Nicotinamide Adenine Dinucleotide (Phosphate) Reduced:Quinone Oxidoreductase and Glutathione S-Transferase M1 Polymorphisms and Childhood Asthma (original) (raw)
Genetic polymorphism of GSTM1 and antioxidant supplementation influence lung function in relation to ozone exposure in asthmatic children in Mexico City
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Airway oxidative stress is a cardinal feature and an important pathway in asthma pathogenesis.[1] Although a growing body of evidence supports a joint role for air pollutants and variants in genes involved in oxidant defenses in asthma pathogenesis,[2]
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Glutathione-S-transferase gene polymorphisms (GSTT1, GSTM1, GSTP1) as increased risk factors for asthma
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