Benign Intracranial Hypertension Secondary to Vitamin a Deficiency in Rat and Man: Reversible Collagenous Obstruction of CSF Outflow by Metabolically Deranged Fibroblasts (original) (raw)

Springer eBooks, 1980

Abstract

Pseudotumor cerebri or benign intracranial hypertension can result from impaired outflow of newly-formed cerebrospinal fluid (CSF) from functionally deranged arachnoid villi. An infant with a fat malabsorption syndrome accompanied by deficiency of circulating retinol developed sustained intracranial hypertension that responded to Vitamin A (Vit A) replacement. Simulation of this condition in the rat revealed the reversible pathophysiological mechanism for decreased CSF outflow.

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