Cyclin D1 is a nuclear protein required for cell cycle progression in G1 - PubMed (original) (raw)
Cyclin D1 is a nuclear protein required for cell cycle progression in G1
V Baldin et al. Genes Dev. 1993 May.
Free article
Abstract
A cascade of events is triggered upon the addition of growth factor to quiescent mammalian cells, which ultimately restarts proliferation by inducing the transition from G0/G1 to S-phase. We have studied cyclin D1, a putative G1 cyclin, in normal diploid human fibroblasts. Cyclin D1 accumulated and reached a maximum level before S-phase upon the addition of serum to quiescent cells. The protein was localized to the nucleus, and it disappeared from the nucleus as cells proceeded into S-phase. Microinjection of anti-cyclin D1 antibodies or antisense plasmid prevented cells from entering S-phase, and the kinetics of inhibition showed that cyclin D1 is required at a point in the cell cycle earlier than cyclin A. These results demonstrate that cyclin D1 is a critical target of proliferative signals in G1.
Similar articles
- Human cyclin E, a nuclear protein essential for the G1-to-S phase transition.
Ohtsubo M, Theodoras AM, Schumacher J, Roberts JM, Pagano M. Ohtsubo M, et al. Mol Cell Biol. 1995 May;15(5):2612-24. doi: 10.1128/MCB.15.5.2612. Mol Cell Biol. 1995. PMID: 7739542 Free PMC article. - Different roles for cyclins D1 and E in regulation of the G1-to-S transition.
Resnitzky D, Reed SI. Resnitzky D, et al. Mol Cell Biol. 1995 Jul;15(7):3463-9. doi: 10.1128/MCB.15.7.3463. Mol Cell Biol. 1995. PMID: 7791752 Free PMC article. - Cyclic AMP negatively controls c-myc transcription and G1 cell cycle progression in p210 BCR-ABL transformed cells: inhibitory activity exerted through cyclin D1 and cdk4.
Williamson EA, Burgess GS, Eder P, Litz-Jackson S, Boswell HS. Williamson EA, et al. Leukemia. 1997 Jan;11(1):73-85. doi: 10.1038/sj.leu.2400551. Leukemia. 1997. PMID: 9001421 - Regulation of cell cycle entry and G1 progression by CSF-1.
Roussel MF. Roussel MF. Mol Reprod Dev. 1997 Jan;46(1):11-8. doi: 10.1002/(SICI)1098-2795(199701)46:1<11::AID-MRD3>3.0.CO;2-U. Mol Reprod Dev. 1997. PMID: 8981358 Review. - Regulation of the G1/S transition phase in mesangial cells by E2F1.
Inoshita S, Terada Y, Nakashima O, Kuwahara M, Sasaki S, Marumo F. Inoshita S, et al. Kidney Int. 1999 Oct;56(4):1238-41. doi: 10.1046/j.1523-1755.1999.00705.x. Kidney Int. 1999. PMID: 10504464 Review.
Cited by
- Non-coding RNAs as potential targets in metformin therapy for cancer.
Zhang Y, Wu Y, Liu Z, Yang K, Lin H, Xiong K. Zhang Y, et al. Cancer Cell Int. 2024 Oct 1;24(1):333. doi: 10.1186/s12935-024-03516-w. Cancer Cell Int. 2024. PMID: 39354464 Free PMC article. Review. - Apolipoprotein E is a novel marker for chondrocytes in the growth plate resting zone.
Otsuru S, Kodama J, Oichi T, Wilkinson K, Abzug J, Kaito T, Iwamoto-Enomoto M, Iwamoto M. Otsuru S, et al. Res Sq [Preprint]. 2024 Aug 5:rs.3.rs-4656728. doi: 10.21203/rs.3.rs-4656728/v1. Res Sq. 2024. PMID: 39149484 Free PMC article. Preprint. - In vitro study on anti-proliferative and anti-cancer activity of picrosides in triple-negative breast cancer.
Soni D, Wahi D, Verma S. Soni D, et al. Med Oncol. 2024 May 8;41(6):143. doi: 10.1007/s12032-024-02397-3. Med Oncol. 2024. PMID: 38717628 - The MYC-NFATC2 axis maintains the cell cycle and mitochondrial function in acute myeloid leukaemia cells.
Patterson SD, Massett ME, Huang X, Jørgensen HG, Michie AM. Patterson SD, et al. Mol Oncol. 2024 Sep;18(9):2234-2254. doi: 10.1002/1878-0261.13630. Epub 2024 Mar 8. Mol Oncol. 2024. PMID: 38459421 Free PMC article. - CDK-independent role of D-type cyclins in regulating DNA mismatch repair.
Rona G, Miwatani-Minter B, Zhang Q, Goldberg HV, Kerzhnerman MA, Howard JB, Simoneschi D, Lane E, Hobbs JW, Sassani E, Wang AA, Keegan S, Laverty DJ, Piett CG, Pongor LS, Xu ML, Andrade J, Thomas A, Sicinski P, Askenazi M, Ueberheide B, Fenyö D, Nagel ZD, Pagano M. Rona G, et al. Mol Cell. 2024 Apr 4;84(7):1224-1242.e13. doi: 10.1016/j.molcel.2024.02.010. Epub 2024 Mar 7. Mol Cell. 2024. PMID: 38458201
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Research Materials