Atypical pacemaker-mediated tachycardia from the atrial channel: What is the mechanism? (original) (raw)

A 79-year-old woman with history of hypertension, dyslipidemia, obesity, bulbar stroke , and paroxysmal atrial fibrillation was evaluated for an abrupt syncope. Echocardiogram showed no abnormalities, and Holter monitoring showed sinus arrhythmia with right bundle branch block (RBBB) and signs of sick sinus syndrome due to significant pauses. A dual-chamber pacemaker was implanted without complications (ventricular electrode in the right ventricular apex and atrial electrode at the high right atrial appendage). Pacemaker settings were DDDR mode, basic rate 65 bpm, maximum sensor rate 110 bpm, maximum tracking rate 150 bpm, and dynamic AV interval of maximum 280/minimum KEYWORDS Pacemaker-mediated tachycardia; electrodes dislodgement ABBREVIATIONS ECG ϭ electrocardiogram; PMT ϭ pacemaker-mediated tachycardia; RBBB ϭ right bundle branch block (Heart Rhythm 2011;8:636 -638)

Tachycardia after pacemaker implantation in a patient with complete atrioventricular block

Europace, 2007

The atrioventricular (AV) node allows ante-and retrograde conduction between atria and ventricles. It is commonly assumed that these AV nodal conduction properties go hand in hand. However, ante-and retrograde AV conduction can be completely independent from each other in individual patients. We report about a patient with permanent AV block III8 requiring implantation of a pacemaker. As soon as a dual-chamber device was connected to the implanted leads, a tachycardia started at the maximum tracking rate, which was subsequently reprogrammed from 120 to 170 bpm. Non-invasive electrophysiologic testing showed that this patient demonstrated 1:1 ventriculoatrial (VA) conduction up to 170 bpm leading to endless loop tachycardia (ELT) while the antegrade AV block III8 persisted. This case impressively illustrates that one has to take into account that patients with antegrade AV block III8 may still have a high VA conduction capacity leading to ELT. Dual-chamber devices therefore have to be programmed accordingly, activating dedicated reactions after ventricular premature beats and automatic ELT detection and termination algorithms.

Pacemaker-mediated tachycardias: a new modality of treatment

Pacing and clinical electrophysiology : PACE, 1984

Three patients with pacemaker interactive drug resistant tachycardia underwent invasive electrophysiological studies. In the first patient, the retrograde conduction of the artificial reciprocating tachycardia was provided by two right-sided accessory pathways and the antegrade conduction by an atrial synchronous pulse generator. In addition, AV-nodal tachycardia occurred alternately. In the second patient with intermittent atrial flutter, the AV node and, coincidentally, an AV sequential pulse generator provided high-rate antegrade conduction to the ventricles. In the third patient with surgical complete heart block, intermittent AV-nodal tachycardia induced retrograde atrial activation while an atrial synchronous pacemaker provided the antegrade conduction. Electrode catheter exploration of the heart allowed localization and closed-chest ablation of the accessory pathways or AV node by delivering two to seven 200-joule direct-current shocks through the appropriate electrode of the...

Wide complex tachycardia in a patient with paroxysmal atrial fibrillation

Heart Rhythm, 2008

A 63-year old man was admitted to the hospital for catheter ablation of paroxysmal atrial fibrillation. He first experienced recurrent episodes of palpitations and weakness related to atrial fibrillation in 2001. Treatment with oral amiodarone was discontinued a few months later because of development of thyroiditis. A Medtronic AT500 dual-chamber pacemaker was implanted in 2005. Symptoms of palpitations increased in frequency and became daily, requiring repeated hospitalizations. On the day before the ablation procedure, a wide complex tachycardia was recorded on the telemetry monitor. Onset and 12-lead ECG of the tachycardia are shown in . The patient remained in this tachycardia for 2 hours until pacemaker interrogation was performed ( ).The tachycardia terminated as soon as the pacemaker wand was applied. What is the mechanism of this tachycardia?

Pacemaker-mediated tachycardia with varying cycle length: what is the mechanism?

Europace, 2009

Two months before the ablation procedure, the patient underwent a 16-slice spiral computed tomography (CT), which showed a typical pattern of dextrocardia as depicted in . In addition, the CT images were essential to understand the patient's anatomy: as shown in , the pulmonary veins (PVs) had a common trunk on the right side of the morphological left atrium and there were two separate pulmonary veins with early branching on the left side of the morphological left atrium.

Wide complex tachycardia in a patient with a dual chamber pacemaker

Europace, 2008

An 81-year-old patient was admitted to the coronary care unit due to unstable angina and respiratory distress after urgent eye surgery for retinal detachment. He had a medical history of hypertension, diabetes mellitus, coronary artery disease, and valvular heart disease. He underwent coronary artery bypass grafting and aortic valve replacement in 1998 and received a dual chamber pacemaker (Pulsar Max DR Guidant/Boston Scientific Natick, MA, USA) for third-degree heart block in 1999. The diagnosis of myocardial infarction complicated by acute pulmonary oedema was withheld, in view of a rise in troponin I level to a maximum of 14.66 mg/L (normal value ,0.14 mg/L). ST-segment elevation could not be assessed because of ventricular pacing ( . He developed respiratory failure for which mechanical ventilation was initiated. His echocardiography showed a depressed left ventricular function with an ejection fraction of 30% due to a large antero-lateral myocardial infarction. An urgent coronary angiography showed a critical stenosis of the left coronary artery, and a percutaneous coronary intervention of the native left anterior descending and circumflex artery was performed.

Ventricular tachycardia induced by pacing algorithm designed to avoid atrial fibrillation

Revista portuguesa de cardiologia, 2020

A patient with a dual chamber pacemaker was admitted to the emergency room after out-of-hospital cardioversion for syncopal sustained monomorphic ventricular tachycardia. Device interrogation revealed an abnormally timed ventricular spike after a ventricular premature beat at the beginning of the event, caused by a pacemaker algorithm designed to avoid atrial fibrillation, non-competitive atrial pacing. Despite the absence of significant coronary lesions, in the setting of a vulnerable substrate-a hypokinetic and hyperechogenic region of ventricular myocardium-a n upgrade to a dual-chamber implantable cardioverter-defibrillator was performed, and substrate ablation was planned.

Loading...

Loading Preview

Sorry, preview is currently unavailable. You can download the paper by clicking the button above.