Ischemic preconditioning and diazoxide limit mitochondrial Ca overload during ischemia/reperfusion: Role of reactive oxygen species (original) (raw)
Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channels
Vincent Figueredo
American journal of physiology. Heart and circulatory physiology, 2001
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Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial K+ channel activity
Célio Santos
Free Radical Biology and Medicine, 2006
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Protection of cardiac mitochondria by diazoxide and protein kinase C: Implications for ischemic preconditioning
James Weiss
Proceedings of the National Academy of Sciences, 2002
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Protection against ischemia-induced ventricular arrhythmias and myocardial dysfunction conferred by preconditioning in the rat heart: involvement of mitochondrial K(ATP) channels and reactive oxygen species
T. Ravingerová
Physiological research / Academia Scientiarum Bohemoslovaca, 2009
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Cardiac mitochondrial preconditioning by Big Ca2+-sensitive K+ channel opening requires superoxide radical generation
Amadou Camara
American Journal of Physiology-heart and Circulatory Physiology, 2005
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The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respiration
Kelvin Lim
The Journal of physiology, 2002
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Anesthetic Preconditioning Attenuates Mitochondrial Ca2+ Overload During Ischemia in Guinea Pig Intact Hearts: Reversal by 5-Hydroxydecanoic Acid
Samhita Rhodes
Anesthesia & Analgesia, 2002
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Ischemic Preconditioning Improves Mitochondrial Tolerance to Experimental Calcium Overload
Nicolai Doliba, Mary Osbakken
Journal of Surgical Research, 2002
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Cardioprotective Effect of Diazoxide and Its Interaction With Mitochondrial ATP-Sensitive K+ Channels : Possible Mechanism of Cardioprotection
Keith Garlid
Circulation Research, 1997
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MitoK ATP -dependent changes in mitochondrial volume and in complex II activity during ischemic and pharmacological preconditioning of langendorff-perfused rat heart
Pierre Santos
Journal of Bioenergetics and Biomembranes, 2006
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Post–conditioning induced cardioprotection requires signaling through a redox–sensitive mechanism, mitochondrial ATP–sensitive K+ channel and protein kinase C …
claudia penna
Basic Research in Cardiology, 2006
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Improved Mitochondrial Bioenergetics by Anesthetic Preconditioning During and After 2 Hours of 27°C Ischemia in Isolated Hearts
Samhita Rhodes
Journal of Cardiovascular Pharmacology, 2005
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Ischemic Preconditioning Decreases Mitochondrial Proton Leak and Reactive Oxygen Species Production in the Postischemic Heart
Douglas Pfeiffer
Journal of Surgical Research, 2011
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Ischemic preconditioning prevents in vivo hyperoxygenation in postischemic myocardium with preservation of mitochondrial oxygen consumption
Yeong-renn Chen
American Journal of Physiology-Heart and Circulatory Physiology, 2007
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Activation of Mitochondrial ATP-Sensitive K+ Channel for Cardiac Protection Against Ischemic Injury Is Dependent on Protein Kinase C Activity
Sandra Mirandola
Circulation Research, 1999
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Mitochondrial ATP-Dependent Potassium Channels: Viable Candidate Effectors of Ischemic Preconditioninga
Raúl Luján Domenech
Annals of the New York Academy of Sciences, 1999
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Post–conditioning induced cardioprotection requires signaling through a redox–sensitive mechanism, mitochondrial ATP–sensitive K + channel and protein kinase C activation
Gianni Losano
Basic Research in Cardiology, 2006
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Activation of Mitochondrial ATP-Sensitive K1 Channel for Cardiac Protection Against Ischemic Injury Is Dependent on Protein Kinase C Activity
Muhammad Ashraf
2010
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Enhanced Na+/H+ Exchange During Ischemia and Reperfusion Impairs Mitochondrial Bioenergetics and Myocardial Function
Amadou Camara
Journal of Cardiovascular Pharmacology, 2008
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Redox signaling at reperfusion is required for protection from ischemic preconditioning but not from a direct PKC activator
TURHAN DOST
Basic Research in Cardiology, 2008
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Pharmacological preconditioning by diazoxide downregulates cardiac L-type Ca2+ channels
Eddy Carrillo
British Journal of Pharmacology, 2010
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Ischemic preconditioning changes the pattern of coronary reactive hyperemia regardless of mitochondrial ATP-sensitive K+ channel blockade
Pasquale Pagliaro
Life sciences, 2002
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Stimulation of Mitochondrial ATP Synthase Activity – a New Diazoxide-Mediated Mechanism of Cardioprotection
T. Ravingerová
Physiological Research
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Reduced mitochondrial Ca2+ loading and improved functional recovery after ischemia-reperfusion injury in old vs. young guinea pig hearts
Samhita Rhodes
American Journal of Physiology-Heart and Circulatory Physiology, 2011
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Attenuation of oxidant damage in the postconditioned heart involves non-enzymatic response and partial catalytic protection
Francisco Correa, Mabel Buelna
Experimental Physiology, 2012
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Pharmacological preconditioning by diazoxide downregulates cardiac L-type Ca2+ channels: Diazoxide and Ca2+channels
Ed Carrillo
British Journal of Pharmacology, 2010
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Opening of mitochondrial KATPchannel induces early and delayed cardioprotective effect: role of nitric oxide
Venkata Emani
American Journal of Physiology-Heart and Circulatory Physiology, 1999
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Diazoxide amelioration of myocardial injury and mitochondrial damage during cardiac surgery
James McCully
The Annals of Thoracic Surgery, 2002
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The K ATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation
Chris Lawrence
British Journal of Pharmacology, 2001
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Opening of mitochondrial KATPchannels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration
Micheline Federman
American Journal of Physiology-Heart and Circulatory Physiology, 2004
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Cardioprotection by Ischemic Preconditioning Preserves Mitochondrial Function and Functional Coupling Between Adenine Nucleotide Translocase and Creatine Kinase
Keith Garlid
Journal of Molecular and Cellular Cardiology, 2001
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