Endothelial cells overexpressing IL-8 receptor reduce cardiac remodeling and dysfunction following myocardial infarction (original) (raw)

Inhibition Of Interleukin-8 Blocks Myocardial Ischemia-Reperfusion Injury

Timothy Pohlman

The Journal of Thoracic and Cardiovascular Surgery, 1998

View PDFchevron_right

Interleukin8 gene induction in the myocardium after ischemia and reperfusion in vivo

Leonardo Mendoza

Journal of Clinical Investigation, 1995

View PDFchevron_right

Endothelial Cells Overexpressing Interleukin-8 Receptors Reduce Inflammatory and Neointimal Responses to Arterial Injury

Ziheng Yang

Circulation, 2012

View PDFchevron_right

Targeted Delivery of Human iPS-ECs Overexpressing IL-8 Receptors Inhibits Neointimal and Inflammatory Responses to Vascular Injury in the Rat

Ngan Huang

American Journal of Physiology - Heart and Circulatory Physiology, 2016

View PDFchevron_right

Interleukin-8 is not involved in the increased chemotactic activity of peripheral blood plasma during acute myocardial infarction

Tomasz Siminiak

Basic research in cardiology

View PDFchevron_right

Interleukin 8 released after acute myocardial infarction is mainly bound to erythrocytes

Rob Adams

Journal of Pharmacology and Experimental Therapeutics, 1997

View PDFchevron_right

Potential advantages of cell administration on the inflammatory response compared to standard ACE inhibitor treatment in experimental myocardial infarction

Michele M Ciulla

Journal of Translational Medicine, 2008

View PDFchevron_right

Anti-Inflammatory Strategies for Ventricular Remodeling Following ST-Segment Elevation Acute Myocardial Infarction

Benjamin Van Tassell

Journal of the American College of Cardiology, 2014

View PDFchevron_right

Is inflammation good for the ischemic heart – perspectives beyond the ordinary

Karla Kurrelmeyer

Zeitschrift f�r Kardiologie, 2000

View PDFchevron_right

Neutrophils Sustain Pathogenic CD8+ T Cell Responses in the Heart

Jason Westrich

The American Journal of Pathology, 2003

View PDFchevron_right

Effects of Endotoxin on Neutrophil-Mediated Ischemia/Reperfusion Injury in the Rat Heart In Vivo

Brian Lipton

Experimental Biology and Medicine, 2001

View PDFchevron_right

Toll-like receptor 4 mediates ischemia/reperfusion injury of the heart

Timothy Pohlman

The Journal of Thoracic and Cardiovascular Surgery, 2004

View PDFchevron_right

Potential benefits of cell therapy in coronary heart disease

Teresa Infante

Journal of Cardiology, 2013

View PDFchevron_right

The role of neutrophils in myocardial ischemia–reperfusion injury

J. Vinten-johansen

Cardiovascular Research, 1999

View PDFchevron_right

Identification of Noncytotoxic and IL-10-Producing CD8+AT2R+ T Cell Population in Response to Ischemic Heart Injury

Hans Imboden

The Journal of Immunology, 2010

View PDFchevron_right

Effects of endotoxin on neutrophil-mediated I/R injury in isolated perfused rat hearts

Brian Lipton

American Journal of Physiology-Heart and Circulatory Physiology, 2001

View PDFchevron_right

Cardiac-Selective Expression of Extracellular Superoxide Dismutase After Systemic Injection of Adeno-Associated Virus 9 Protects the Heart Against Post-Myocardial Infarction Left Ventricular Remodeling

Prasad Konkalmatt

Circulation: Cardiovascular Imaging, 2013

View PDFchevron_right

Induced Pluripotent Stem Cell-Derived Endothelial Cells Overexpressing Interleukin-8 Receptors A/B and/or C-C Chemokine Receptors 2/5 Inhibit Vascular Injury Response

Tim Townes

Stem Cells Translational Medicine, 2017

View PDFchevron_right

Pathophysiological role of neutrophils in acute myocardial infarction

Nicolas Vuilleumier

Thrombosis and Haemostasis, 2013

View PDFchevron_right

Gene Therapy With Inducible Nitric Oxide Synthase Protects Against Myocardial Infarction via a Cyclooxygenase-2-Dependent Mechanism

Yu-Ting Xuan

Circulation Research, 2003

View PDFchevron_right

Interleukin-8 as a sensitive marker of unstable coronary artery disease

Tsugiyasu Kanda

The American Journal of Cardiology, 1996

View PDFchevron_right

Adeno-associated virus serotype 9 administered systemically after reperfusion preferentially targets cardiomyocytes in the infarct border zone with pharmacodynamics suitable for the attenuation of left ventricular remodeling

Prasad Konkalmatt

The Journal of Gene Medicine, 2012

View PDFchevron_right

Mice Lacking Inducible Nitric Oxide Synthase Have Improved Left Ventricular Contractile Function and Reduced Apoptotic Cell Death Late After Myocardial Infarction

Krishna Singh

View PDFchevron_right

UltraRapid Communication IL10 Inhibits Inflammation and Attenuates Left Ventricular Remodeling After Myocardial Infarction via Activation of STAT3 and Suppression of HuR

Raj Kishore

2010

View PDFchevron_right