Nuclear and non-nuclear targets for Gâ delay in mammalian cells (original) (raw)

Environmental and Molecular Mutagenesis, 1995

Abstract

The role of DNA double-strand breaks in producing mitotic (Gâ) delay was examined in Chinese hamster ovary (CHO) cells. Restriction enzymes, which produce only DNA double-stranded breaks, were introduced by electroporation into CHO-K1 and its radiation-sensitive derivativexrs-5; at several time points after treatment, the fraction of cells in Gâ was determined. Electroporation of Alu I or Sau 3A1 into CHO-K1

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