Destabilization of tracts of simple repetitive DNA in yeast by mutations affecting DNA mismatch repair - PubMed (original) (raw)
. 1993 Sep 16;365(6443):274-6.
doi: 10.1038/365274a0.
Affiliations
- PMID: 8371783
- DOI: 10.1038/365274a0
Destabilization of tracts of simple repetitive DNA in yeast by mutations affecting DNA mismatch repair
M Strand et al. Nature. 1993.
Erratum in
- Nature 1994 Apr 7;368(6471);569
Abstract
The genomes of all eukaryotes contain tracts of DNA in which a single base or a small number of bases is repeated. Expansions of such tracts have been associated with several human disorders including the fragile X syndrome. In addition, simple repeats are unstable in certain forms of colorectal cancer, suggesting a defect in DNA replication or repair. We show here that mutations in any three yeast genes involved in DNA mismatch repair (PMS1, MLH1 and MSH2) lead to 100- to 700-fold increases in tract instability, whereas mutations that eliminate the proof-reading function of DNA polymerases have little effect. The meiotic stability of the tracts is similar to the mitotic stability. These results suggest that tract instability is associated with DNA polymerases slipping during replication, and that some types of colorectal cancer may reflect mutations in genes involved in DNA mismatch repair.
Comment in
- Nucleotide repeats. Slippery DNA and diseases.
Kunkel TA. Kunkel TA. Nature. 1993 Sep 16;365(6443):207-8. doi: 10.1038/365207a0. Nature. 1993. PMID: 8371775 No abstract available.
Similar articles
- Meiotic instability of human minisatellite CEB1 in yeast requires DNA double-strand breaks.
Debrauwère H, Buard J, Tessier J, Aubert D, Vergnaud G, Nicolas A. Debrauwère H, et al. Nat Genet. 1999 Nov;23(3):367-71. doi: 10.1038/15557. Nat Genet. 1999. PMID: 10545956 - Editing DNA replication and recombination by mismatch repair: from bacterial genetics to mechanisms of predisposition to cancer in humans.
Radman M, Matic I, Halliday JA, Taddei F. Radman M, et al. Philos Trans R Soc Lond B Biol Sci. 1995 Jan 30;347(1319):97-103. doi: 10.1098/rstb.1995.0015. Philos Trans R Soc Lond B Biol Sci. 1995. PMID: 7746861 Review. - Evidence from mutational specificity studies that yeast DNA polymerases delta and epsilon replicate different DNA strands at an intracellular replication fork.
Karthikeyan R, Vonarx EJ, Straffon AF, Simon M, Faye G, Kunz BA. Karthikeyan R, et al. J Mol Biol. 2000 Jun 2;299(2):405-19. doi: 10.1006/jmbi.2000.3744. J Mol Biol. 2000. PMID: 10860748 - Selection for genome instability by DNA damage in human cells: unstable microsatellites and their consequences for tumourigenesis.
Hampson R. Hampson R. Radiat Oncol Investig. 1997;5(3):111-4. doi: 10.1002/(SICI)1520-6823(1997)5:3<111::AID-ROI5>3.0.CO;2-0. Radiat Oncol Investig. 1997. PMID: 9303066 Review. - Microsatellite instability in yeast: dependence on repeat unit size and DNA mismatch repair genes.
Sia EA, Kokoska RJ, Dominska M, Greenwell P, Petes TD. Sia EA, et al. Mol Cell Biol. 1997 May;17(5):2851-8. doi: 10.1128/MCB.17.5.2851. Mol Cell Biol. 1997. PMID: 9111357 Free PMC article.
Cited by
- MutLα suppresses error-prone DNA mismatch repair and preferentially protects noncoding DNA from mutations.
Kadyrova LY, Mieczkowski PA, Kadyrov FA. Kadyrova LY, et al. J Biol Chem. 2024 Jun;300(6):107406. doi: 10.1016/j.jbc.2024.107406. Epub 2024 May 21. J Biol Chem. 2024. PMID: 38782208 Free PMC article. - MutLα suppresses error-prone DNA mismatch repair and preferentially protects noncoding DNA from mutations.
Kadyrova LY, Mieczkowski PA, Kadyrov FA. Kadyrova LY, et al. bioRxiv [Preprint]. 2024 Apr 2:2024.04.01.587563. doi: 10.1101/2024.04.01.587563. bioRxiv. 2024. PMID: 38617288 Free PMC article. Updated. Preprint. - Efficient prime editing in two-cell mouse embryos using PEmbryo.
Kim-Yip RP, McNulty R, Joyce B, Mollica A, Chen PJ, Ravisankar P, Law BK, Liu DR, Toettcher JE, Ivakine EA, Posfai E, Adamson B. Kim-Yip RP, et al. Nat Biotechnol. 2024 Feb 6. doi: 10.1038/s41587-023-02106-x. Online ahead of print. Nat Biotechnol. 2024. PMID: 38321114 - LUSTR: a new customizable tool for calling genome-wide germline and somatic short tandem repeat variants.
Lu J, Toro C, Adams DR; Undiagnosed Diseases Network; Moreno CAM, Lee WP, Leung YY, Harms MB, Vardarajan B, Heinzen EL. Lu J, et al. BMC Genomics. 2024 Jan 26;25(1):115. doi: 10.1186/s12864-023-09935-9. BMC Genomics. 2024. PMID: 38279154 Free PMC article. - The sequence of the repetitive motif influences the frequency of multistep mutations in Short Tandem Repeats.
Antão-Sousa S, Pinto N, Rende P, Amorim A, Gusmão L. Antão-Sousa S, et al. Sci Rep. 2023 Jun 24;13(1):10251. doi: 10.1038/s41598-023-32137-y. Sci Rep. 2023. PMID: 37355683 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Molecular Biology Databases
Research Materials