Endothelial cells overexpressing IL-8 receptor reduce cardiac remodeling and dysfunction following myocardial infarction (original) (raw)
Inhibition Of Interleukin-8 Blocks Myocardial Ischemia-Reperfusion Injury
Timothy Pohlman
The Journal of Thoracic and Cardiovascular Surgery, 1998
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Interleukin8 gene induction in the myocardium after ischemia and reperfusion in vivo
Leonardo Mendoza
Journal of Clinical Investigation, 1995
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Endothelial Cells Overexpressing Interleukin-8 Receptors Reduce Inflammatory and Neointimal Responses to Arterial Injury
Ziheng Yang
Circulation, 2012
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Targeted Delivery of Human iPS-ECs Overexpressing IL-8 Receptors Inhibits Neointimal and Inflammatory Responses to Vascular Injury in the Rat
Ngan Huang
American Journal of Physiology - Heart and Circulatory Physiology, 2016
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Interleukin-8 is not involved in the increased chemotactic activity of peripheral blood plasma during acute myocardial infarction
Tomasz Siminiak
Basic research in cardiology
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Interleukin 8 released after acute myocardial infarction is mainly bound to erythrocytes
Rob Adams
Journal of Pharmacology and Experimental Therapeutics, 1997
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Potential advantages of cell administration on the inflammatory response compared to standard ACE inhibitor treatment in experimental myocardial infarction
Michele M Ciulla
Journal of Translational Medicine, 2008
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Anti-Inflammatory Strategies for Ventricular Remodeling Following ST-Segment Elevation Acute Myocardial Infarction
Benjamin Van Tassell
Journal of the American College of Cardiology, 2014
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Is inflammation good for the ischemic heart – perspectives beyond the ordinary
Karla Kurrelmeyer
Zeitschrift f�r Kardiologie, 2000
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Neutrophils Sustain Pathogenic CD8+ T Cell Responses in the Heart
Jason Westrich
The American Journal of Pathology, 2003
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Effects of Endotoxin on Neutrophil-Mediated Ischemia/Reperfusion Injury in the Rat Heart In Vivo
Brian Lipton
Experimental Biology and Medicine, 2001
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Toll-like receptor 4 mediates ischemia/reperfusion injury of the heart
Timothy Pohlman
The Journal of Thoracic and Cardiovascular Surgery, 2004
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Potential benefits of cell therapy in coronary heart disease
Teresa Infante
Journal of Cardiology, 2013
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The role of neutrophils in myocardial ischemia–reperfusion injury
J. Vinten-johansen
Cardiovascular Research, 1999
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Identification of Noncytotoxic and IL-10-Producing CD8+AT2R+ T Cell Population in Response to Ischemic Heart Injury
Hans Imboden
The Journal of Immunology, 2010
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Effects of endotoxin on neutrophil-mediated I/R injury in isolated perfused rat hearts
Brian Lipton
American Journal of Physiology-Heart and Circulatory Physiology, 2001
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Cardiac-Selective Expression of Extracellular Superoxide Dismutase After Systemic Injection of Adeno-Associated Virus 9 Protects the Heart Against Post-Myocardial Infarction Left Ventricular Remodeling
Prasad Konkalmatt
Circulation: Cardiovascular Imaging, 2013
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Induced Pluripotent Stem Cell-Derived Endothelial Cells Overexpressing Interleukin-8 Receptors A/B and/or C-C Chemokine Receptors 2/5 Inhibit Vascular Injury Response
Tim Townes
Stem Cells Translational Medicine, 2017
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Pathophysiological role of neutrophils in acute myocardial infarction
Nicolas Vuilleumier
Thrombosis and Haemostasis, 2013
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Gene Therapy With Inducible Nitric Oxide Synthase Protects Against Myocardial Infarction via a Cyclooxygenase-2-Dependent Mechanism
Yu-Ting Xuan
Circulation Research, 2003
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Interleukin-8 as a sensitive marker of unstable coronary artery disease
Tsugiyasu Kanda
The American Journal of Cardiology, 1996
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Adeno-associated virus serotype 9 administered systemically after reperfusion preferentially targets cardiomyocytes in the infarct border zone with pharmacodynamics suitable for the attenuation of left ventricular remodeling
Prasad Konkalmatt
The Journal of Gene Medicine, 2012
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Mice Lacking Inducible Nitric Oxide Synthase Have Improved Left Ventricular Contractile Function and Reduced Apoptotic Cell Death Late After Myocardial Infarction
Krishna Singh
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UltraRapid Communication IL10 Inhibits Inflammation and Attenuates Left Ventricular Remodeling After Myocardial Infarction via Activation of STAT3 and Suppression of HuR
Raj Kishore
2010
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