Temporal changes in gene expression following cryogenic rat brain injury - PubMed (original) (raw)
Temporal changes in gene expression following cryogenic rat brain injury
J L Cook et al. Brain Res Mol Brain Res. 1998.
Abstract
Expression of 18 genes was examined at 8 different time points between 1 h and 28 days following cryogenic rat brain injury. The genes include thymidine kinase (TK), p53 tumor suppressor, c-fos, renin, myelin basic protein (MBP), proteolipid protein (PLP), transferrin, transferrin receptor, platelet-derived growth factor A (PDGF A), platelet-derived growth factor B (PDGF B), platelet-derived growth factor receptor alpha (PDGF alpha receptor), platelet-derived growth factor receptor beta (PDGF beta receptor), glial fibrillary acidic protein (GFAP), transforming growth factor-beta 1 (TGF-beta 1), basic fibroblast growth factor (bFGF), fibroblast growth factor receptor-1 (FGF-R1), insulin-like growth factor-1 (IGF-1), and somatostatin. Time courses of gene expression were determined for RNAs derived from hippocampus and cortex. Genes were divided into categories based upon those in which statistically significant changes in expression were first observed at or before 24 h (early genes) and those in which changes were first observed at or after 72 h (late genes). In the present model, many genes demonstrate elevated RNA levels in the cortex prior to hippocampus, following injury. RNAs transcribed from late genes tend to be elevated concurrently in cortex and hippocampus.
Similar articles
- Simultaneous analysis of multiple gene expression patterns as a function of development, injury or senescence.
Cook JL, Marcheselli V, Alam J, Deininger PL, Bazan NG. Cook JL, et al. Brain Res Brain Res Protoc. 1998 Sep;3(1):1-6. doi: 10.1016/s1385-299x(98)00012-9. Brain Res Brain Res Protoc. 1998. PMID: 9767074 - Cryogenic spinal cord injury induces astrocytic gene expression of insulin-like growth factor I and insulin-like growth factor binding protein 2 during myelin regeneration.
Yao DL, West NR, Bondy CA, Brenner M, Hudson LD, Zhou J, Collins GH, Webster HD. Yao DL, et al. J Neurosci Res. 1995 Apr 1;40(5):647-59. doi: 10.1002/jnr.490400510. J Neurosci Res. 1995. PMID: 7541476 - Sequential changes in glial fibrillary acidic protein and gene expression following parasagittal fluid-percussion brain injury in rats.
Dietrich WD, Truettner J, Zhao W, Alonso OF, Busto R, Ginsberg MD. Dietrich WD, et al. J Neurotrauma. 1999 Jul;16(7):567-81. doi: 10.1089/neu.1999.16.567. J Neurotrauma. 1999. PMID: 10447069 - Vimentin and GFAP responses in astrocytes after contusion trauma to the murine brain.
Ekmark-Lewén S, Lewén A, Israelsson C, Li GL, Farooque M, Olsson Y, Ebendal T, Hillered L. Ekmark-Lewén S, et al. Restor Neurol Neurosci. 2010;28(3):311-21. doi: 10.3233/RNN-2010-0529. Restor Neurol Neurosci. 2010. PMID: 20479526 - Dysregulated expression of growth factors and their receptors in the development of prostate cancer.
Djakiew D. Djakiew D. Prostate. 2000 Feb 1;42(2):150-60. doi: 10.1002/(sici)1097-0045(20000201)42:2<150::aid-pros10>3.0.co;2-h. Prostate. 2000. PMID: 10617873 Review. No abstract available.
Cited by
- Endogenous repair signaling after brain injury and complementary bioengineering approaches to enhance neural regeneration.
Addington CP, Roussas A, Dutta D, Stabenfeldt SE. Addington CP, et al. Biomark Insights. 2015 May 12;10(Suppl 1):43-60. doi: 10.4137/BMI.S20062. eCollection 2015. Biomark Insights. 2015. PMID: 25983552 Free PMC article. Review. - Blood-brain barrier pathophysiology in traumatic brain injury.
Chodobski A, Zink BJ, Szmydynger-Chodobska J. Chodobski A, et al. Transl Stroke Res. 2011 Dec;2(4):492-516. doi: 10.1007/s12975-011-0125-x. Transl Stroke Res. 2011. PMID: 22299022 Free PMC article. - Role and Importance of IGF-1 in Traumatic Brain Injuries.
Mangiola A, Vigo V, Anile C, De Bonis P, Marziali G, Lofrese G. Mangiola A, et al. Biomed Res Int. 2015;2015:736104. doi: 10.1155/2015/736104. Epub 2015 Aug 31. Biomed Res Int. 2015. PMID: 26417600 Free PMC article. Review.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Research Materials
Miscellaneous