Change in smoking frequency and affecting factors among the students of a medical faculty in Ankara – Turkey: 2013-2016 (original) (raw)
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Smoking prevalence and related factors among workers of Bornova Municipality in Izmir, Turkey
Tobacco Induced Diseases
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
Use of tobacco products among a Turkish Foundation University students, Izmir 2018
Tobacco Induced Diseases, 2018
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
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Tobacco Induced Diseases
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
Effects of smoking on non-surgical periodontal therapy in generalized aggressive periodontitis
Tobacco Induced Diseases
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
Tobacco Induced Diseases, 2018
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
What is the rate of tobacco usage among dental school students?
Tobacco Induced Diseases
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
Tobacco Induced Diseases, 2018
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.
2018
Background: Environmental tobacco smoke (ETS) or passive smoke exposure is one of the most common preventable health hazards in the community. The risk of disease and death as a result of smoking is not limited to smokers; non-smokers are also at risk from exposure to SHS. Passive smoking has recently been implicated in the development of several systemic diseases. Aims of the study: The purpose of this study was to evaluate the effects of passive smoking on periodontal health status and on the salivary levels of alkaline phosphatase (ALP), lactate dehydrogenase (LDH) and creatine kinase (CK). Materials and methods: The study comprised of 25 non-smokers and 22 passive smokers. Unstimulated saliva sample was collected from each subject for biochemical analysis of salivary enzymes. Periodontal parameters including: plaque index, gingival index, bleeding on probing, probing pocket depth and clinical attachment level were recorded. Results: statistical analysis revealed that ALP in saliva was higher in smoker than the non smoker group and there was significant difference in the salivary LDH and CK levels between smokers and non-smokers groups. Probing pocket depth (PPD) and clinical attachment level (CAL) score 1 was higher in passive smokers compared with non smokers, while there was decrease in the number of bleeding sites. Conclusions: Passive smoking has negative impact on periodontal health and can consider as risk factor for periodontal disease although further studies are needed. Salivary enzymes (ALP, LDH and CK) are considered as good biochemical markers of periodontal tissue destruction and can be used to evaluate the effect of passive smoking on periodontal health status.
Smoking cessation services for health staff: Experiences of a university hospital in Izmir, Turkey
Tobacco Induced Diseases
The aim of this study was to identify probable intermediate biomarkers of disturbed pathways and their link between smoking. Methods Un-stimulated whole saliva and serum samples were collected from a total of 30 systemically healthy participants with periodontally healthy smokers (S) (n=15) and nonsmokers (n=15). Periodontal indices (plaque index, gingival index, probing depth, bleeding on probing, clinical attachment level) were recorded to confirm periodontal health. Saliva was purified, and a total of 28 amino acids and metabolites were analyzed by liquid chromatographymass spectrometry (LC-MS/MS). Smoking status was validated measuring serum cotinine levels. Intergroup comparisons were assessed using the Mann Whitney U test. Results When 28 amino acids were evaluated, smokers had statistically significantly higher cystathionine levels than non-smokers (p <0.05). Conclusions Saliva cystathionine is associated with smoking in periodontally healthy individuals, and is possibly related to altered sulfuration pathway.