The β and α2δ auxiliary subunits of Cav1 channels are required for Th2-lymphocyte function and acute allergic airway inflammation (original) (raw)
Knocking Down Cav1 Calcium Channels Implicated in Th2 Cell Activation Prevents Experimental Asthma
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American Journal of Respiratory and Critical Care Medicine, 2010
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Protein kinase C–dependent activation of CaV1.2 channels selectively controls human TH2-lymphocyte functions
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Severely blunted allergen-induced pulmonary Th2 cell response and lung hyperresponsiveness in type 1 transient receptor potential channel-deficient mice
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Increased expression of bronchial epithelial transient receptor potential vanilloid 1 channels in patients with severe asthma
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Singularities of calcium signaling in effector T-lymphocytes
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Critical role for the regulatory subunits of Cav channels in T lymphocyte function
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Loss of classical transient receptor potential 6 channel reduces allergic airway response
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Clinical & Experimental Allergy, 2008
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Requirement of voltage-gated calcium channel beta4 subunit for T lymphocyte functions
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Science (New York, N.Y.), 2005
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The α2δ subunit augments functional expression and modifies the pharmacology of CaV1.3 L-type channels
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Cavβ1 regulates T cell expansion and apoptosis independently of voltage-gated Ca2+ channel function
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T Cell Receptor Mediated Calcium Entry Requires Alternatively Spliced Cav1.1 Channels
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PLOS ONE, 2016
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Adriana Sumoza-Toledo
Journal of Inflammation, 2013
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Experimental Physiology, 2008
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