Identification of IL-5 and RANTES as the major eosinophil chemoattractants in the asthmatic lung - PubMed (original) (raw)
Identification of IL-5 and RANTES as the major eosinophil chemoattractants in the asthmatic lung
J Venge et al. J Allergy Clin Immunol. 1996 May.
Abstract
The study was carried out to identify those molecules that are important in vivo in the attraction of eosinophil granulocytes to the lungs of patients with asthma. Asthmatic patients with birch pollen allergy had lavages performed before and during the pollen season, and the chemotactic activity of the bronchoalveolar lavage fluid was tested against normal eosinophils. The activity was significantly increased during the pollen season as compared with the activity before the pollen season (p less than 0.01). Neutralizing antibodies to IL-2, IL-5 and IL-8, leukemia inhibitory factor, and to RANTES were added to the bronchoalveolar lavage fluid. Antibodies to IL-5 and RANTES, but not to IL-2 and IL-8 or leukemia inhibitory factor, significantly inhibited the chemotactic activity for eosinophils (p less than 0.001). It is concluded that IL-5 and RANTES are important chemoattractants in the lungs of patients with allergic asthma. The effect of IL-5 may be that of a cofactor to the chemotactic molecules, of which RANTES may be one of the most important in allergic asthma.
Similar articles
- The role of interleukin-5, interleukin-8 and RANTES in the chemotactic attraction of eosinophils to the allergic lung.
Lampinen M, Rak S, Venge P. Lampinen M, et al. Clin Exp Allergy. 1999 Mar;29(3):314-22. doi: 10.1046/j.1365-2222.1999.00390.x. Clin Exp Allergy. 1999. PMID: 10202337 - The effect of immunotherapy on eosinophil accumulation and production of eosinophil chemotactic activity in the lung of subjects with asthma during natural pollen exposure.
Rak S, Björnson A, Håkanson L, Sörenson S, Venge P. Rak S, et al. J Allergy Clin Immunol. 1991 Dec;88(6):878-88. doi: 10.1016/0091-6749(91)90244-i. J Allergy Clin Immunol. 1991. PMID: 1744358 Clinical Trial. - CC chemokines and interleukin-5 in bronchial lavage fluid from patients with status asthmaticus. Potential implication in eosinophil recruitment.
Tillie-Leblond I, Hammad H, Desurmont S, Pugin J, Wallaert B, Tonnel AB, Gosset P. Tillie-Leblond I, et al. Am J Respir Crit Care Med. 2000 Aug;162(2 Pt 1):586-92. doi: 10.1164/ajrccm.162.2.9907014. Am J Respir Crit Care Med. 2000. PMID: 10934091 - Mechanisms regulating eosinophil extravasation in asthma.
Knol EF, Roos D. Knol EF, et al. Eur Respir J Suppl. 1996 Aug;22:136s-140s. Eur Respir J Suppl. 1996. PMID: 8871059 Review. - Distinct spatial requirement for eosinophil-induced airways hyperreactivity.
Webb DC, McKenzie AN, Matthaei KI, Rothenberg ME, Foster PS. Webb DC, et al. Immunol Cell Biol. 2001 Apr;79(2):165-9. doi: 10.1046/j.1440-1711.2001.00989.x. Immunol Cell Biol. 2001. PMID: 11264712 Review.
Cited by
- Functional genomics of human bronchial epithelial cells directly interacting with conidia of Aspergillus fumigatus.
Gomez P, Hackett TL, Moore MM, Knight DA, Tebbutt SJ. Gomez P, et al. BMC Genomics. 2010 Jun 4;11:358. doi: 10.1186/1471-2164-11-358. BMC Genomics. 2010. PMID: 20525375 Free PMC article. - Asthma during pregnancy alters immune cell profile and airway epithelial chemokine release.
Osei-Kumah A, Wark PA, Smith R, Clifton VL. Osei-Kumah A, et al. Inflamm Res. 2010 May;59(5):349-58. doi: 10.1007/s00011-009-0102-y. Epub 2009 Oct 30. Inflamm Res. 2010. PMID: 19876718 - Chemokines in acute respiratory distress syndrome.
Puneet P, Moochhala S, Bhatia M. Puneet P, et al. Am J Physiol Lung Cell Mol Physiol. 2005 Jan;288(1):L3-15. doi: 10.1152/ajplung.00405.2003. Am J Physiol Lung Cell Mol Physiol. 2005. PMID: 15591040 Free PMC article. Review. - Role of Cytokines in EGPA and the Possibility of Treatment with an Anti-IL-5 Antibody.
Isozaki T, Homma T, Sagara H, Kasama T. Isozaki T, et al. J Clin Med. 2020 Nov 30;9(12):3890. doi: 10.3390/jcm9123890. J Clin Med. 2020. PMID: 33265990 Free PMC article. Review. - PI3Kδ inhibition prevents IL33, ILC2s and inflammatory eosinophils in persistent airway inflammation.
Uddin S, Amour A, Lewis DJ, Edwards CD, Williamson MG, Hall S, Lione LA, Hessel EM. Uddin S, et al. BMC Immunol. 2021 Dec 17;22(1):78. doi: 10.1186/s12865-021-00461-5. BMC Immunol. 2021. PMID: 34920698 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical