The mean A beta load in the hippocampus correlates with duration and severity of dementia in subgroups of Alzheimer disease - PubMed (original) (raw)
The mean A beta load in the hippocampus correlates with duration and severity of dementia in subgroups of Alzheimer disease
G T Bartoo et al. J Neuropathol Exp Neurol. 1997 May.
Abstract
Using image analysis techniques to quantify the percentage area covered by the immunopositive marker for amyloid beta-peptide (A beta), we examined subjects with combinations of either early-onset or late-onset Alzheimer disease (AD) and either familial Alzheimer disease (FAD) or sporadic Alzheimer disease (SAD). We measured the mean and maximum A beta loads, in the hippocampus of each subject. There were no statistically significant differences in the mean A beta load between familial and sporadic AD subjects. Although sample sizes were too small for statistical testing, subjects with the epsilon 4/epsilon 4 allele of the apolipoprotein E (ApoE) gene had higher mean A beta loads than those with the epsilon 3/epsilon 3 or epsilon 3/epsilon 4 alleles. Members of the Volga German families (recently linked to chromosome 1) all had high mean A beta loads, and one of the chromosome 14-linked subjects had the highest mean A beta load while the other had a relatively small load, but the sample was too small for statistical comparisons. The duration of dementia and neuropsychological test scores showed a statistically significant correlation with the mean A beta load in the hippocampus, but not with the maximum A beta load. This difference indicates that the mean A beta load may be a more useful feature than the maximum A beta load as an objective neuropathological measure for cognitive status. This finding may help to improve the established methods for quantitative assessment of the neuropathological changes in AD.
Similar articles
- The apolipoprotein E/CI/CII gene cluster and late-onset Alzheimer disease.
Yu CE, Payami H, Olson JM, Boehnke M, Wijsman EM, Orr HT, Kukull WA, Goddard KA, Nemens E, White JA, et al. Yu CE, et al. Am J Hum Genet. 1994 Apr;54(4):631-42. Am J Hum Genet. 1994. PMID: 8128960 Free PMC article. - Clinical and pathological correlates of apolipoprotein E epsilon 4 in Alzheimer's disease.
Gomez-Isla T, West HL, Rebeck GW, Harr SD, Growdon JH, Locascio JJ, Perls TT, Lipsitz LA, Hyman BT. Gomez-Isla T, et al. Ann Neurol. 1996 Jan;39(1):62-70. doi: 10.1002/ana.410390110. Ann Neurol. 1996. PMID: 8572669 - Genetic associations between cathepsin D exon 2 C-->T polymorphism and Alzheimer's disease, and pathological correlations with genotype.
Davidson Y, Gibbons L, Pritchard A, Hardicre J, Wren J, Tian J, Shi J, Stopford C, Julien C, Thompson J, Payton A, Thaker U, Hayes AJ, Iwatsubo T, Pickering-Brown SM, Pendleton N, Horan MA, Burns A, Purandare N, Lendon CL, Neary D, Snowden JS, Mann DM. Davidson Y, et al. J Neurol Neurosurg Psychiatry. 2006 Apr;77(4):515-7. doi: 10.1136/jnnp.2005.063917. J Neurol Neurosurg Psychiatry. 2006. PMID: 16543533 Free PMC article. - [Apolipoprotein E4 and late-onset Alzheimer's disease].
Tamaoka A. Tamaoka A. Nihon Rinsho. 1994 Dec;52(12):3257-65. Nihon Rinsho. 1994. PMID: 7853720 Review. Japanese. - [Genetics of late-onset Alzheimer's disease: vascular risk and beta-amyloid metabolism].
Panza F, Solfrizzi V, D'Introno A, Capurso C, Colacicco AM, Torres F, Altomare E, Capurso A. Panza F, et al. Recenti Prog Med. 2002 Sep;93(9):489-97. Recenti Prog Med. 2002. PMID: 12355988 Review. Italian.
Cited by
- Quantitative neuropathology: an update on automated methodologies and implications for large scale cohorts.
Walker L, McAleese KE, Johnson M, Khundakar AA, Erskine D, Thomas AJ, McKeith IG, Attems J. Walker L, et al. J Neural Transm (Vienna). 2017 Jun;124(6):671-683. doi: 10.1007/s00702-017-1702-2. Epub 2017 Mar 6. J Neural Transm (Vienna). 2017. PMID: 28265813 Free PMC article. - Impairment of context memory by beta-amyloid peptide in terrestrial snail.
Korshunova TA, Bravarenko NI, Balaban PM. Korshunova TA, et al. Front Behav Neurosci. 2008 Sep 2;2:3. doi: 10.3389/neuro.08/003.2008. eCollection 2008. Front Behav Neurosci. 2008. PMID: 18958193 Free PMC article. - The role of beta-amyloid protein in synaptic function: implications for Alzheimer's disease therapy.
Peña F, Gutiérrez-Lerma A, Quiroz-Baez R, Arias C. Peña F, et al. Curr Neuropharmacol. 2006 Apr;4(2):149-63. doi: 10.2174/157015906776359531. Curr Neuropharmacol. 2006. PMID: 18615129 Free PMC article. - Effects of beta-amyloid (25-35) on learning in the common snail.
Korshunova TA, Samarova EI, Bravarenko NI, Balaban PM. Korshunova TA, et al. Neurosci Behav Physiol. 2008 May;38(4):377-82. doi: 10.1007/s11055-008-0054-7. Neurosci Behav Physiol. 2008. PMID: 18401730 - Studies of the effects of fragment (25-35) of beta-amyloid peptide on the behavior of rats in a radial maze.
Stepanichev MY, Moiseeva YV, Lazareva NA, Gulyaeva NV. Stepanichev MY, et al. Neurosci Behav Physiol. 2005 Jun;35(5):511-8. doi: 10.1007/s11055-005-0086-1. Neurosci Behav Physiol. 2005. PMID: 16033199
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Medical
Miscellaneous