and therapy response in cervical cancer.
Komdeur FL, Prins TM, van de Wall S, Plat A, Wisman GBA, Hollema H, Daemen T, Church DN, de Bruyn M, Nijman HW. Komdeur FL, et al. Oncoimmunology. 2017 Jul 24;6(9):e1338230. doi: 10.1080/2162402X.2017.1338230. eCollection 2017. Oncoimmunology. 2017. PMID: 28932636 Free PMC article. - Interplay between viruses and bacterial microbiota in cancer development.
Vyshenska D, Lam KC, Shulzhenko N, Morgun A. Vyshenska D, et al. Semin Immunol. 2017 Aug;32:14-24. doi: 10.1016/j.smim.2017.05.003. Epub 2017 Jun 9. Semin Immunol. 2017. PMID: 28602713 Free PMC article. Review. - CUL2 overexpression driven by CUL2/E2F1/miR-424 regulatory loop promotes HPV16 E7 induced cervical carcinogenesis.
Xu J, Fang Y, Wang X, Wang F, Tian Q, Li Y, Xie X, Cheng X, Lu W. Xu J, et al. Oncotarget. 2016 May 24;7(21):31520-33. doi: 10.18632/oncotarget.9127. Oncotarget. 2016. PMID: 27153550 Free PMC article. - Variant-specific persistence of infections with human papillomavirus Types 31, 33, 45, 56 and 58 and risk of cervical intraepithelial neoplasia.
Xi LF, Schiffman M, Koutsky LA, Hughes JP, Hulbert A, Shen Z, Galloway DA, Kiviat NB. Xi LF, et al. Int J Cancer. 2016 Sep 1;139(5):1098-105. doi: 10.1002/ijc.30164. Epub 2016 May 14. Int J Cancer. 2016. PMID: 27121353 Free PMC article.
Publication types
MeSH terms
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical
Research Materials