Topological characterization of the essential Escherichia coli cell division protein FtsN - PubMed (original) (raw)
Topological characterization of the essential Escherichia coli cell division protein FtsN
K Dai et al. J Bacteriol. 1996 Mar.
Abstract
Genetic and biochemical approaches were used to analyze a topological model for FtsN, a 36-kDa protein with a putative transmembrane segment near the N terminus, and to ascertain the requirements of the putative cytoplasmic and membrane-spanning domains for the function of this protein. Analysis of FtsN-PhoA fusions revealed that the putative transmembrane segment of FtsN could act as a translocation signal. Protease accessibility studies of FtsN in spheroblasts and inverted membrane vesicles confirmed that FtsN had a simple bitopic topology with a short cytoplasmic amino terminus, a single membrane-spanning domain, and a large periplasmic carboxy terminus. To ascertain the functional requirements of the N-terminal segments of FtsN, various constructs were made. Deletion of the N-terminal cytoplasmic and membrane-spanning domains led to intracellular localization of the carboxy domain, instability,and loss of function. Replacement of the N-terminal cytoplasmic and membrane-spanning domains with a membrane-spanning domain from MalG restored subcellular localization and function. These N-terminal domains of FtsN could also be replaced by the cleavable MalE signal sequence with restoration of subcellular localization and function. It is concluded that the N-terminal, cytoplasmic, and transmembrane domains of FtsN are not required for function of the carboxy domain other than to transport it to the periplasm. FtsQ and FtsI were also analyzed.
Similar articles
- Domain-swapping analysis of FtsI, FtsL, and FtsQ, bitopic membrane proteins essential for cell division in Escherichia coli.
Guzman LM, Weiss DS, Beckwith J. Guzman LM, et al. J Bacteriol. 1997 Aug;179(16):5094-103. doi: 10.1128/jb.179.16.5094-5103.1997. J Bacteriol. 1997. PMID: 9260951 Free PMC article. - Cell division in Escherichia coli: role of FtsL domains in septal localization, function, and oligomerization.
Ghigo JM, Beckwith J. Ghigo JM, et al. J Bacteriol. 2000 Jan;182(1):116-29. doi: 10.1128/JB.182.1.116-129.2000. J Bacteriol. 2000. PMID: 10613870 Free PMC article. - FtsN, a late recruit to the septum in Escherichia coli.
Addinall SG, Cao C, Lutkenhaus J. Addinall SG, et al. Mol Microbiol. 1997 Jul;25(2):303-9. doi: 10.1046/j.1365-2958.1997.4641833.x. Mol Microbiol. 1997. PMID: 9282742 - Tinkering with transporters: periplasmic binding protein-dependent maltose transport in E. coli.
Shuman HA, Panagiotidis CH. Shuman HA, et al. J Bioenerg Biomembr. 1993 Dec;25(6):613-20. doi: 10.1007/BF00770248. J Bioenerg Biomembr. 1993. PMID: 7511584 Review. - Membrane protein spanning segments as export signals.
Calamia J, Manoil C. Calamia J, et al. J Mol Biol. 1992 Apr 5;224(3):539-43. doi: 10.1016/0022-2836(92)90542-r. J Mol Biol. 1992. PMID: 1569545 Review.
Cited by
- Overexpression of the Escherichia coli TolQ protein leads to a null-FtsN-like division phenotype.
Teleha MA, Miller AC, Larsen RA. Teleha MA, et al. Microbiologyopen. 2013 Aug;2(4):618-32. doi: 10.1002/mbo3.101. Epub 2013 Jul 2. Microbiologyopen. 2013. PMID: 23818486 Free PMC article. - An altered FtsA can compensate for the loss of essential cell division protein FtsN in Escherichia coli.
Bernard CS, Sadasivam M, Shiomi D, Margolin W. Bernard CS, et al. Mol Microbiol. 2007 Jun;64(5):1289-305. doi: 10.1111/j.1365-2958.2007.05738.x. Mol Microbiol. 2007. PMID: 17542921 Free PMC article. - ATP-binding site lesions in FtsE impair cell division.
Arends SJ, Kustusch RJ, Weiss DS. Arends SJ, et al. J Bacteriol. 2009 Jun;191(12):3772-84. doi: 10.1128/JB.00179-09. Epub 2009 Apr 17. J Bacteriol. 2009. PMID: 19376877 Free PMC article. - FtsI and FtsW are localized to the septum in Escherichia coli.
Wang L, Khattar MK, Donachie WD, Lutkenhaus J. Wang L, et al. J Bacteriol. 1998 Jun;180(11):2810-6. doi: 10.1128/JB.180.11.2810-2816.1998. J Bacteriol. 1998. PMID: 9603865 Free PMC article. - The early divisome protein FtsA interacts directly through its 1c subdomain with the cytoplasmic domain of the late divisome protein FtsN.
Busiek KK, Eraso JM, Wang Y, Margolin W. Busiek KK, et al. J Bacteriol. 2012 Apr;194(8):1989-2000. doi: 10.1128/JB.06683-11. Epub 2012 Feb 10. J Bacteriol. 2012. PMID: 22328664 Free PMC article.
References
- Nature. 1992 Sep 17;359(6392):251-4 - PubMed
- Nature. 1991 Nov 14;354(6349):161-4 - PubMed
- J Bacteriol. 1992 Oct;174(19):6145-51 - PubMed
- J Bacteriol. 1992 Oct;174(19):6314-6 - PubMed
- J Bacteriol. 1992 Dec;174(23):7716-28 - PubMed
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Molecular Biology Databases